Regional, reversible ultrastructural changes in rat brain with chronic neuroleptic treatment

[1]  G. Breese,et al.  Behavioral and neurochemical responses to haloperidol and SCH-23390 in rats treated neonatally or as adults with 6-hydroxydopamine. , 1987, The Journal of pharmacology and experimental therapeutics.

[2]  F. Morrell,et al.  Axospinous synapses with segmented postsynaptic densities: a morphologically distinct synaptic subtype contributing to the number of profiles of ‘perforated’ synapses visualized in random sections , 1987, Brain Research.

[3]  C. Blaha,et al.  Chronic treatment with classical and atypical antipsychotic drugs differentially decreases dopamine release in striatum and nucleus accumbens in vivo , 1987, Neuroscience Letters.

[4]  M. A. Rogers,et al.  Principles of Neural Science, 2nd ed , 1987 .

[5]  H. Meltzer,et al.  Psychopharmacology : the third generation of progress , 1987 .

[6]  J. E. Vaughn,et al.  Immunocytochemical localization of choline acetyltransferase in rat ventral striatum: a light and electron microscopic study , 1986, Journal of neurocytology.

[7]  F. Morrell,et al.  Loss of perforated synapses in the dentate gyrus: morphological substrate of memory deficit in aged rats. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[8]  T. Ljungberg,et al.  A rapid and simple behavioural screening method for simultaneous assessment of limbic and striatal blocking effects of neuroleptic drugs , 1985, Pharmacology Biochemistry and Behavior.

[9]  J. E. Vaughn,et al.  Immunocytochemical localization of choline acetyltransferase within the rat neostriatum: A correlated light and electron microscopic study of cholinergic neurons and synapses , 1985, The Journal of comparative neurology.

[10]  Francine M. Benes,et al.  The effects of haloperidol on synaptic patterns in the rat striatum , 1985, Brain Research.

[11]  J. Glowinski,et al.  The mesocortico-prefrontal dopaminergic neurons , 1984, Trends in Neurosciences.

[12]  J. Thibault,et al.  Ultrastructural morphology of dopaminergic nerve terminals and synapses in the striatum of the rat using tyrosine hydroxylase immunocytochemistry: A topographical study , 1984, Brain Research Bulletin.

[13]  J. Bouyer,et al.  Chemical and structural analysis of the relation between cortical inputs and tyrosine hydroxylase-containing terminals in rat neostriatum , 1984, Brain Research.

[14]  C. Marsden,et al.  Changes in apomorphine-induced stereotypy as a result of subacute neuroleptic treatment correlates with increased D-2 receptors, but not with increases in D-1 receptors. , 1983, Biochemical pharmacology.

[15]  P Siekevitz,et al.  Plasticity in the central nervous system: do synapses divide? , 1983, Proceedings of the National Academy of Sciences of the United States of America.

[16]  C. Marsden,et al.  The effect of chronic neuroleptic administration on cerebral dopamine receptor function. , 1983, Life sciences.

[17]  A. Kelley,et al.  The distribution of the projection from the hippocampal formation to the nucleus accumbens in the rat: An anterograde and retrograde-horseradish peroxidase study , 1982, Neuroscience.

[18]  C. Cotman,et al.  Perforated postsynaptic densities: probable intermediates in synapse turnover. , 1982, Proceedings of the National Academy of Sciences of the United States of America.

[19]  Mark Ellisman,et al.  A restructuring off hypothalamic synapses is associated with motherhood , 1982, The Journal of neuroscience : the official journal of the Society for Neuroscience.

[20]  G. Vrensen,et al.  Changes in size and shape of synaptic connections after visual training: An ultrastructural approach of synaptic plasticity , 1981, Brain Research.

[21]  J. Stevens Schizophrenia and dopamine regulation in the mesolimbic system , 1979, Trends in Neurosciences.

[22]  C. Marsden,et al.  Changes in dopamine-mediated behaviour during one year's neuroleptic administration. , 1979, European journal of pharmacology.

[23]  R. M. Beckstead An autoradiographic examination of corticocortical and subcortical projections of the mediodorsal‐projection (prefrontal) cortex in the rat , 1979, The Journal of comparative neurology.

[24]  W. Greenough,et al.  Subsynaptic plate perforations: changes with age and experience in the rat. , 1978, Science.

[25]  P. Siekevitz,et al.  Form of the postsynaptic density. A serial section study , 1978, The Journal of cell biology.

[26]  F E Bloom,et al.  Central catecholamine neuron systems: anatomy and physiology of the dopamine systems. , 1978, Annual review of neuroscience.

[27]  B. Scatton Differential regional development of tolerance to increase in dopamine turnover upon repeated neuroleptic administration. , 1977, European journal of pharmacology.

[28]  C. Mao,et al.  Turnover times of gamma-aminobutyric acid and acetylcholine in nucleus caudatus, nucleus accumbens, globus pallidus and substantia nigra: effects of repeated administration of haloperidol , 1977, Brain Research.

[29]  J. Korf,et al.  Regional effects of neuroleptics on dopamine metabolism and dopamine-sensitive adenylate cyclase activity. , 1977, European journal of pharmacology.

[30]  S. Snyder,et al.  Antischizophrenic drugs: chronic treatment elevates dopamine receptor binding in brain. , 1977, Science.

[31]  P. Seeman,et al.  Tolerance of haloperidol catalepsy. , 1977, European journal of pharmacology.

[32]  J. Hyttel,et al.  Changes in catecholamine concentrations and synthesis rate in mouse brain during the ‘supersensitivity’ phase after treatment with neuroleptic drugs , 1976, Journal of neurochemistry.

[33]  G. Bartholini,et al.  Differential effect of neuroleptic drugs on dopamine turnover in the extrapyramidal and limbic system , 1976, The Journal of pharmacy and pharmacology.

[34]  M. Stanley,et al.  Differential sensitivity of two dopaminergic structures in rat brain to haloperidol and to clozapine. , 1975, The Journal of pharmacology and experimental therapeutics.

[35]  J. Hyttel LONG‐TERM EFFECTS OF TEFLUTIXOL ON THE SYNTHESIS AND ENDOGENOUS LEVELS OF MOUSE BRAIN CATECHOLAMINES , 1975, Journal of neurochemistry.

[36]  A. Guidotti,et al.  Effect of thioridazine, clozapine and other antipsychotics on the kinetic state of tyrosine hydroxylase and on the turnover rate of dopamine in striatum and nucleus accumbens. , 1975, The Journal of pharmacology and experimental therapeutics.

[37]  H. H. Keller,et al.  Drug‐induced changes of dopamine turnover in striatum and limbic system of the rat , 1975, The Journal of pharmacy and pharmacology.

[38]  V. Tennyson,et al.  Dopamine-containing neurons of the substantia nigra and their terminals in the neostriatum. , 1975, UCLA forum in medical sciences.

[39]  V. Tennyson,et al.  5-Hydroxydopamine 'tagged' neuronal boutons in rabbit neostriatum: interrelationship between vesicles and axonal membrane. , 1974, Brain research.

[40]  R. Baldessarini,et al.  Behavioural supersensitivity to apomorphine following chronic treatment with drugs which interfere with the synaptic function of catecholamines. , 1974, Neuropharmacology.

[41]  M. Baggiolini,et al.  Tolerance phenomena with neuroleptics catalepsy, apomorphine stereotypies and striatal dopamine metabolism in the rat after single and repeated administration of loxapine and haloperidol. , 1973, European journal of pharmacology.

[42]  N. Andén,et al.  Effect of clozapine on the turnover of dopamine in the corpus striatum and in the limbic system , 1973, The Journal of pharmacy and pharmacology.

[43]  T. Powell,et al.  The site of termination of afferent fibres in the caudate nucleus. , 1971, Philosophical transactions of the Royal Society of London. Series B, Biological sciences.

[44]  W M COWAN,et al.  A bilateral cortico-striate projection , 1965, Journal of neurology, neurosurgery, and psychiatry.