Effects of lidoflazine and mioflazine against potassium and veratrine induced shape changes in isolated rat cardiac myocytes

[1]  H. E. Keurs,et al.  Localization of calcium in skeletal and cardiac muscle , 1984, The Histochemical Journal.

[2]  A. Vleugels,et al.  Hypoxia increases potassium efflux from mammalian myocardium , 1976, Experientia.

[3]  Hideyuki Kobayashi,et al.  Modulation by ouabain and diphenylhydantoin of veratridine-induced 22Na influx and its relation to 45Ca influx and the secretion of catecholamines in cultured bovine adrenal medullary cells , 2004, Naunyn-Schmiedeberg's Archives of Pharmacology.

[4]  M. Borgers,et al.  Evaluation of protective effects of lidoflazine and mioflazine in cardiac ischemia , 1986 .

[5]  M. Borgers,et al.  Calcium-shifts in anoxic cardiac myocytes. A cytochemical study. , 1986, Journal of molecular and cellular cardiology.

[6]  P. Pauwels,et al.  Isolated rat cardiac myocytes as an experimental model to study calcium overload: the effect of calcium-entry blockers. , 1986, Life sciences.

[7]  U. Ravens,et al.  Responses to hypertonic solutions in guinea-pig atria: changes in action potentials, force of contraction and calcium content. , 1986, Journal of molecular and cellular cardiology.

[8]  J. W. Wei,et al.  Studies of [3H]nitrendipine binding and KCl-induced calcium uptake in rat cortical synaptosomes. , 1987, General pharmacology.

[9]  M. Noble,et al.  Mioflazine, a potentially protective drug against ischaemic damage: a study in dogs. , 1985, European heart journal.

[10]  M. Borgers,et al.  Sarcolemma-bound calcium. Its importance for cell viability. , 1985, Basic Research in Cardiology.

[11]  S. Govoni,et al.  Calcium Entry Blockers and Tissue Protection , 1984 .

[12]  P. Janssen,et al.  Cardioprotective effects of mioflazine during 1 h normothermic global ischaemia in the canine heart. , 1984, Cardiovascular research.

[13]  P. Poole‐Wilson,et al.  Calcium out of control. , 1984, Journal of molecular and cellular cardiology.

[14]  M. Borgers,et al.  Basic and clinical aspects of myocardial protection by calcium entry blockers , 1984 .

[15]  N. Severs,et al.  Isolated calcium-tolerant myocytes and the calcium paradox: an ultrastructural comparison. , 1983, European heart journal.

[16]  P. Poole‐Wilson,et al.  The Effects of Verapamil, Quiescence, and Cardioplegia on Calcium Exchange and Mechanical Function in Ischemic Rabbit Myocardium , 1982, Circulation research.

[17]  M. Lazdunski,et al.  Neurotoxins specific for the sodium channel stimulate calcium entry into neuroblastoma cells. , 1981, Biochemistry.

[18]  M. Borgers,et al.  Cardioprotective Effects of Lidoflazine During 1‐hour Normothermic Global Ischemia , 1981, Circulation.

[19]  R. Jennings,et al.  Lethal myocardial ischemic injury. , 1981, The American journal of pathology.

[20]  K. Shine.  Ionic events in ischemia and anoxia , 1981 .

[21]  D. Bers,et al.  The role of phospholipids in the Ca2+ binding of isolated cardiac sarcolemma. , 1980, Journal of molecular and cellular cardiology.

[22]  P. Poole‐Wilson,et al.  Hypoxia and calcium. , 1979, Journal of molecular and cellular cardiology.

[23]  M. Lazdunski,et al.  Analysis of molecular aspects of Na+ and Ca2+ uptakes by embryonic cardiac cells in culture. , 1977, The Journal of biological chemistry.

[24]  J. Gethmann,et al.  [The effect of the coronary dilator lidoflazine on myocardial adenosine metabolism in ischemia and anoxia]. , 1972, Arzneimittel-Forschung.

[25]  A. Jageneau,et al.  The cardiovascular pharmacology of lidoflazine. , 1970, Bruxelles medical.

[26]  H. Belle.  The disappearance of adenosine in blood. Effect of lidoflazine and other drugs. , 1970 .