Pth1r Signal in Gli1+ Cells Maintains Postnatal Cranial Base Synchondrosis
暂无分享,去创建一个
M. Kogo | D. Okuzaki | T. Aikawa | M. Fujiwara | K. Amano | S. Iida | Y. Kitaoka | S. Kato
[1] R. Franceschi,et al. Cranial Base Synchondrosis: Chondrocytes at the Hub , 2022, International journal of molecular sciences.
[2] W. Ono,et al. Cranial Base Synchondrosis Lacks PTHrP-Expressing Column-Forming Chondrocytes , 2022, International journal of molecular sciences.
[3] Y. Mishina,et al. The Posterior Part Influences the Anterior Part of the Mouse Cranial Base Development , 2021, JBMR plus.
[4] N. Funato. New Insights Into Cranial Synchondrosis Development: A Mini Review , 2020, Frontiers in Cell and Developmental Biology.
[5] R. Xu,et al. Role of PTH1R Signaling in Prx1+ Mesenchymal Progenitors during Eruption , 2020, Journal of dental research.
[6] D. Srivastava,et al. TBX1 Regulates Chondrocyte Maturation in the Spheno-occipital Synchondrosis , 2020, Journal of dental research.
[7] W. Ono,et al. Mesenchymal Progenitor Regulation of Tooth Eruption: A View from PTHrP , 2020, Journal of dental research.
[8] Y. Mishina,et al. Developmental Regulation of the Growth Plate and Cranial Synchondrosis , 2016, Journal of dental research.
[9] B. Lanske,et al. Conditional Deletion of Indian Hedgehog in Limb Mesenchyme Results in Complete Loss of Growth Plate Formation but Allows Mature Osteoblast Differentiation , 2015, Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research.
[10] C. Loomis,et al. Regulation of cranial morphogenesis and cell fate at the neural crest-mesoderm boundary by engrailed 1 , 2012, Development.
[11] S. Mackem,et al. Parathyroid hormone/parathyroid hormone-related protein receptor signaling is required for maintenance of the growth plate in postnatal life , 2010, Proceedings of the National Academy of Sciences.
[12] R. Baron,et al. Zfp521 is a target gene and key effector of parathyroid hormone-related peptide signaling in growth plate chondrocytes. , 2010, Developmental cell.
[13] Buer Song,et al. Conditional Kif3a ablation causes abnormal hedgehog signaling topography, growth plate dysfunction, and excessive bone and cartilage formation during mouse skeletogenesis , 2007, Development.
[14] M. Razzaque,et al. Conditional deletion of Indian hedgehog from collagen type 2α1‐expressing cells results in abnormal endochondral bone formation , 2005, The Journal of pathology.
[15] A. Joyner,et al. Dynamic Changes in the Response of Cells to Positive Hedgehog Signaling during Mouse Limb Patterning , 2004, Cell.
[16] D. Rifkin,et al. Bone defects in latent TGF-beta binding protein (Ltbp)-3 null mice; a role for Ltbp in TGF-beta presentation. , 2002, The Journal of endocrinology.
[17] B. Lanske,et al. PTHrP and Indian hedgehog control differentiation of growth plate chondrocytes at multiple steps. , 2002, Development.
[18] B. Lanske,et al. PTH/PTHrP Receptor in Early Development and Indian Hedgehog--Regulated Bone Growth , 1996, Science.
[19] H. Jüppner,et al. A constitutively active mutant PTH-PTHrP receptor in Jansen-type metaphyseal chondrodysplasia. , 1995, Science.
[20] M. Freeman,et al. A G protein-linked receptor for parathyroid hormone and parathyroid hormone-related peptide. , 1991, Science.
[21] B. Olsen,et al. Col2-Cre and tamoxifen-inducible Col2-CreER target different cell populations in the knee joint. , 2016, Osteoarthritis and cartilage.