OGT and OGA expression in postmenopausal skeletal muscle associates with hormone replacement therapy and muscle cross-sectional area

[1]  S. A. Marsh,et al.  Exercise and diabetes have opposite effects on the assembly and O-GlcNAc modification of the mSin3A/HDAC1/2 complex in the heart , 2013, Cardiovascular Diabetology.

[2]  J. Shearer,et al.  Exercise training mitigates aberrant cardiac protein O-GlcNAcylation in streptozotocin-induced diabetic mice. , 2013, Life sciences.

[3]  A. Mehta,et al.  Ontogenic development of cardiomyocytes derived from transgene-free human induced pluripotent stem cells and its homology with human heart. , 2013, Life sciences.

[4]  D. Belke Swim-exercised mice show a decreased level of protein O-GlcNAcylation and expression of O-GlcNAc transferase in heart. , 2011, Journal of applied physiology.

[5]  Ping Huang,et al.  Muscle-specific overexpression of NCOATGK, splice variant of O-GlcNAcase, induces skeletal muscle atrophy. , 2011, American journal of physiology. Cell physiology.

[6]  Sreenath S. Andrali,et al.  Modulation of transcription factor function by O-GlcNAc modification. , 2010, Biochimica et biophysica acta.

[7]  H. Suominen,et al.  Power training and postmenopausal hormone therapy affect transcriptional control of specific co-regulated gene clusters in skeletal muscle , 2010, AGE.

[8]  T. Lefebvre,et al.  Dysregulation of the nutrient/stress sensor O-GlcNAcylation is involved in the etiology of cardiovascular disorders, type-2 diabetes and Alzheimer's disease. , 2010, Biochimica et biophysica acta.

[9]  V. Montel,et al.  O-GlcNAcylation, an original modulator of contractile activity in striated muscle , 2009, Journal of Muscle Research and Cell Motility.

[10]  I. Dionne,et al.  Changes in muscle mass and strength after menopause. , 2009, Journal of musculoskeletal & neuronal interactions.

[11]  V. Tiffreau,et al.  Phenotypical transitions and Ca2+ activation properties in human muscle fibers: effects of a 60-day bed rest and countermeasures. , 2009, Journal of applied physiology.

[12]  W. V. So,et al.  Phosphoinositide signalling links O-GlcNAc transferase to insulin resistance , 2008, Nature.

[13]  R. Marchase,et al.  Aging leads to increased levels of protein O-linked N-acetylglucosamine in heart, aorta, brain and skeletal muscle in Brown-Norway rats , 2008, Biogerontology.

[14]  H. Suominen,et al.  Muscular transcriptome in postmenopausal women with or without hormone replacement. , 2007, Rejuvenation research.

[15]  G. Hart,et al.  O-GlcNAc modification in diabetes and Alzheimer's disease. , 2007, Molecular bioSystems.

[16]  W. Yang,et al.  Modification of p53 with O-linked N-acetylglucosamine regulates p53 activity and stability , 2006, Nature Cell Biology.

[17]  H. Suominen,et al.  The effect of hormone replacement therapy and/or exercise on skeletal muscle attenuation in postmenopausal women: a yearlong intervention , 2005, Clinical physiology and functional imaging.

[18]  G. Cartee,et al.  Prolonged incubation in PUGNAc results in increased protein O-Linked glycosylation and insulin resistance in rat skeletal muscle. , 2004, Diabetes.

[19]  Xiaoyong Yang,et al.  O-GlcNAc Modification Is an Endogenous Inhibitor of the Proteasome , 2003, Cell.

[20]  H. Suominen,et al.  Effects of hormone replacement therapy and high-impact physical exercise on skeletal muscle in post-menopausal women: a randomized placebo-controlled study. , 2001, Clinical science.

[21]  G. Butler-Browne,et al.  Clones of human satellite cells can express in vitro both fast and slow myosin heavy chains. , 1994, Developmental biology.

[22]  G. Hart,et al.  O-linked N-acetylglucosamine and cancer: messages from the glycosylation of c-Myc. , 2001, Advances in experimental medicine and biology.