Identification of MNDA as a new marker for nodal marginal zone lymphoma
暂无分享,去创建一个
J. Cigudosa | L. Maestre | L. Sánchez-Verde | S. Montes-Moreno | J. Martínez-Torrecuadrada | M. Mollejo | G. Kanellis | M. Piris | A. Arribas | G. Roncador | R. Pajares | M. Martín | Y. Campos-Martín | J. L. R. Gonzalez | J. González
[1] J. Cigudosa,et al. Splenic Follicular Lymphoma: Clinicopathologic Characteristics of a Series of 32 Cases , 2009, The American journal of surgical pathology.
[2] J. Armitage,et al. ATM, CTLA4, MNDA, and HEM1 in High versus Low CD38–Expressing B-Cell Chronic Lymphocytic Leukemia , 2007, Clinical Cancer Research.
[3] H. Tagawa,et al. CD10-MUM1+ follicular lymphoma lacks BCL2 gene translocation and shows characteristic biologic and clinical features. , 2007, Blood.
[4] H. Tagawa,et al. CD 10 (-) MUM 1 ( + ) follicular lymphoma lacks BCL 2 gene translocation and shows characteristic biological and clinical features , 2006 .
[5] B. Jungnickel,et al. Human splenic marginal zone B cells lack expression of activation‐induced cytidine deaminase , 2005, European journal of immunology.
[6] R. Johnstone,et al. The HIN-200 family: more than interferon-inducible genes? , 2005, Experimental cell research.
[7] Nerea Martínez,et al. Expression of the NF‐κB targets BCL2 and BIRC5/Survivin characterizes small B‐cell and aggressive B‐cell lymphomas, respectively , 2005, The Journal of pathology.
[8] S. Pillai,et al. Marginal zone B cells. , 2005, Annual review of immunology.
[9] L. Staudt,et al. Human blood IgM "memory" B cells are circulating splenic marginal zone B cells harboring a prediversified immunoglobulin repertoire. , 2004, Blood.
[10] M. Piris,et al. Transcriptional Response of T Cells to IFN-α: Changes Induced in IFN-α-Sensitive and Resistant Cutaneous T Cell Lymphoma , 2004 .
[11] D. Mason,et al. Immunofluorescent and FISH Analysis of Skin Biopsies , 2004, The American Journal of dermatopathology.
[12] M. Piris,et al. Transcriptional response of T cells to IFN-alpha: changes induced in IFN-alpha-sensitive and resistant cutaneous T cell lymphoma. , 2004, Journal of interferon & cytokine research : the official journal of the International Society for Interferon and Cytokine Research.
[13] S. Pileri,et al. Phenotype and genotype of interfollicular large B cells, a subpopulation of lymphocytes often with dendritic morphology. , 2003, Blood.
[14] A. Godzik,et al. Homology modeling provides insights into the binding mode of the PAAD/DAPIN/pyrin domain, a fourth member of the CARD/DD/DED domain family , 2003, Protein science : a publication of the Protein Society.
[15] Juan F. García,et al. Mycosis fungoides shows concurrent deregulation of multiple genes involved in the TNF signaling pathway: an expression profile study. , 2003, Blood.
[16] Juan F. García,et al. Nodal Marginal Zone Lymphoma: A Heterogeneous Tumor: A Comprehensive Analysis of a Series of 27 Cases , 2003, The American journal of surgical pathology.
[17] Ruchi M. Newman,et al. Comparative analysis of apoptosis and inflammation genes of mice and humans. , 2003, Genome research.
[18] E. Campo,et al. Analysis of the IgV(H) somatic mutations in splenic marginal zone lymphoma defines a group of unmutated cases with frequent 7q deletion and adverse clinical course. , 2002, Blood.
[19] O. Kallioniemi,et al. Tissue microarray technology for high-throughput molecular profiling of cancer. , 2001, Human molecular genetics.
[20] E. Dahl,et al. The DAPIN family: a novel domain links apoptotic and interferon response proteins. , 2001, Trends in biochemical sciences.
[21] J C Reed,et al. PAAD - a new protein domain associated with apoptosis, cancer and autoimmune diseases. , 2001, Trends in biochemical sciences.
[22] D. Choubey,et al. Differential induction of the 200-family proteins in Daudi Burkitt's lymphoma cells by interferon-alpha. , 2000, Journal of biological regulators and homeostatic agents.
[23] A. Fischer,et al. Activation-Induced Cytidine Deaminase (AID) Deficiency Causes the Autosomal Recessive Form of the Hyper-IgM Syndrome (HIGM2) , 2000, Cell.
[24] T. Honjo,et al. Class Switch Recombination and Hypermutation Require Activation-Induced Cytidine Deaminase (AID), a Potential RNA Editing Enzyme , 2000, Cell.
[25] D. Mason,et al. Double immunofluorescence labelling of routinely processed paraffin sections , 2000, The Journal of pathology.
[26] R. Miranda,et al. Immunocytochemical analysis of MNDA in tissue sections and sorted normal bone marrow cells documents expression only in maturing normal and neoplastic myelomonocytic cells and a subset of normal and neoplastic B lymphocytes. , 1999, Human pathology.
[27] J. Trapani,et al. Transcription and Growth Regulatory Functions of the HIN-200 Family of Proteins , 1999, Molecular and Cellular Biology.
[28] R. Briggs,et al. Expression of human myeloid cell nuclear differentiation antigen (MNDA) in acute leukemias. , 1990, Leukemia research.
[29] L. Hnilica,et al. Properties of a nuclear protein marker of human myeloid cell differentiation. , 1986, The Journal of biological chemistry.
[30] R C Briggs,et al. Nonhistone protein antigen profiles of five leukemic cell lines reflect the extent of myeloid differentiation. , 1984, Blood.