Anti‐ischemic effects of inotropic agents in experimental right ventricular infarction
暂无分享,去创建一个
R. Rossaint | P. Steendijk | J. Baumert | M. Hein | A. Roehl | K. Scherer | A. B. ROEHL | Anna B. Roehl | R. Rossaint
[1] R. Rossaint,et al. Establishment of a porcine right ventricular infarction model for cardioprotective actions of xenon and isoflurane , 2008, Acta anaesthesiologica Scandinavica.
[2] A. Domínguez-Rodríguez,et al. Effects of levosimendan versus dobutamine on long-term survival of patients with cardiogenic shock after primary coronary angioplasty. , 2008, International journal of cardiology.
[3] M. Miyazaki,et al. 6-[4-(1-Cyclohexyl-1H-tetrazol-5-yl)butoxy]-3,4-dihydro-2-(1H)quinolinone (Cilostazol), a Phosphodiesterase Type 3 Inhibitor, Reduces Infarct Size via Activation of Mitochondrial Ca2+-Activated K+ Channels in Rabbit Hearts , 2008, Journal of Pharmacology and Experimental Therapeutics.
[4] J. Spear,et al. beta1-Adrenoreceptor activation contributes to ischemia-reperfusion damage as well as playing a role in ischemic preconditioning. , 2007, American journal of physiology. Heart and circulatory physiology.
[5] L. Algotsson,et al. Levosimendan cardioprotection reduces the metabolic response during temporary regional coronary occlusion in an open chest pig model , 2007, Acta anaesthesiologica Scandinavica.
[6] M. Maeng,et al. Effects of Levosimendan on Myocardial Infarct Size and Hemodynamics in a Closed-Chest Porcine Ischemia–Reperfusion Model , 2006, Cardiovascular Drugs and Therapy.
[7] H. Sakamoto,et al. Effects of olprinone on myocardial ischemia-reperfusion injury in dogs. , 2006, The Journal of veterinary medical science.
[8] E. Berenshtein,et al. Flumazenil Mimics whereas Midazolam Abolishes Ischemic Preconditioning in a Rabbit Heart Model of Ischemia–Reperfusion , 2006, Anesthesiology.
[9] J. Vinten-johansen,et al. Postconditioning: reduction of reperfusion-induced injury. , 2006, Cardiovascular research.
[10] K. Ropella,et al. Ischemia-reperfusion injury changes the dynamics of Ca2+-contraction coupling due to inotropic drugs in isolated hearts. , 2006, Journal of applied physiology.
[11] I. Paraskevaidis,et al. Anti-inflammatory and anti-apoptotic effects of levosimendan in decompensated heart failure: a novel mechanism of drug-induced improvement in contractile performance of the failing heart. , 2005, Current medicinal chemistry. Cardiovascular and hematological agents.
[12] J. Goldstein. Pathophysiology and management of right heart ischemia. , 2002, Journal of the American College of Cardiology.
[13] M. Hori,et al. Cardioprotective Effect Afforded by Transient Exposure to Phosphodiesterase III Inhibitors: The Role of Protein Kinase A and p38 Mitogen-Activated Protein Kinase , 2001, Circulation.
[14] W. Schlack,et al. Ketamine, but Not S (+)-ketamine, Blocks Ischemic Preconditioning in Rabbit Hearts In Vivo , 2001, Anesthesiology.
[15] E. Pehkonen,et al. Beneficial effects of ischemic preconditioning on right ventricular function after coronary artery bypass grafting. , 2000, The Annals of thoracic surgery.
[16] H. Majewski,et al. Amrinone reduces ischaemia-reperfusion injury in rat heart. , 2000, European journal of pharmacology.
[17] P. Pagel,et al. Levosimendan, a New Positive Inotropic Drug, Decreases Myocardial Infarct Size via Activation of KATP Channels , 2000, Anesthesia and analgesia.
[18] J. Vinten-johansen,et al. The role of neutrophils in myocardial ischemia-reperfusion injury. , 1999, Cardiovascular research.
[19] L. Opie,et al. Levosimendan: effects of a calcium sensitizer on function and arrhythmias and cyclic nucleotide levels during ischemia/reperfusion in the Langendorff-perfused guinea pig heart. , 1999, The Journal of pharmacology and experimental therapeutics.
[20] L. Agati. Microvascular integrity after reperfusion therapy. , 1999, American heart journal.
[21] W. O’Neill,et al. Effect of reperfusion on biventricular function and survival after right ventricular infarction. , 1998, The New England journal of medicine.
[22] Division on Earth. Guide for the Care and Use of Laboratory Animals , 1996 .
[23] W. Herzog,et al. Timing of magnesium therapy affects experimental infarct size. , 1995, Circulation.
[24] A. Branzi,et al. Limitation of Myocardial Infarct Size by Nicorandil After Sustained Ischemia in Pigs , 1995, Journal of cardiovascular pharmacology.
[25] B. Lucchesi,et al. Cardioprotective effects of heparin or N-acetylheparin in an in vivo model of myocardial ischaemic and reperfusion injury. , 1995, Cardiovascular research.
[26] W. Klaus,et al. A quantitative comparison of functional and anti‐ischaemic effects of the phosphodiesterase‐inhibitors, amrinone, milrinone and levosimendan in rabbit isolated hearts , 1994, British journal of pharmacology.
[27] J. Moran,et al. Long-term amiodarone administration protects against global myocardial ischemia. , 1990, The Annals of thoracic surgery.
[28] L. Becker,et al. Neutrophil depletion limited to reperfusion reduces myocardial infarct size after 90 minutes of ischemia. Evidence for neutrophil-mediated reperfusion injury. , 1989, Circulation.
[29] K. Mullane,et al. Myeloperoxidase activity as a quantitative assessment of neutrophil infiltration into ischemic myocardium. , 1985, Journal of pharmacological methods.
[30] M. Fishbein,et al. Early phase acute myocardial infarct size quantification: validation of the triphenyl tetrazolium chloride tissue enzyme staining technique. , 1981, American heart journal.
[31] R. Fryer,et al. Comparative effects of levosimendan, OR-1896, OR-1855, dobutamine, and milrinone on vascular resistance, indexes of cardiac function, and O2 consumption in dogs. , 2008, American journal of physiology. Heart and circulatory physiology.
[32] T. Miura,et al. Limitation of myocardial infarct size in the clinical setting: current status and challenges in translating animal experiments into clinical therapy , 2008, Basic Research in Cardiology.
[33] M. Zioga,et al. Dopamine mimics the cardioprotective effect of ischemic preconditioning via activation of alpha1-adrenoceptors in the isolated rat heart. , 2006, Physiological research.
[34] M. Entman,et al. The inflammatory response in myocardial infarction. , 2002, Cardiovascular research.