Effect of sialyl Lewis(x) oligosaccharide on myocardial and cerebral injury in the pig.

[1]  D. Morse,et al.  Eicosanoid production from porcine neutrophils and platelets: differential production with various agonists. , 1997, The American journal of physiology.

[2]  E. Block,et al.  Molecular cloning, characterization and expression of a nitric oxide synthase from porcine pulmonary artery endothelial cells. , 1997, Comparative biochemistry and physiology. Part B, Biochemistry & molecular biology.

[3]  A. Buda,et al.  Combined inhibition of P-selectin and ICAM-1 reduces myocardial injury following ischemia and reperfusion. , 1996, The American journal of physiology.

[4]  J. Mayer,et al.  Blockade of selectin-mediated leukocyte adhesion improves postischemic function in lamb hearts. , 1996, The Annals of thoracic surgery.

[5]  W. Cohn,et al.  Cardiopulmonary bypass, myocardial management, and support techniques Changes in autonomic response of the cerebral circulation after normothermic extracorporeal circulation , 1996 .

[6]  A. Buda,et al.  Effects of a monoclonal antibody directed against P-selectin after myocardial ischemia and reperfusion. , 1996, The American journal of physiology.

[7]  W. Reenstra,et al.  Complement-mediated loss of endothelium-dependent relaxation of porcine coronary arteries. Role of the terminal membrane attack complex. , 1995, Circulation research.

[8]  S. Rendig,et al.  Limitation of reperfusion injury by a monoclonal antibody to C5a during myocardial infarction in pigs. , 1995, The American journal of physiology.

[9]  A. M. Lefer,et al.  Sialyl Lewisx-containing oligosaccharide attenuates myocardial reperfusion injury in cats. , 1994, The Journal of clinical investigation.

[10]  F. Sellke,et al.  Coronary Endothelial Injury After Cardiopulmonary Bypass and Ischemic Cardioplegia Is Mediated by Oxygen‐Derived Free Radicals , 1993, Circulation.

[11]  A. M. Lefer,et al.  Monoclonal Antibody to L‐Selectin Attenuates Neutrophil Accumulation and Protects Ischemic Reperfused Cat Myocardium , 1993, Circulation.

[12]  A. M. Lefer,et al.  In vivo neutralization of P-selectin protects feline heart and endothelium in myocardial ischemia and reperfusion injury. , 1993, The Journal of clinical investigation.

[13]  R. Engler,et al.  Role of cardiac mast cells in complement C5a-induced myocardial ischemia. , 1993, The American journal of physiology.

[14]  S H Snyder,et al.  Molecular cloning and expression of inducible nitric oxide synthase from human hepatocytes. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[15]  A. M. Lefer,et al.  Diminished basal nitric oxide release after myocardial ischemia and reperfusion promotes neutrophil adherence to coronary endothelium. , 1993, Circulation research.

[16]  F. Sellke,et al.  Impaired endothelium-dependent coronary microvascular relaxation after cold potassium cardioplegia and reperfusion. , 1993, The Journal of thoracic and cardiovascular surgery.

[17]  S. Watson,et al.  The three members of the selectin receptor family recognize a common carbohydrate epitope, the sialyl Lewis(x) oligosaccharide , 1992, The Journal of cell biology.

[18]  G. Zimmerman,et al.  Coexpression of GMP-140 and PAF by endothelium stimulated by histamine or thrombin: a juxtacrine system for adhesion and activation of neutrophils , 1991, The Journal of cell biology.

[19]  J. Longhurst,et al.  Role of granulocytes and C5a in myocardial response to zymosan-activated serum. , 1991, The American journal of physiology.

[20]  P. Kubes,et al.  Nitric oxide: an endogenous modulator of leukocyte adhesion. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[21]  G. Zimmerman,et al.  Oxygen radicals induce human endothelial cells to express GMP-140 and bind neutrophils , 1991, The Journal of cell biology.

[22]  A. M. Lefer,et al.  Time course of endothelial dysfunction and myocardial injury during myocardial ischemia and reperfusion in the cat. , 1990, Circulation.

[23]  E. Fosse,et al.  Complement activation with bubble and membrane oxygenators in aortocoronary bypass grafting. , 1990, The Annals of thoracic surgery.

[24]  R. Bolli,et al.  Marked Reduction of Free Radical Generation and Contractile Dysfunction by Antioxidant Therapy Begun at the Time of Reperfusion Evidence That Myocardial "Stunning" Is a Manifestation of Reperfusion Injury , 1989, Circulation research.

[25]  J. Longhurst,et al.  C5a Decreases Regional Coronary Blood Flow and Myocardial Function in Pigs: Implications for a Granulocyte Mechanism , 1988, Circulation research.