Deficiency of miR-15a/16 upregulates NKG2D in CD8+ T cells to exacerbate dextran sulfate sodium-induced colitis.
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W. Xiao | Yingying Wei | W. Gong | Tao Zhu | Xiaoqin Jia | X. Miao | Yu Zhang | Xiangyu Hu | Zhengbing Wang | Zhijie Lin
[1] T. Zhu,et al. Low miR-16 expression induces regulatory CD4+NKG2D+ T cells involved in colorectal cancer progression. , 2021, American journal of cancer research.
[2] T. Rülicke,et al. Differential roles of miR‐15a/16‐1 and miR‐497/195 clusters in immune cell development and homeostasis , 2020, The FEBS journal.
[3] M. Caligiuri,et al. Combined loss of function of two different loci of miR-15/16 drives the pathogenesis of acute myeloid leukemia , 2020, Proceedings of the National Academy of Sciences.
[4] Michael T. McManus,et al. miR-15/16 Restrain Memory T Cell Differentiation, Cell Cycle, and Survival , 2019, Cell reports.
[5] Zhitao Feng,et al. The role of microRNA-16 in the pathogenesis of autoimmune diseases: A comprehensive review. , 2019, Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie.
[6] Y. Pekarsky,et al. Noncoding RNA genes in cancer pathogenesis. , 2019, Advances in biological regulation.
[7] Sen Han,et al. Increased M1 macrophages in young miR‐15a/16−/− mice with tumour grafts or dextran sulphate sodium‐induced colitis , 2018, Scandinavian journal of immunology.
[8] Guang-Hui Li,et al. MicroRNA‐16 functions as a tumor‐suppressor gene in oral squamous cell carcinoma by targeting AKT3 and BCL2L2 , 2018, Journal of cellular physiology.
[9] Y. Pekarsky,et al. BCL2 and miR-15/16: from gene discovery to treatment. , 2018 .
[10] Y. Chu,et al. MiR‐15a/16 deficiency enhances anti‐tumor immunity of glioma‐infiltrating CD8+ T cells through targeting mTOR , 2017, International journal of cancer.
[11] G. Zhu,et al. NK1.1− CD4+ NKG2D+ T cells suppress DSS‐induced colitis in mice through production of TGF‐β , 2017, Journal of cellular and molecular medicine.
[12] R. Yao,et al. MiR-15a/16 regulates the growth of myeloma cells, angiogenesis and antitumor immunity by inhibiting Bcl-2, VEGF-A and IL-17 expression in multiple myeloma. , 2016, Leukemia research.
[13] Guangping Li,et al. MicroRNA-16 suppresses the activation of inflammatory macrophages in atherosclerosis by targeting PDCD4 , 2016, International journal of molecular medicine.
[14] Y. Chu,et al. miRNA-15a/16: as tumor suppressors and more. , 2015, Future oncology.
[15] Y. Pekarsky,et al. Role of miR-15/16 in CLL , 2014, Cell Death and Differentiation.
[16] M. Mann,et al. microRNA-mediated regulation of mTOR complex components facilitates discrimination between activation and anergy in CD4 T cells , 2014, The Journal of experimental medicine.
[17] Hongbing Shen,et al. A five-microRNA panel in plasma was identified as potential biomarker for early detection of gastric cancer , 2014, British Journal of Cancer.
[18] Yanbing Ding,et al. CD4+ NKG2D+ T cells induce NKG2D down‐regulation in natural killer cells in CD86‐RAE‐1ε transgenic mice , 2014, Immunology.
[19] H. Lindner,et al. Adenosine A2A Receptor Upregulation in Human PMNs Is Controlled by miRNA-214, miRNA-15, and miRNA-16 , 2012, Shock.
[20] Yan-Yan Zhang,et al. MicroRNAs modulate the noncanonical transcription factor NF-kappaB pathway by regulating expression of the kinase IKKalpha during macrophage differentiation. , 2010 .
[21] C. Croce,et al. miR-15a and miR-16-1 in cancer: discovery, function and future perspectives , 2010, Cell Death and Differentiation.
[22] Pablo Landgraf,et al. Abnormal microRNA-16 locus with synteny to human 13q14 linked to CLL in NZB mice. , 2007, Blood.