Mechanisms of postischemic contractile dysfunction.

[1]  C. Valdivia,et al.  Ryanodine receptor dysfunction in porcine stunned myocardium. , 1997, The American journal of physiology.

[2]  D. Atar,et al.  Role of troponin I proteolysis in the pathogenesis of stunned myocardium. , 1997, Circulation research.

[3]  E. Marbán,et al.  Selective effects of oxygen free radicals on excitation-contraction coupling in ventricular muscle. Implications for the mechanism of stunned myocardium. , 1996, Circulation.

[4]  P. Pagel,et al.  Mechanism of Myocardial Protection by Isoflurane: Role of Adenosine Triphosphate‐regulated Potassium (K sub ATP) Channels , 1996, Anesthesiology.

[5]  K. McDonald,et al.  Onset of reduced Ca2+ sensitivity of tension during stunning in porcine myocardium. , 1996, Journal of molecular and cellular cardiology.

[6]  Yongge Liu,et al.  Intrinsic myofilament alterations underlying the decreased contractility of stunned myocardium. A consequence of Ca2+-dependent proteolysis? , 1996, Circulation research.

[7]  G. Gross,et al.  Monophosphoryl lipid A preserves myocardial contractile function following multiple, brief periods of coronary occlusion in dogs. , 1995, Pharmacology.

[8]  R. Bolli,et al.  Late preconditioning against myocardial stunning. An endogenous protective mechanism that confers resistance to postischemic dysfunction 24 h after brief ischemia in conscious pigs. , 1995, The Journal of clinical investigation.

[9]  J. Thornby,et al.  Direct evidence that the hydroxyl radical plays a pathogenetic role in myocardial "stunning" in the conscious dog and demonstration that stunning can be markedly attenuated without subsequent adverse effects. , 1993, Circulation research.

[10]  R. Bolli,et al.  Demonstration of free radical generation in the "stunned" myocardium in the conscious dog and identification of major differences between conscious and open-chest dogs. , 1993, The Journal of clinical investigation.

[11]  G. Gross,et al.  Glibenclamide Antagonizes Adenosine A, Receptor‐Mediated Cardioprotection in Stunned Canine Myocardium , 1993, Circulation.

[12]  M. Karmazyn,et al.  Role of Na+/H+ exchange in cardiac physiology and pathophysiology: mediation of myocardial reperfusion injury by the pH paradox. , 1993, Cardiovascular research.

[13]  R. Moss,et al.  Altered calcium sensitivity of isometric tension in myocyte-sized preparations of porcine postischemic stunned myocardium. , 1993, Circulation research.

[14]  W. Grossman,et al.  Decreased myofilament responsiveness in myocardial stunning follows transient calcium overload during ischemia and reperfusion. , 1992, Circulation research.

[15]  R. Mentzer,et al.  Adenosine improves recovery of postischemic myocardial function via an adenosine A1 receptor mechanism. , 1992, The American journal of physiology.

[16]  G. Gross,et al.  Role of ATP dependent potassium channels in myocardial ischaemia. , 1992, Cardiovascular research.

[17]  J. Downey,et al.  Ischemic preconditioning Nature's own cardioprotective intervention. , 1992, Trends in cardiovascular medicine.

[18]  G. Gross,et al.  Pharmacological Evidence for a Role of ATP‐Dependent Potassium Channels in Myocardial Stunning , 1992, Circulation.

[19]  R. Kloner,et al.  Preconditioning Does Not Attenuate Myocardial Stunning , 1992, Circulation.

[20]  D. Miller,et al.  Depression of peak force without altering calcium sensitivity by the superoxide anion in chemically skinned cardiac muscle of rat. , 1992, Circulation research.

[21]  R. Bolli.  Mechanism of Myocardial “Stunning” , 1990, Circulation.

[22]  V. P. Chacko,et al.  Calcium and Its Role in Myocardial Cell Injury During Ischemia and Reperfusion , 1989, Circulation.

[23]  R. Kloner,et al.  Is "stunned myocardium" a protective mechanism? Effect of acute recruitment and acute beta-blockade on recovery of contractile function and high-energy phosphate stores at 1 day post-reperfusion. , 1989, American heart journal.

[24]  R. Bolli,et al.  Marked Reduction of Free Radical Generation and Contractile Dysfunction by Antioxidant Therapy Begun at the Time of Reperfusion Evidence That Myocardial "Stunning" Is a Manifestation of Reperfusion Injury , 1989, Circulation research.

[25]  L. Becker,et al.  Alterations in cardiac sarcoplasmic reticulum calcium transport in the postischemic "stunned" myocardium. , 1989, Circulation research.

[26]  R. Kloner,et al.  Nifedipine administered after reperfusion ablates systolic contractile dysfunction of postischemic "stunned" myocardium. , 1989, Journal of the American College of Cardiology.

[27]  R. Bolli,et al.  Demonstration of free radical generation in "stunned" myocardium of intact dogs with the use of the spin trap alpha-phenyl N-tert-butyl nitrone. , 1988, The Journal of clinical investigation.

[28]  G. Gross,et al.  Evidence For a Role of Iron‐Catalyzed Oxidants in Functional and Metabolic Stunning in the Canine Heart , 1988, Circulation research.

[29]  R. Kloner,et al.  Effect of verapamil on postischemic "stunned" myocardium: importance of the timing of treatment. , 1988, Journal of the American College of Cardiology.

[30]  C. Hartley,et al.  Attenuation of dysfunction in the postischemic 'stunned' myocardium by dimethylthiourea. , 1987, Circulation.

[31]  M. Weisfeldt,et al.  Pathophysiology and pathogenesis of stunned myocardium. Depressed Ca2+ activation of contraction as a consequence of reperfusion-induced cellular calcium overload in ferret hearts. , 1987, The Journal of clinical investigation.

[32]  D. Warltier,et al.  Beneficial actions of superoxide dismutase and catalase in stunned myocardium of dogs. , 1986, The American journal of physiology.

[33]  R. Kloner,et al.  Superoxide Dismutase Plus Catalase Improve Contractile Function in the Canine Model of the “Stunned Myocardium” , 1986, Circulation research.

[34]  C. Hartley,et al.  Enhancement of recovery of myocardial function by oxygen free-radical scavengers after reversible regional ischemia. , 1985, Circulation.

[35]  G. Gross,et al.  Improved Recovery of Myocardial Segment Function Following a Short Coronary Occlusion in Dogs by Nicorandil, a Potential New Antianginal Agent, and Nifedipine , 1985, Journal of cardiovascular pharmacology.

[36]  E. Braunwald,et al.  Studies of experimental coronary artery reperfusion. Effects on infarct size, myocardial function, biochemistry, ultrastructure and microvascular damage. , 1983, Circulation.

[37]  E. Braunwald,et al.  The Stunned Myocardium: Prolonged, Postischemic Ventricular Dysfunction , 1982, Circulation.

[38]  S. Vatner,et al.  Regional myocardial functional and electrophysiological alterations after brief coronary artery occlusion in conscious dogs. , 1975, The Journal of clinical investigation.