Tumour necrosis factor-α enhances bradykinin-induced signal transduction via activation of Ras/Raf/MEK/MAPK in canine tracheal smooth muscle cells

[1]  Chih‐Chung Lin,et al.  Tumour necrosis factor‐α‐ and interleukin‐1β‐stimulated cell proliferation through activation of mitogen‐activated protein kinase in canine tracheal smooth muscle cells , 2000 .

[2]  G. Nixon,et al.  Tumour necrosis factor‐α activates a calcium sensitization pathway in guinea‐pig bronchial smooth muscle , 1999 .

[3]  S. Pyne,et al.  Platelet-derived-growth-factor stimulation of the p42/p44 mitogen-activated protein kinase pathway in airway smooth muscle: role of pertussis-toxin-sensitive G-proteins, c-Src tyrosine kinases and phosphoinositide 3-kinase. , 1999, The Biochemical journal.

[4]  E. Fleck,et al.  TNF-α–Induced Migration of Vascular Smooth Muscle Cells Is MAPK Dependent , 1999 .

[5]  R. Panettieri.  Cellular and molecular mechanisms regulating airway smooth muscle proliferation and cell adhesion molecule expression. , 1998, American journal of respiratory and critical care medicine.

[6]  U. Rapp,et al.  Cell cycle targets of Ras/Raf signalling , 1998, Oncogene.

[7]  K. Mehta,et al.  Differential Roles of Extracellular Signal-regulated Kinase-1/2 and p38MAPK in Interleukin-1β- and Tumor Necrosis Factor-α-induced Low Density Lipoprotein Receptor Expression in HepG2 Cells* , 1998, The Journal of Biological Chemistry.

[8]  S. Pyne,et al.  PDGF-stimulated cyclic AMP formation in airway smooth muscle: assessment of the roles of MAP kinase, cytosolic phospholipase A2, and arachidonate metabolites. , 1998, Cellular signalling.

[9]  J. Tocker,et al.  Pharmacological evidence for tumor necrosis factor as a mediator of allergic inflammation in the airways. , 1996, The Journal of pharmacology and experimental therapeutics.

[10]  C. Bronner,et al.  Activation of the TNF alpha-p55 receptor induces myocyte proliferation and modulates agonist-evoked calcium transients in cultured human tracheal smooth muscle cells. , 1996, American journal of respiratory cell and molecular biology.

[11]  J. Brown,et al.  G protein‐coupled receptors and signaling pathways regulating growth responses 1 , 1996, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[12]  A. Bridges,et al.  A synthetic inhibitor of the mitogen-activated protein kinase cascade. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[13]  D. Yates,et al.  Tumor necrosis factor-alpha increases airway responsiveness and sputum neutrophilia in normal human subjects. , 1995, American journal of respiratory and critical care medicine.

[14]  C. Marshall,et al.  Specificity of receptor tyrosine kinase signaling: Transient versus sustained extracellular signal-regulated kinase activation , 1995, Cell.

[15]  C. Bronner,et al.  Potentiation by tumour necrosis factor‐α of calcium signals induced by bradykinin and carbachol in human tracheal smooth muscle cells , 1995, British journal of pharmacology.

[16]  Chuen-Mao Yang,et al.  Pharmacological characterization of bradykinin receptors in canine cultured tracheal smooth muscle cells , 1995, British journal of pharmacology.

[17]  T. Harris,et al.  Tumor necrosis factor alpha modulates mitogenic responses of human cultured airway smooth muscle. , 1995, American journal of respiratory cell and molecular biology.

[18]  S. Powers,et al.  Ras-15A protein shares highly similar dominant-negative biological properties with Ras-17N and forms a stable, guanine-nucleotide resistant complex with CDC25 exchange factor. , 1994, Oncogene.

[19]  S. Luo,et al.  Bradykinin-stimulated calcium mobilization in cultured canine tracheal smooth muscle cells. , 1994, Cell calcium.

[20]  K. Blumer,et al.  Diversity in function and regulation of MAP kinase pathways. , 1994, Trends in biochemical sciences.

[21]  Chuen-Mao Yang,et al.  Bradykinin‐stimulated phosphoinositide metabolism in cultured canine tracheal smooth muscle cells , 1994, British journal of pharmacology.

[22]  C. Strader,et al.  Differential pharmacology of cloned human and mouse B2 bradykinin receptors. , 1994, Molecular pharmacology.

[23]  C. Marshall,et al.  A dominant-negative mutant of raf blocks mitogen-activated protein kinase activation by growth factors and oncogenic p21ras. , 1993, The Journal of biological chemistry.

[24]  R. Pauwels,et al.  The potential role of tumour necrosis factor a in asthma , 1993, Clinical and experimental allergy : journal of the British Society for Allergy and Clinical Immunology.

[25]  A. Inglot,et al.  Elevated release of tumor necrosis factor-alpha and interferon-gamma by bronchoalveolar leukocytes from patients with bronchial asthma. , 1993, The American review of respiratory disease.

[26]  B. Löwenberg,et al.  Involvement of tumor necrosis factor (TNF) receptors p55 and p75 in TNF responses of acute myeloid leukemia blasts in vitro , 1992 .

[27]  C. L. Johnson,et al.  The effect of TNFa on bradykinin receptor binding, phosphatidylinositol turnover and cell growth in human A431 epidermoid carcinoma cells. , 1992, Immunopharmacology.

[28]  P. Paré,et al.  A model of airway narrowing in asthma and in chronic obstructive pulmonary disease. , 1992, The American review of respiratory disease.

[29]  D. Broide,et al.  Cytokines in symptomatic asthma airways. , 1992, The Journal of allergy and clinical immunology.

[30]  J. O'flaherty,et al.  Tumor necrosis factor-alpha regulates expression of receptors for formyl-methionyl-leucyl-phenylalanine, leukotriene B4, and platelet-activating factor. Dissociation from priming in human polymorphonuclear neutrophils. , 1991, Journal of immunology.

[31]  J. Klein,et al.  Modulation of transmembrane signalling in HL-60 granulocytes by tumour necrosis factor-alpha. , 1991, The Biochemical journal.

[32]  J B Gibbs,et al.  Dominant inhibitory Ras mutants selectively inhibit the activity of either cellular or oncogenic Ras , 1991, Molecular and cellular biology.

[33]  M. Tanemura,et al.  Hyperreactive site in the airway tree of asthmatic patients revealed by thickening of bronchial muscles. A morphometric study. , 1990, The American review of respiratory disease.

[34]  N. Rhaleb,et al.  New selective bradykinin receptor antagonists and bradykinin B2 receptor characterization. , 1990, Trends in pharmacological sciences.

[35]  C. Armour,et al.  Relationship between smooth muscle volume and contractile response in airway tissue. Isometric versus isotonic measurement. , 1988, The Journal of pharmacology and experimental therapeutics.

[36]  M. Vadas,et al.  Recombinant human tumor necrosis factor-alpha. Regulation of N-formylmethionylleucylphenylalanine receptor affinity and function on human neutrophils. , 1988, The Journal of clinical investigation.

[37]  A. Gown,et al.  A smooth muscle-specific monoclonal antibody recognizes smooth muscle actin isozymes , 1985, The Journal of cell biology.

[38]  C. Cepko,et al.  Construction and applications of a highly transmissible murine retrovirus shuttle vector , 1984, Cell.

[39]  M. M. Bradford.  A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding. , 1976, Analytical biochemistry.