B cell sub-types following acute malaria and associations with clinical immunity

[1]  M. Feeney,et al.  B cell sub-types following acute malaria and associations with clinical immunity , 2016, Malaria Journal.

[2]  Arlo Z. Randall,et al.  Biosignatures of Exposure/Transmission and Immunity , 2015, The American journal of tropical medicine and hygiene.

[3]  Peter D. Crompton,et al.  Malaria-associated atypical memory B cells exhibit markedly reduced B cell receptor signaling and effector function , 2015, eLife.

[4]  Charles C. Kim,et al.  FCRL5 Delineates Functionally Impaired Memory B Cells Associated with Plasmodium falciparum Exposure , 2015, PLoS pathogens.

[5]  J. Beeson Faculty Opinions recommendation of Atypical memory B cells are greatly expanded in individuals living in a malaria-endemic area. , 2014 .

[6]  A. Adegnika,et al.  Interleukin 10 (IL-10)-producing CD1dhi regulatory B cells from Schistosoma haematobium-infected individuals induce IL-10-positive T cells and suppress effector T-cell cytokines. , 2014, The Journal of infectious diseases.

[7]  D. Conway,et al.  Dynamics of the antibody response to Plasmodium falciparum infection in African children. , 2014, The Journal of infectious diseases.

[8]  L. Hviid,et al.  Kinetics of B Cell Responses to Plasmodium falciparum Erythrocyte Membrane Protein 1 in Ghanaian Women Naturally Exposed to Malaria Parasites , 2014, The Journal of Immunology.

[9]  M. Feeney,et al.  IFNγ/IL-10 Co-producing Cells Dominate the CD4 Response to Malaria in Highly Exposed Children , 2014, PLoS pathogens.

[10]  David L Smith,et al.  Estimating the annual entomological inoculation rate for Plasmodium falciparum transmitted by Anopheles gambiae s.l. using three sampling methods in three sites in Uganda , 2014, Malaria Journal.

[11]  Beatrix Ueberheide,et al.  Atypical and classical memory B cells produce Plasmodium falciparum neutralizing antibodies , 2013, The Journal of experimental medicine.

[12]  Peter D. Crompton,et al.  Chronic Exposure to Plasmodium falciparum Is Associated with Phenotypic Evidence of B and T Cell Exhaustion , 2013, The Journal of Immunology.

[13]  P. Rosenthal,et al.  Increasing incidence of malaria in children despite insecticide-treated bed nets and prompt anti-malarial therapy in Tororo, Uganda , 2012, Malaria Journal.

[14]  Yu Qian,et al.  Advances in Human B Cell Phenotypic Profiling , 2012, Front. Immun..

[15]  D. Conway,et al.  The Breadth, but Not the Magnitude, of Circulating Memory B Cell Responses to P. falciparum Increases with Age/Exposure in an Area of Low Transmission , 2011, PloS one.

[16]  M. Wahlgren,et al.  TLRs innate immunereceptors and Plasmodium falciparum erythrocyte membrane protein 1 (PfEMP1) CIDR1α-driven human polyclonal B-cell activation. , 2011, Acta tropica.

[17]  S. Magez,et al.  Acute Disruption of Bone Marrow B Lymphopoiesis and Apoptosis of Transitional and Marginal Zone B Cells in the Spleen following a Blood-Stage Plasmodium chabaudi Infection in Mice , 2011, Journal of parasitology research.

[18]  Peter D. Crompton,et al.  A Positive Correlation between Atypical Memory B Cells and Plasmodium falciparum Transmission Intensity in Cross-Sectional Studies in Peru and Mali , 2011, PloS one.

[19]  E. Nduati,et al.  Distinct Kinetics of Memory B-Cell and Plasma-Cell Responses in Peripheral Blood Following a Blood-Stage Plasmodium chabaudi Infection in Mice , 2010, PloS one.

[20]  M. Bagot,et al.  IL‐10 produced by activated human B cells regulates CD4+ T‐cell activation in vitro , 2010, European journal of immunology.

[21]  Peter D. Crompton,et al.  The Plasmodium falciparum-Specific Human Memory B Cell Compartment Expands Gradually with Repeated Malaria Infections , 2010, PLoS pathogens.

[22]  D. Isenberg,et al.  CD19(+)CD24(hi)CD38(hi) B cells exhibit regulatory capacity in healthy individuals but are functionally impaired in systemic Lupus Erythematosus patients. , 2010, Immunity.

[23]  A Orfao,et al.  Human peripheral blood B‐cell compartments: A crossroad in B‐cell traffic , 2010, Cytometry. Part B, Clinical cytometry.

[24]  G. Blanchard-Rohner,et al.  Appearance of peripheral blood plasma cells and memory B cells in a primary and secondary immune response in humans. , 2009, Blood.

[25]  P. Rosenthal,et al.  Artemether-lumefantrine versus dihydroartemisinin-piperaquine for falciparum malaria: a longitudinal, randomized trial in young Ugandan children. , 2009, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.

[26]  J. Tappero,et al.  Safety and tolerability of artemether-lumefantrine versus dihydroartemisinin-piperaquine for malaria in young HIV-infected and uninfected children , 2009, Malaria Journal.

[27]  C. Caruso,et al.  A double-negative (IgD−CD27−) B cell population is increased in the peripheral blood of elderly people , 2009, Mechanisms of Ageing and Development.

[28]  J. Coura,et al.  Age-dependent acquisition of protective immunity to malaria in riverine populations of the Amazon Basin of Brazil. , 2009, The American journal of tropical medicine and hygiene.

[29]  K. Marsh,et al.  Analysis of Immunity to Febrile Malaria in Children That Distinguishes Immunity from Lack of Exposure , 2009, Infection and Immunity.

[30]  R. Ploutz-Snyder,et al.  Alterations on peripheral B cell subsets following an acute uncomplicated clinical malaria infection in children , 2008, Malaria Journal.

[31]  Wei Wang,et al.  Evidence for HIV-associated B cell exhaustion in a dysfunctional memory B cell compartment in HIV-infected viremic individuals , 2008, The Journal of experimental medicine.

[32]  O. Ohara,et al.  Discriminating gene expression profiles of memory B cell subpopulations , 2008, The Journal of experimental medicine.

[33]  Kevin Marsh,et al.  Immunity to malaria: more questions than answers , 2008, Nature Immunology.

[34]  P. Lipsky,et al.  Activated memory B cell subsets correlate with disease activity in systemic lupus erythematosus: delineation by expression of CD27, IgD, and CD95. , 2008, Arthritis and rheumatism.

[35]  D. Conway,et al.  Duration of Naturally Acquired Antibody Responses to Blood-Stage Plasmodium falciparum Is Age Dependent and Antigen Specific , 2008, Infection and Immunity.

[36]  E. Milner,et al.  A New Population of Cells Lacking Expression of CD27 Represents a Notable Component of the B Cell Memory Compartment in Systemic Lupus Erythematosus1 , 2007, The Journal of Immunology.

[37]  K. Marsh,et al.  Immune effector mechanisms in malaria , 2006, Parasite immunology.

[38]  M. Cooper,et al.  Expression of the immunoregulatory molecule FcRH4 defines a distinctive tissue-based population of memory B cells , 2005, The Journal of experimental medicine.

[39]  S. Nutt,et al.  Early appearance of germinal center–derived memory B cells and plasma cells in blood after primary immunization , 2005, The Journal of experimental medicine.

[40]  E. Riley,et al.  Does malaria suffer from lack of memory? , 2004, Immunological reviews.

[41]  M. Wahlgren,et al.  Identification of a Polyclonal B-Cell Activator in Plasmodium falciparum , 2004, Infection and Immunity.

[42]  S. Hoffman,et al.  Incidence of symptomatic and asymptomatic Plasmodium falciparum infection following curative therapy in adult residents of northern Ghana. , 2001, The American journal of tropical medicine and hygiene.

[43]  S. Hoffman,et al.  Impact of transmission intensity and age on Plasmodium falciparum density and associated fever: implications for malaria vaccine trial design. , 1995, The Journal of infectious diseases.

[44]  H. Whittle,et al.  The effects of Plasmodium falciparum malaria on immune control of B lymphocytes in Gambian children , 1990, Clinical and experimental immunology.

[45]  K Y Liang,et al.  Longitudinal data analysis for discrete and continuous outcomes. , 1986, Biometrics.

[46]  I. McGregor,et al.  Gamma-Globulin and Acquired Immunity to Human Malaria , 1961, Nature.

[47]  W H TALIAFERRO,et al.  Acquired immunity in malaria. , 1948, Abstracts. International Congress on Tropical Medicine and Malaria.