Failure to detect in vivo inverse agonism of the 5-HT1B receptor antagonist SB-224289 in 5-HT-depleted guinea-pigs
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[1] J. Javitch,et al. Mechanisms of inverse agonism of antipsychotic drugs at the D2 dopamine receptor: use of a mutant D2 dopamine receptor that adopts the activated conformation , 2001, Journal of neurochemistry.
[2] S. Ross,et al. Pharmacological characterisation of the decrease in 5-HT synthesis in the mouse brain evoked by the selective serotonin re-uptake inhibitor citalopram , 2001, Naunyn-Schmiedeberg's Archives of Pharmacology.
[3] Hong Yu,et al. Enhanced 5-HT metabolism and synthesis rate by the new selective r5-HT1B receptor antagonist, NAS-181 in the rat brain , 2000, Neuropharmacology.
[4] D. Middlemiss,et al. Enhancement of 5-HT1B and 5-HT1D receptor antagonist effects on extracellular 5-HT levels in the guinea-pig brain following concurrent 5-HT1A or 5-HT re-uptake site blockade , 1999, Neuropharmacology.
[5] A. Gobert,et al. Inverse Agonists and Serotonergic Transmission: From Recombinant, Human Serotonin (5-HT)1B Receptors to G-Protein Coupling and Function in Corticolimbic Structures in vivo , 1999, Neuropsychopharmacology.
[6] L. Larsson,et al. Synergism between 5-HT1B/1D and 5-HT1A receptor antagonists on turnover and release of 5-HT in guinea-pig brain in vivo , 1999, Naunyn-Schmiedeberg's Archives of Pharmacology.
[7] D. Middlemiss,et al. SB‐224289–a novel selective (human) 5‐HT1B receptor antagonist with negative intrinsic activity , 1998, British journal of pharmacology.
[8] D. Middlemiss,et al. Differential effects of 5-HT1B/1D receptor antagonists in dorsal and median raphe innervated brain regions. , 1998, European journal of pharmacology.
[9] P. Pauwels,et al. Pharmacological analysis of G‐protein activation mediated by guinea‐pig recombinant 5‐HT1B receptors in C6‐glial cells: similarities with the human 5‐HT1B receptor , 1998, British journal of pharmacology.
[10] G. Milligan,et al. Inverse agonism and the regulation of receptor number. , 1997, Trends in pharmacological sciences.
[11] J. Hagan,et al. Stimulation of 5-HT1B receptors causes hypothermia in the guinea pig. , 1997, European journal of pharmacology.
[12] H. Rollema,et al. Combined Administration of a 5‐Hydroxytryptamine (5‐HT)1D Antagonist and a 5‐HT Reuptake Inhibitor Synergistically Increases 5‐HT Release in Guinea Pig Hypothalamus In Vivo , 1996, Journal of neurochemistry.
[13] S. Hjorth,et al. Studies on the role of 5-HT1A autoreceptors and α 1-adrenoceptors in the inhibition of 5-HT release—I. BMY7378 and prazosin , 1995, Neuropharmacology.
[14] M. Diksic,et al. The Acute Effects of Reserpine and NSD-1015 on the Brain Serotonin Synthesis Rate Measured by an Autoradiographic Method , 1995, Neuropsychopharmacology.
[15] D. Pearce,et al. The effects of GR127935, a putative 5-HT1D receptor antagonist, on brain 5-HT metabolism, extracellular 5-HTT concentration and behaviour in the guinea pig , 1995, Neuropharmacology.
[16] A. Sleight,et al. Effects of the 5-HT1D receptor antagonist GR127935 on extracellular levels of 5-HT in the guinea-pig frontal cortex as measured by microdialysis , 1995, Neuropharmacology.
[17] M. Foguet,et al. Long‐term regulation of serotonergic activity in the rat brain via activation of protein kinase A. , 1993, The EMBO journal.
[18] R. Tao,et al. The putative 5-HT1B receptor agonist CP-93,129 suppresses rat hippocampal 5-HT release in vivo: comparison with RU 24969. , 1991, European journal of pharmacology.
[19] P. Pévet,et al. Tryptophan Hydroxylase Synthesis Is Induced by 3′, 5′‐Cyclic Adenosine Monophosphate During Circadian Rhythm in the Rat Pineal Gland , 1991, Journal of neurochemistry.
[20] M. Millan,et al. Inability of an opioid antagonist lacking negative intrinsic activity to induce opioid receptor up‐regulation in vivo , 1991, British journal of pharmacology.
[21] M. Goldstein,et al. Receptor reserve for 5-hydroxytryptamine1A-mediated inhibition of serotonin synthesis: possible relationship to anxiolytic properties of 5-hydroxytryptamine1A agonists. , 1990, Molecular pharmacology.
[22] S. Ross,et al. Different effects on the responses of functional pre- and postsynaptic 5-HT1A receptors by repeated treatment of rats with the 5-HT1A receptor agonist 8-OH-DPAT , 1990, Neuropharmacology.
[23] D G Grahame-Smith,et al. 5‐HT1 agonists reduce 5‐hydroxytryptamine release in rat hippocampus in vivo as determined by brain microdialysis , 1989, British journal of pharmacology.
[24] G. Aghajanian,et al. (-)-Propranolol blocks the inhibition of serotonergic dorsal raphe cell firing by 5-HT1A selective agonists. , 1986, European journal of pharmacology.
[25] D. Westerlund,et al. Simultaneous determination of dopamine, DOPAC and homovanillic acid. Direct injection of supernatants from brain tissue homogenates in a liquid chromatography--electrochemical detection system. , 1980, Journal of chromatography.
[26] E. Schlicker,et al. Identity of inhibitory presynaptic 5-hydroxytryptamine (5-HT) autoreceptors in the rat brain cortex with 5-HT1B binding sites , 2004, Naunyn-Schmiedeberg's Archives of Pharmacology.
[27] E. Schlicker,et al. Regulation of 5-HT Release in the CNS by Presynaptic 5-HT Autoreceptors and by 5-HT Heteroreceptors , 2000 .
[28] C. de Montigny,et al. Modification of 5‐HT neuron properties by sustained administration of the 5‐HT1A agonist gepirone: Electrophysiological studies in the rat brain , 1987, Synapse.