Neuroprotection by aminoguanidine after lateral fluid-percussive brain injury in rats: a combined magnetic resonance imaging, histopathologic and functional study

[1]  S. Kraydieh,et al.  Temporal and segmental distribution of constitutive and inducible nitric oxide synthases after traumatic spinal cord injury: effect of aminoguanidine treatment. , 2002, Journal of neurotrauma.

[2]  T. Uliasz,et al.  SIN‐1‐induced cytotoxicity in mixed cortical cell culture: peroxynitrite‐dependent and ‐independent induction of excitotoxic cell death , 2001, Journal of neurochemistry.

[3]  Steven C. R. Williams,et al.  Neuroprotective effect of aminoguanidine on transient focal ischaemia in the rat brain , 2001, Brain Research.

[4]  A. Deckel Nitric oxide and nitric oxide synthase in Huntington's disease , 2001, Journal of neuroscience research.

[5]  N. Marklund,et al.  Paradoxical Increase in Neuronal DNA Fragmentation after Neuroprotective Free Radical Scavenger Treatment in Experimental Traumatic Brain Injury , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[6]  I. Whittle,et al.  Nitric oxide and glioma: a target for novel therapy? , 2001, British journal of neurosurgery.

[7]  H. Iwata,et al.  Nitric oxide via macrophage iNOS induces apoptosis following traumatic spinal cord injury. , 2000, Brain research. Molecular brain research.

[8]  D. Graham,et al.  Apoptosis after traumatic brain injury. , 2000, Journal of neurotrauma.

[9]  E. Ling,et al.  Changes in apoptosis-related protein (p53, Bax, Bcl-2 and Fos) expression with DNA fragmentation in the central nervous system in rats after closed head injury , 2000, Neuroscience Letters.

[10]  A. A. Parsons,et al.  Functional assessments in mice and rats after focal stroke , 2000, Neuropharmacology.

[11]  H. Koprowski,et al.  Uric acid, a peroxynitrite scavenger, inhibits CNS inflammation, blood–CNS barrier permeability changes, and tissue damage in a mouse model of multiple sclerosis , 2000, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[12]  K. Takeda,et al.  Cytokine-induced nitric oxide production inhibits mitochondrial energy production and impairs contractile function in rat cardiac myocytes. , 2000, Journal of the American College of Cardiology.

[13]  A. Dávalos,et al.  Nitric oxide-related brain damage in acute ischemic stroke. , 2000, Stroke.

[14]  Simon C Watkins,et al.  Caspase‐3 Mediated Neuronal Death After Traumatic Brain Injury in Rats , 2000, Journal of neurochemistry.

[15]  B. Johansson,et al.  The rotating pole test: evaluation of its effectiveness in assessing functional motor deficits following experimental head injury in the rat , 2000, Journal of Neuroscience Methods.

[16]  B. Halliwell,et al.  Nitric oxide and peroxynitrite. The ugly, the uglier and the not so good: a personal view of recent controversies. , 1999, Free radical research.

[17]  T. Akaike,et al.  Direct Evidence of in Vivo Nitric Oxide Production and Inducible Nitric Oxide Synthase mRNA Expression in the Brain of Living Rat during Experimental Meningitis , 1999, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[18]  B. Nilsson Biological effects of aminoguanidine: An update , 1999, Inflammation Research.

[19]  Simon C Watkins,et al.  Inducible nitric oxide synthase is an endogenous neuroprotectant after traumatic brain injury in rats and mice , 1999 .

[20]  B. Pike,et al.  Temporal Profile of Apoptotic-like Changes in Neurons and Astrocytes Following Controlled Cortical Impact Injury in the Rat , 1999, Experimental Neurology.

[21]  T. Kumanishi,et al.  Role of Nitric Oxide in Pathogenesis Underlying Ischemic Cerebral Damage , 1999, Cellular and Molecular Neurobiology.

[22]  R. Busto,et al.  Inducible nitric oxide synthase expression after traumatic brain injury and neuroprotection with aminoguanidine treatment in rats. , 1998, Neurosurgery.

[23]  S. Rees,et al.  Depletion of nitric oxide synthase-containing neurons in the diabetic retina: reversal by aminoguanidine , 1998, Diabetologia.

[24]  W. Pryor,et al.  Oxidative chemistry of nitric oxide: the roles of superoxide, peroxynitrite, and carbon dioxide. , 1998, Free radical biology & medicine.

[25]  A. T. Demiryürek,et al.  Comparison of antioxidant activities of aminoguanidine, methylguanidine and guanidine by luminol‐enhanced chemiluminescence , 1998, British journal of pharmacology.

[26]  Carlos Portera-Cailliau,et al.  Neurodegeneration in Excitotoxicity, Global Cerebral Ischemia, and Target Deprivation: A Perspective on the Contributions of Apoptosis and Necrosis , 1998, Brain Research Bulletin.

[27]  T. Akaike,et al.  Free Radicals in Viral Pathogenesis: Molecular Mechanisms Involving Superoxide and NO , 1998, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

[28]  Fangyi Zhang,et al.  Delayed Reduction of Ischemic Brain Injury and Neurological Deficits in Mice Lacking the Inducible Nitric Oxide Synthase Gene , 1997, The Journal of Neuroscience.

[29]  A. Yakovlev,et al.  Activation of CPP32-Like Caspases Contributes to Neuronal Apoptosis and Neurological Dysfunction after Traumatic Brain Injury , 1997, The Journal of Neuroscience.

[30]  Joseph S. Beckman,et al.  Widespread Peroxynitrite-Mediated Damage in Alzheimer’s Disease , 1997, The Journal of Neuroscience.

[31]  M. Freund,et al.  Long-term follow up of children with head injuries-classified as “good recovery” using the Glasgow Outcome Scale: neurological, neuropsychological and magnetic resonance imaging results , 1997, European Journal of Pediatrics.

[32]  P. Dash,et al.  Apoptotic morphology of dentate gyrus granule cells following experimental cortical impact injury in rats: possible role in spatial memory deficits , 1996, Brain Research.

[33]  D. Hovda,et al.  Fluid percussion brain injury in the developing and adult rat: a comparative study of mortality, morphology, intracranial pressure and mean arterial blood pressure. , 1996, Brain research. Developmental brain research.

[34]  M. Ross,et al.  Aminoguanidine ameliorates and L-arginine worsens brain damage from intraluminal middle cerebral artery occlusion. , 1996, Stroke.

[35]  A. Weintrob,et al.  Potential role of nitric oxide in the pathophysiology of experimental bacterial meningitis in rats , 1995, Infection and immunity.

[36]  R. Hayes,et al.  Time course of increased vulnerability of cholinergic neurotransmission following traumatic brain injury in the rat , 1995, Behavioural Brain Research.

[37]  H. Levin,et al.  Prediction of recovery from traumatic brain injury. , 1995, Journal of neurotrauma.

[38]  V. Dawson NITRIC OXIDE: ROLE IN NEUROTOXICITY , 1995, Clinical and experimental pharmacology & physiology.

[39]  E. Shohami,et al.  Long-term effect of HU-211, a novel non-competitive NMDA antagonist, on motor and memory functions after closed head injury in the rat , 1995, Brain Research.

[40]  A. Perin,et al.  Diamine oxidase in relation to diamine and polyamine metabolism , 1994, Agents and Actions.

[41]  P. Riederer,et al.  Oxygen Free Radicals and Neurodegeneration in Parkinson's Disease: A Role for Nitric Oxide a , 1994, Annals of the New York Academy of Sciences.

[42]  S. J. Sullivan,et al.  Agreement of classification decisions using two measures of motor co-ordination in persons with a traumatic brain injury. , 1994, Brain injury.

[43]  D. Dickson,et al.  Microglial involvement in the acquired immunodeficiency syndrome (AIDS). , 1994, Neuropathology and applied neurobiology.

[44]  Joseph Loscalzo,et al.  A redox-based mechanism for the neuroprotective and neurodestructive effects of nitric oxide and related nitroso-compounds , 1993, Nature.

[45]  M. Mcdaniel,et al.  Selective inhibition of inducible nitric oxide synthase by aminoguanidine. , 1993, Methods in enzymology.

[46]  S. Snyder,et al.  Nitric oxide stimulates auto-ADP-ribosylation of glyceraldehyde-3-phosphate dehydrogenase. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[47]  L. Ignarro,et al.  Nitric oxide-mediated neuronal injury in multiple sclerosis. , 1992, Medical hypotheses.

[48]  S H Snyder,et al.  A novel neuronal messenger molecule in brain: The free radical, nitric oxide , 1992, Annals of neurology.

[49]  S. Tannenbaum,et al.  DNA damage and mutation in human cells exposed to nitric oxide in vitro. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[50]  C. Nathan,et al.  Inhibition of tumor cell ribonucleotide reductase by macrophage-derived nitric oxide , 1991, The Journal of experimental medicine.

[51]  L. Noble,et al.  Traumatic brain injury in the rat: Characterization of a lateral fluid-percussion model , 1989, Neuroscience.

[52]  N. Plesnila,et al.  Attenuation of secondary lesion growth in the brain after trauma by selective inhibition of the inducible NO-synthase. , 2000, Acta neurochirurgica. Supplement.

[53]  A. Faden Pharmacological treatment of central nervous system trauma. , 1996, Pharmacology & toxicology.

[54]  J. Trojanowski,et al.  Evidence of apoptotic cell death after experimental traumatic brain injury in the rat. , 1995, The American journal of pathology.

[55]  D. J. Wolff,et al.  Aminoguanidine is an isoform-selective, mechanism-based inactivator of nitric oxide synthase. , 1995, Archives of biochemistry and biophysics.

[56]  C. Iadecola,et al.  Inhibition of inducible nitric oxide synthase ameliorates cerebral ischemic damage. , 1995, The American journal of physiology.

[57]  D. Stein,et al.  Unilateral cortical contusion injury in the rat: vascular disruption and temporal development of cortical necrosis. , 1993, Journal of neurotrauma.