Inflammation in Atherosclerosis
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[1] P. Libby,et al. Extramedullary Hematopoiesis Generates Ly-6Chigh Monocytes That Infiltrate Atherosclerotic Lesions , 2012, Circulation.
[2] P. Libby,et al. Selective Inhibition of Matrix Metalloproteinase-13 Increases Collagen Content of Established Mouse Atherosclerosis , 2011, Arteriosclerosis, thrombosis, and vascular biology.
[3] P. Libby,et al. Interleukin-1β inhibition and the prevention of recurrent cardiovascular events: rationale and design of the Canakinumab Anti-inflammatory Thrombosis Outcomes Study (CANTOS). , 2011, American heart journal.
[4] P. Libby,et al. IgE stimulates human and mouse arterial cell apoptosis and cytokine expression and promotes atherogenesis in Apoe-/- mice. , 2011, The Journal of clinical investigation.
[5] P. Libby,et al. Progress and challenges in translating the biology of atherosclerosis , 2011, Nature.
[6] C. Uyttenhove,et al. B cell depletion reduces the development of atherosclerosis in mice , 2010, The Journal of experimental medicine.
[7] J. Danesh,et al. C-reactive protein concentration and risk of coronary heart disease, stroke, and mortality: an individual participant meta-analysis , 2010, The Lancet.
[8] I. Tabas. Macrophage death and defective inflammation resolution in atherosclerosis , 2010, Nature Reviews Immunology.
[9] G. Hansson,et al. The discovery of cellular immunity in the atherosclerotic plaque. , 2009, Arteriosclerosis, thrombosis, and vascular biology.
[10] P. Libby,et al. Kruppel-like Factor KLF10 Targets Transforming Growth Factor-β1 to Regulate CD4+CD25− T Cells and T Regulatory Cells* , 2009, The Journal of Biological Chemistry.
[11] P. Ridker. Testing the inflammatory hypothesis of atherothrombosis: scientific rationale for the cardiovascular inflammation reduction trial (CIRT) , 2009, Journal of thrombosis and haemostasis : JTH.
[12] G. Hansson,et al. Vaccination against atherosclerosis? Induction of atheroprotective immunity , 2009, Seminars in Immunopathology.
[13] Børge G Nordestgaard,et al. Reduction in C-reactive protein and LDL cholesterol and cardiovascular event rates after initiation of rosuvastatin: a prospective study of the JUPITER trial , 2009, The Lancet.
[14] P. Libby,et al. Rosuvastatin to prevent vascular events in men and women with elevated C-reactive protein. , 2008, The New England journal of medicine.
[15] J. Witztum,et al. Natural antibodies in murine atherosclerosis. , 2008, Current drug targets.
[16] P. Libby,et al. Matrix Metalloproteinase-14 Deficiency in Bone Marrow–Derived Cells Promotes Collagen Accumulation in Mouse Atherosclerotic Plaques , 2008, Circulation.
[17] P. Libby,et al. Mast cells promote atherosclerosis by releasing proinflammatory cytokines , 2007, Nature Medicine.
[18] A. Zernecke,et al. Perivascular Mast Cells Promote Atherogenesis and Induce Plaque Destabilization in Apolipoprotein E–Deficient Mice , 2007, Circulation.
[19] N. Cook,et al. Development and validation of improved algorithms for the assessment of global cardiovascular risk in women: the Reynolds Risk Score. , 2007, JAMA.
[20] F. Tacke,et al. Monocyte subsets differentially employ CCR2, CCR5, and CX3CR1 to accumulate within atherosclerotic plaques. , 2007, The Journal of clinical investigation.
[21] P. Libby,et al. Ly-6Chi monocytes dominate hypercholesterolemia-associated monocytosis and give rise to macrophages in atheromata. , 2007, The Journal of clinical investigation.
[22] R. Flavell,et al. Natural regulatory T cells control the development of atherosclerosis in mice , 2006, Nature Medicine.
[23] Elena Aikawa,et al. Matrix Metalloproteinase-13/Collagenase-3 Deletion Promotes Collagen Accumulation and Organization in Mouse Atherosclerotic Plaques , 2005, Circulation.
[24] P. Libby,et al. Genetically Determined Resistance to Collagenase Action Augments Interstitial Collagen Accumulation in Atherosclerotic Plaques , 2004, Circulation.
[25] M. Nieminen,et al. For Personal Use. Only Reproduce with Permission from the Lancet Publishing Group , 2022 .
[26] G. Hansson,et al. Protective immunity against atherosclerosis carried by B cells of hypercholesterolemic mice. , 2002, The Journal of clinical investigation.
[27] P. Libby,et al. Inflammation and Atherosclerosis , 2002, Circulation.
[28] R. Virmani,et al. Pathology of the unstable plaque. , 2002, Progress in cardiovascular diseases.
[29] P. Libby,et al. Expression of Neutrophil Collagenase (Matrix Metalloproteinase-8) in Human Atheroma: A Novel Collagenolytic Pathway Suggested by Transcriptional Profiling , 2001, Circulation.
[30] Peter Libby,et al. Current Concepts of the Pathogenesis of the Acute Coronary Syndromes , 2001, Circulation.
[31] Paul M. Ridker,et al. Measurement of C-reactive protein for the targeting of statin therapy in the primary prevention of acute coronary events. , 2001, The New England journal of medicine.
[32] M. Cybulsky,et al. A major role for VCAM-1, but not ICAM-1, in early atherosclerosis. , 2001, The Journal of clinical investigation.
[33] P. Libby,et al. Macrophage myeloperoxidase regulation by granulocyte macrophage colony-stimulating factor in human atherosclerosis and implications in acute coronary syndromes. , 2001, The American journal of pathology.
[34] M. Cybulsky,et al. NF-κB: pivotal mediator or innocent bystander in atherogenesis? , 2001 .
[35] B. Jude,et al. PPARα Agonists Inhibit Tissue Factor Expression in Human Monocytes and Macrophages , 2001 .
[36] P. Libby,et al. An HMG-CoA Reductase Inhibitor, Cerivastatin, Suppresses Growth of Macrophages Expressing Matrix Metalloproteinases and Tissue Factor In Vivo and In Vitro , 2001, Circulation.
[37] P. Libby,et al. PPAR&agr; Activators Inhibit Tissue Factor Expression and Activity in Human Monocytes , 2001, Circulation.
[38] Xinghua Zhou,et al. Transfer of CD4+ T Cells Aggravates Atherosclerosis in Immunodeficient Apolipoprotein E Knockout Mice , 2000, Circulation.
[39] Richard T. Lee,et al. Overexpression of Eotaxin and the CCR3 Receptor in Human Atherosclerosis: Using Genomic Technology to Identify a Potential Novel Pathway of Vascular Inflammation , 2000, Circulation.
[40] S. Yusuf,et al. Effects of an angiotensin-converting-enzyme inhibitor, ramipril, on cardiovascular events in high-risk patients. The Heart Outcomes Prevention Evaluation Study Investigators. , 2000 .
[41] Frank J. Gonzalez,et al. Peroxisome Proliferator-activated Receptor α Negatively Regulates the Vascular Inflammatory Gene Response by Negative Cross-talk with Transcription Factors NF-κB and AP-1* , 1999, The Journal of Biological Chemistry.
[42] P. Libby,et al. Differential expression of three T lymphocyte-activating CXC chemokines by human atheroma-associated cells. , 1999, The Journal of clinical investigation.
[43] T Suzuki,et al. Process of progression of coronary artery lesions from mild or moderate stenosis to moderate or severe stenosis: A study based on four serial coronary arteriograms per year. , 1999, Circulation.
[44] P. Libby,et al. Angiotensin induces inflammatory activation of human vascular smooth muscle cells. , 1999, Arteriosclerosis, thrombosis, and vascular biology.
[45] P. Libby,et al. PPARα Activators Inhibit Cytokine-Induced Vascular Cell Adhesion Molecule-1 Expression in Human Endothelial Cells , 1999 .
[46] P. Libby,et al. Evidence for increased collagenolysis by interstitial collagenases-1 and -3 in vulnerable human atheromatous plaques. , 1999, Circulation.
[47] P. Libby,et al. Inflammatory Cytokines and Oxidized Low Density Lipoproteins Increase Endothelial Cell Expression of Membrane Type 1-Matrix Metalloproteinase* , 1999, The Journal of Biological Chemistry.
[48] M A Konerding,et al. Angiogenesis inhibitors endostatin or TNP-470 reduce intimal neovascularization and plaque growth in apolipoprotein E-deficient mice. , 1999, Circulation.
[49] A. Schmidt,et al. Activation of receptor for advanced glycation end products: a mechanism for chronic vascular dysfunction in diabetic vasculopathy and atherosclerosis. , 1999, Circulation research.
[50] A. Becker,et al. Leucocyte recruitment in rupture prone regions of lipid-rich plaques: a prominent role for neovascularization? , 1999, Cardiovascular research.
[51] M. Gimbrone,et al. Blood flow and vascular gene expression: fluid shear stress as a modulator of endothelial phenotype. , 1999, Molecular medicine today.
[52] G. Fonarow,et al. High density associated enzymes: their role in vascular biology. , 1998, Current opinion in lipidology.
[53] M. Pfeffer,et al. Inflammation, pravastatin, and the risk of coronary events after myocardial infarction in patients with average cholesterol levels. Cholesterol and Recurrent Events (CARE) Investigators. , 1998, Circulation.
[54] I. Charo,et al. Decreased lesion formation in CCR2−/− mice reveals a role for chemokines in the initiation of atherosclerosis , 1998, Nature.
[55] P. Libby,et al. Absence of monocyte chemoattractant protein-1 reduces atherosclerosis in low density lipoprotein receptor-deficient mice. , 1998, Molecular cell.
[56] R. Hynes,et al. The combined role of P- and E-selectins in atherosclerosis. , 1998, The Journal of clinical investigation.
[57] M. Kuzuya,et al. Induction of macrophage VEGF in response to oxidized LDL and VEGF accumulation in human atherosclerotic lesions. , 1998, Arteriosclerosis, thrombosis, and vascular biology.
[58] P. Libby,et al. Heterozygous osteopetrotic (op) mutation reduces atherosclerosis in LDL receptor- deficient mice. , 1998, The Journal of clinical investigation.
[59] R. Terkeltaub,et al. A leukocyte homologue of the IL-8 receptor CXCR-2 mediates the accumulation of macrophages in atherosclerotic lesions of LDL receptor-deficient mice. , 1998, The Journal of clinical investigation.
[60] P. Libby,et al. The unstable atheroma. , 1997, Arteriosclerosis, thrombosis, and vascular biology.
[61] P. Libby,et al. Interferon-gamma deficiency prevents coronary arteriosclerosis but not myocardial rejection in transplanted mouse hearts. , 1997, The Journal of clinical investigation.
[62] P. Libby,et al. Activation of monocyte/macrophage functions related to acute atheroma complication by ligation of CD40: induction of collagenase, stromelysin, and tissue factor. , 1997, Circulation.
[63] Alan D. Lopez,et al. Global mortality, disability, and the contribution of risk factors: Global Burden of Disease Study , 1997, The Lancet.
[64] P. Libby,et al. Role of macrophage colony-stimulating factor in atherosclerosis: studies of osteopetrotic mice. , 1997, The American journal of pathology.
[65] P. Ridker,et al. Inflammation, aspirin, and the risk of cardiovascular disease in apparently healthy men. , 1997, The New England journal of medicine.
[66] T. Mayadas,et al. Absence of P-selectin delays fatty streak formation in mice. , 1997, The Journal of clinical investigation.
[67] P. Kovanen,et al. TNF-alpha and IL-1beta selectively induce expression of 92-kDa gelatinase by human macrophages. , 1996, Journal of immunology.
[68] M J Davies,et al. Stability and instability: two faces of coronary atherosclerosis. The Paul Dudley White Lecture 1995. , 1996, Circulation.
[69] P. Libby,et al. Apoptosis of Vascular Smooth Muscle Cells Induced by In Vitro Stimulation With Interferon-γ, Tumor Necrosis Factor–α, and Interleukin-1β , 1996 .
[70] P. Libby,et al. Components of the protein fraction of oxidized low density lipoprotein stimulate interleukin-1 alpha production by rabbit arterial macrophage-derived foam cells. , 1995, Journal of lipid research.
[71] T. Paavonen,et al. Infiltrates of activated mast cells at the site of coronary atheromatous erosion or rupture in myocardial infarction. , 1995, Circulation.
[72] Michael Ginsberg,et al. Decreased atherosclerosis in mice deficient in both macrophage colony-stimulating factor (op) and apolipoprotein E. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[73] P. Libby,et al. Evidence for apoptosis in advanced human atheroma. Colocalization with interleukin-1 beta-converting enzyme. , 1995, The American journal of pathology.
[74] P. Libby,et al. Nitric oxide decreases cytokine-induced endothelial activation. Nitric oxide selectively reduces endothelial expression of adhesion molecules and proinflammatory cytokines. , 1995, The Journal of clinical investigation.
[75] P. Libby. Molecular bases of the acute coronary syndromes. , 1995, Circulation.
[76] P. Libby,et al. Increased expression of matrix metalloproteinases and matrix degrading activity in vulnerable regions of human atherosclerotic plaques. , 1994, The Journal of clinical investigation.
[77] A. Rebuzzi,et al. The prognostic value of C-reactive protein and serum amyloid a protein in severe unstable angina. , 1994, The New England journal of medicine.
[78] P. Libby,et al. Distinct patterns of expression of fibroblast growth factors and their receptors in human atheroma and nonatherosclerotic arteries. Association of acidic FGF with plaque microvessels and macrophages. , 1993, The Journal of clinical investigation.
[79] P. Libby,et al. An atherogenic diet rapidly induces VCAM-1, a cytokine-regulatable mononuclear leukocyte adhesion molecule, in rabbit aortic endothelium. , 1993, Arteriosclerosis and thrombosis : a journal of vascular biology.
[80] P. Libby,et al. Increased apolipoprotein E and c-fms gene expression without elevated interleukin 1 or 6 mRNA levels indicates selective activation of macrophage functions in advanced human atheroma. , 1992, Proceedings of the National Academy of Sciences of the United States of America.
[81] P. Libby,et al. Macrophage colony-stimulating factor gene expression in vascular cells and in experimental and human atherosclerosis. , 1992, The American journal of pathology.
[82] V. Ord,et al. Macrophage colony-stimulating factor mRNA and protein in atherosclerotic lesions of rabbits and humans. , 1992, The American journal of pathology.
[83] P. Libby,et al. Cytokines as mediators of vascular pathology. , 1992, Nouvelle revue francaise d'hematologie.
[84] P. Libby,et al. Human coronary transplantation-associated arteriosclerosis. Evidence for a chronic immune reaction to activated graft endothelial cells. , 1991, The American journal of pathology.
[85] M. Cybulsky,et al. Endothelial expression of a mononuclear leukocyte adhesion molecule during atherogenesis. , 1991, Science.
[86] W. Weintraub,et al. Elevation of C-reactive protein in "active" coronary artery disease. , 1990, The American journal of cardiology.
[87] P. Libby,et al. Functions of vascular wall cells related to development of transplantation-associated coronary arteriosclerosis. , 1989, Transplantation proceedings.
[88] R. Detrano,et al. The dynamics of progression of coronary atherosclerosis studied in 168 medically treated patients who underwent coronary arteriography three times. , 1989, American heart journal.
[89] R. Cotran,et al. Identification of an inducible endothelial-leukocyte adhesion molecule. , 1987, Proceedings of the National Academy of Sciences of the United States of America.
[90] P. Libby,et al. Inducible interleukin-1 gene expression in human vascular smooth muscle cells. , 1986, The Journal of clinical investigation.
[91] P. Libby,et al. Endotoxin and tumor necrosis factor induce interleukin-1 gene expression in adult human vascular endothelial cells. , 1986, The American journal of pathology.
[92] G. Bondjers,et al. Regional Accumulations of T Cells, Macrophages, and Smooth Muscle Cells in the Human Atherosclerotic Plaque , 1986, Arteriosclerosis.
[93] H. Waldmann,et al. Identification of macrophages and smooth muscle cells in human atherosclerosis using monoclonal antibodies , 1985, The Journal of pathology.
[94] R. Ross,et al. Studies of Hypercholesterolemia in the Nonhuman Primate: I. Changes that Lead to Fatty Streak Formation , 1984, Arteriosclerosis.
[95] M. Karnovsky. Metchnikoff in Messina: a century of studies on phagocytosis. , 1981, The New England journal of medicine.
[96] R. Ross,et al. Hyperlipidemia and atherosclerosis. , 1976, Science.
[97] R. Ross,et al. The pathogenesis of atherosclerosis (second of two parts). , 1976, The New England journal of medicine.
[98] R. Ross,et al. The pathogenesis of atherosclerosis (first of two parts). , 1976, The New England journal of medicine.
[99] H. Florey,et al. Changes in the endothelium of the aorta and the behaviour of macrophages in experimental atheroma of rabbits. , 1958, The Journal of pathology and bacteriology.