Deficiency of miR-15a/16 upregulates NKG2D in CD8+ T cells to exacerbate dextran sulfate sodium-induced colitis.

[1]  T. Zhu,et al.  Low miR-16 expression induces regulatory CD4+NKG2D+ T cells involved in colorectal cancer progression. , 2021, American journal of cancer research.

[2]  T. Rülicke,et al.  Differential roles of miR‐15a/16‐1 and miR‐497/195 clusters in immune cell development and homeostasis , 2020, The FEBS journal.

[3]  M. Caligiuri,et al.  Combined loss of function of two different loci of miR-15/16 drives the pathogenesis of acute myeloid leukemia , 2020, Proceedings of the National Academy of Sciences.

[4]  Michael T. McManus,et al.  miR-15/16 Restrain Memory T Cell Differentiation, Cell Cycle, and Survival , 2019, Cell reports.

[5]  Zhitao Feng,et al.  The role of microRNA-16 in the pathogenesis of autoimmune diseases: A comprehensive review. , 2019, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.

[6]  Y. Pekarsky,et al.  Noncoding RNA genes in cancer pathogenesis. , 2019, Advances in biological regulation.

[7]  Sen Han,et al.  Increased M1 macrophages in young miR‐15a/16−/− mice with tumour grafts or dextran sulphate sodium‐induced colitis , 2018, Scandinavian journal of immunology.

[8]  Guang-Hui Li,et al.  MicroRNA‐16 functions as a tumor‐suppressor gene in oral squamous cell carcinoma by targeting AKT3 and BCL2L2 , 2018, Journal of cellular physiology.

[9]  Y. Pekarsky,et al.  BCL2 and miR-15/16: from gene discovery to treatment. , 2018 .

[10]  Y. Chu,et al.  MiR‐15a/16 deficiency enhances anti‐tumor immunity of glioma‐infiltrating CD8+ T cells through targeting mTOR , 2017, International journal of cancer.

[11]  G. Zhu,et al.  NK1.1− CD4+ NKG2D+ T cells suppress DSS‐induced colitis in mice through production of TGF‐β , 2017, Journal of cellular and molecular medicine.

[12]  R. Yao,et al.  MiR-15a/16 regulates the growth of myeloma cells, angiogenesis and antitumor immunity by inhibiting Bcl-2, VEGF-A and IL-17 expression in multiple myeloma. , 2016, Leukemia research.

[13]  Guangping Li,et al.  MicroRNA-16 suppresses the activation of inflammatory macrophages in atherosclerosis by targeting PDCD4 , 2016, International journal of molecular medicine.

[14]  Y. Chu,et al.  miRNA-15a/16: as tumor suppressors and more. , 2015, Future oncology.

[15]  Y. Pekarsky,et al.  Role of miR-15/16 in CLL , 2014, Cell Death and Differentiation.

[16]  M. Mann,et al.  microRNA-mediated regulation of mTOR complex components facilitates discrimination between activation and anergy in CD4 T cells , 2014, The Journal of experimental medicine.

[17]  Hongbing Shen,et al.  A five-microRNA panel in plasma was identified as potential biomarker for early detection of gastric cancer , 2014, British Journal of Cancer.

[18]  Yanbing Ding,et al.  CD4+ NKG2D+ T cells induce NKG2D down‐regulation in natural killer cells in CD86‐RAE‐1ε transgenic mice , 2014, Immunology.

[19]  H. Lindner,et al.  Adenosine A2A Receptor Upregulation in Human PMNs Is Controlled by miRNA-214, miRNA-15, and miRNA-16 , 2012, Shock.

[20]  Yan-Yan Zhang,et al.  MicroRNAs modulate the noncanonical transcription factor NF-kappaB pathway by regulating expression of the kinase IKKalpha during macrophage differentiation. , 2010 .

[21]  C. Croce,et al.  miR-15a and miR-16-1 in cancer: discovery, function and future perspectives , 2010, Cell Death and Differentiation.

[22]  Pablo Landgraf,et al.  Abnormal microRNA-16 locus with synteny to human 13q14 linked to CLL in NZB mice. , 2007, Blood.