In situ detection of HER2:HER2 and HER2:HER3 protein–protein interactions demonstrates prognostic significance in early breast cancer
暂无分享,去创建一个
J. Bartlett | D. Cameron | E. Mallon | C. Twelves | C. Cunningham | Jeremy S. Thomas | A. Munro | M. Spears | K. Taylor | J. Thomas | Carrie A Cunningham
[1] A. Feuchtinger,et al. In situ quantification of HER2–protein tyrosine kinase 6 (PTK6) protein–protein complexes in paraffin sections from breast cancer tissues , 2010, British Journal of Cancer.
[2] C. Sotiriou,et al. Quantitation of HER2 Expression or HER2:HER2 Dimers and Differential Survival in a Cohort of Metastatic Breast Cancer Patients Carefully Selected for Trastuzumab Treatment Primarily by FISH , 2009, Diagnostic molecular pathology : the American journal of surgical pathology, part B.
[3] R. Prescott,et al. Type 1 receptor tyrosine kinase profiles identify patients with enhanced benefit from anthracyclines in the BR9601 adjuvant breast cancer chemotherapy trial. , 2008, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[4] J. Bartlett,et al. Type I receptor tyrosine kinases as predictive or prognostic markers in early breast cancer. , 2008, Biomarkers in medicine.
[5] U. Landegren,et al. Characterizing proteins and their interactions in cells and tissues using the in situ proximity ligation assay. , 2008, Methods.
[6] L. Goldstein,et al. Epirubicin and Cyclophosphamide, Methotrexate, and Fluorouracil as Adjuvant Therapy for Early Breast Cancer , 2007 .
[7] Hanlee P. Ji,et al. Multiplexed protein detection by proximity ligation for cancer biomarker validation , 2007, Nature Methods.
[8] U. Landegren,et al. Direct observation of individual endogenous protein complexes in situ by proximity ligation , 2006, Nature Methods.
[9] M. Ellis,et al. Can Molecular Markers Predict When to Implement Treatment With Aromatase Inhibitors in Invasive Breast Cancer , 2006 .
[10] J. Bartlett,et al. The ERBB4/HER4 intracellular domain 4ICD is a BH3-only protein promoting apoptosis of breast cancer cells. , 2006, Cancer research.
[11] T. Kirkegaard,et al. Observer variation in immunohistochemical analysis of protein expression, time for a change? , 2006, Histopathology.
[12] E. Tokunaga,et al. Akt is frequently activated in HER2/neu‐positive breast cancers and associated with poor prognosis among hormone‐treated patients , 2006, International journal of cancer.
[13] Ryuji Kobayashi,et al. Insulin-like growth factor-I receptor/human epidermal growth factor receptor 2 heterodimerization contributes to trastuzumab resistance of breast cancer cells. , 2005, Cancer research.
[14] J. Bartlett,et al. AKT activation predicts outcome in breast cancer patients treated with tamoxifen , 2005, The Journal of pathology.
[15] I. Ellis,et al. Expression and co-expression of the members of the epidermal growth factor receptor (EGFR) family in invasive breast carcinoma , 2004, British Journal of Cancer.
[16] J. R. Reeves,et al. Outcome and human epidermal growth factor receptor (HER) 1–4 status in invasive breast carcinomas with proliferation indices evaluated by bromodeoxyuridine labelling , 2004, Breast Cancer Research.
[17] K. Grigor,et al. Androgen receptor gene amplification and protein expression in hormone refractory prostate cancer , 2003, British Journal of Cancer.
[18] F. Maurer,et al. The ErbB2/ErbB3 heterodimer functions as an oncogenic unit: ErbB2 requires ErbB3 to drive breast tumor cell proliferation , 2003, Proceedings of the National Academy of Sciences of the United States of America.
[19] J. R. Reeves,et al. Expression of the HER1–4 family of receptor tyrosine kinases in breast cancer , 2003, The Journal of pathology.
[20] D. Lauffenburger,et al. Quantitative Analysis of HER2-mediated Effects on HER2 and Epidermal Growth Factor Receptor Endocytosis , 2003, Journal of Biological Chemistry.
[21] A. Citri,et al. The deaf and the dumb: the biology of ErbB-2 and ErbB-3. , 2003, Experimental cell research.
[22] F. Maurer,et al. The ErbB 2 ErbB 3 heterodimer functions as an oncogenic unit : ErbB 2 requires ErbB 3 to drive breast tumor cell proliferation , 2003, Nature Reviews Cancer.
[23] U. Landegren,et al. Protein detection using proximity-dependent DNA ligation assays , 2002, Nature Biotechnology.
[24] J. Baselga,et al. Unraveling resistance to trastuzumab (Herceptin): insulin-like growth factor-I receptor, a new suspect. , 2001, Journal of the National Cancer Institute.
[25] J. R. Reeves,et al. Evaluating HER2 amplification and overexpression in breast cancer , 2001, The Journal of pathology.
[26] H. Wiley,et al. ErbB-2 Amplification Inhibits Down-regulation and Induces Constitutive Activation of Both ErbB-2 and Epidermal Growth Factor Receptors* , 1999, The Journal of Biological Chemistry.
[27] S. Ethier,et al. Phosphatidylinositol 3-kinase recruitment by p185erbB-2 and erbB-3 is potently induced by neu differentiation factor/heregulin during mitogenesis and is constitutively elevated in growth factor-independent breast carcinoma cells with c-erbB-2 gene amplification. , 1996, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.