Decreased mortality associated with statin treatment in patients with acute myocardial infarction and lymphotoxin-alpha C804A polymorphism.
暂无分享,去创建一个
Toshihiro Tanaka | K. Ozaki | I. Komuro | M. Hori | M. Nishino | Hiroshi Sato | H. Mizuno | S. Takashima | M. Hara | Y. Sakata | T. Minamino | Y. Matsumura | D. Nakatani | H. Takeda | K. Okuda | Masaya Usami | Sen Matsumoto | M. Shimizu | Shinichiro Suna
[1] P. Libby. Inflammation in Atherosclerosis , 2012, Arteriosclerosis, thrombosis, and vascular biology.
[2] I. Komuro,et al. Endoplasmic reticulum stress as a therapeutic target in cardiovascular disease. , 2010, Circulation research.
[3] Paul T. Williams,et al. Genome-Wide Association of Lipid-Lowering Response to Statins in Combined Study Populations , 2010, PloS one.
[4] Yusuke Nakamura,et al. SNPs in BRAP associated with risk of myocardial infarction in Asian populations , 2009, Nature Genetics.
[5] Toshihiro Tanaka,et al. Lymphotoxin-alpha3 mediates monocyte-endothelial interaction by TNFR I/NF-kappaB signaling. , 2009, Biochemical and biophysical research communications.
[6] M. Hori,et al. Inhibition of Cardiac Remodeling by Pravastatin Is Associated with Amelioration of Endoplasmic Reticulum Stress , 2008, Hypertension Research.
[7] M. Hori,et al. Up-regulation of cell adhesion molecule genes in human endothelial cells stimulated by lymphotoxin alpha: DNA microarray analysis. , 2008, Journal of atherosclerosis and thrombosis.
[8] Elena Galkina,et al. Vascular adhesion molecules in atherosclerosis. , 2007, Arteriosclerosis, thrombosis, and vascular biology.
[9] K. Eagle,et al. Decline in rates of death and heart failure in acute coronary syndromes, 1999-2006. , 2007, JAMA.
[10] Yusuke Nakamura,et al. A functional SNP in PSMA6 confers risk of myocardial infarction in the Japanese population , 2006, Nature Genetics.
[11] 水野 裕八. Impact of atherosclerosis-related gene polymorphisms on mortality and recurrent events after myocardial infarction , 2006 .
[12] Hong Jiang,et al. Fluvastatin inhibits the expression of tumor necrosis factor-alpha and activation of nuclear factor-kappaB in human endothelial cells stimulated by C-reactive protein. , 2005, Clinica chimica acta; international journal of clinical chemistry.
[13] M. Vázquez-Carrera,et al. Atorvastatin improves peroxisome proliferator-activated receptor signaling in cardiac hypertrophy by preventing nuclear factor-kappa B activation. , 2005, Biochimica et biophysica acta.
[14] M. Pfeffer,et al. C-reactive protein levels and outcomes after statin therapy. , 2005, The New England journal of medicine.
[15] M. Hori,et al. Prolonged Endoplasmic Reticulum Stress in Hypertrophic and Failing Heart After Aortic Constriction: Possible Contribution of Endoplasmic Reticulum Stress to Cardiac Myocyte Apoptosis , 2004, Circulation.
[16] T. Wilt,et al. Effectiveness of statin therapy in adults with coronary heart disease. , 2004, Archives of internal medicine.
[17] M. Hori,et al. Reduced collateral circulation to the infarct-related artery in elderly patients with acute myocardial infarction. , 2004, Journal of the American College of Cardiology.
[18] Yusuke Nakamura,et al. Functional variation in LGALS2 confers risk of myocardial infarction and regulates lymphotoxin-α secretion in vitro , 2004, Nature.
[19] C. Napoli,et al. Statin effects beyond lipid lowering--are they clinically relevant? , 2003, European heart journal.
[20] R. Leboeuf,et al. Loss of Lymphotoxin-α but Not Tumor Necrosis Factor-α Reduces Atherosclerosis in Mice* , 2002, The Journal of Biological Chemistry.
[21] T. Awata,et al. Fibrate and Statin Synergistically Increase the Transcriptional Activities of PPARα/RXRα and Decrease the Transactivation of NFκB , 2002 .
[22] P. Hansen,et al. Diverse effects of inhibition of 3-hydroxy-3-methylglutaryl-CoA reductase on the expression of VCAM-1 and E-selectin in endothelial cells. , 2001, The Biochemical journal.
[23] Yoshiji Yamada,et al. Association of the C–509→T polymorphism, alone or in combination with the T869→C polymorphism, of the transforming growth factor-β1 gene with bone mineral density and genetic susceptibility to osteoporosis in Japanese women , 2001, Journal of Molecular Medicine.
[24] B. Aggarwal,et al. Cloning and expression of cDNA for human lymphotoxin, a lymphokine with tumour necrosis activity , 1984, Nature.
[25] G. Granger,et al. Lymphocyte Cytotoxicity in vitro: Activation and Release of a Cytotoxic Factor , 1968, Nature.
[26] B. Waksman,et al. Cytotoxic Effect of Lymphocyte-Antigen Interaction in Delayed Hypersensitivity , 1967, Science.
[27] B. Gersh. Association Between Adoption of Evidence-Based Treatment and Survival for Patients With ST-Elevation Myocardial Infarction , 2012 .
[28] The Procardis Consortium. A trio family study showing association of the lymphotoxin-α N26 (804A) allele with coronary artery disease , 2004, European Journal of Human Genetics.
[29] H. Nonogi,et al. Association analysis between polymorphisms of the lymphotoxin-alpha gene and myocardial infarction in a Japanese population. , 2004, Atherosclerosis.
[30] P. O. Bonettia,et al. Statin effects beyond lipid lowering—are they clinically relevant? , 2003 .
[31] Y. Ohnishi,et al. Functional SNPs in the lymphotoxin-α gene that are associated with susceptibility to myocardial infarction , 2003, Nature Genetics.
[32] T. Awata,et al. Fibrate and statin synergistically increase the transcriptional activities of PPARalpha/RXRalpha and decrease the transactivation of NFkappaB. , 2002, Biochemical and biophysical research communications.
[33] R. Leboeuf,et al. Loss of lymphotoxin-alpha but not tumor necrosis factor-alpha reduces atherosclerosis in mice. , 2002, The Journal of biological chemistry.