Enhanced inflammatory response to coronary angioplasty in patients with severe unstable angina.

We congratulate Liuzzo et al on the recent publication of their study,1 which suggests that disruption of culprit coronary stenoses by PTCA does not necessarily lead to an increase in the level of systemic inflammatory markers (C-reactive protein [CRP], serum amyloid A protein [SAA], and interleukin-6 [IL-6]) in patients with unstable angina. The authors found that serum levels …