Dendritic cells permit immune invasion of the CNS in an animal model of multiple sclerosis
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B. Becher | F. Heppner | R. Noelle | M. Greter | T. Laufer | M. Lemos | B. Odermatt | N. Goebels | Bernhard Odermatt | Maria P. Lemos
[1] G. Trinchieri,et al. Astrocytes as antigen‐presenting cells: expression of IL‐12/IL‐23 , 2005, Journal of neurochemistry.
[2] S. Miller,et al. Epitope spreading initiates in the CNS in two mouse models of multiple sclerosis , 2005, Nature Medicine.
[3] B. Becher,et al. Experimental autoimmune encephalomyelitis repressed by microglial paralysis , 2005, Nature Medicine.
[4] R. Ravid,et al. Expression of CCR7 in multiple sclerosis: Implications for CNS immunity , 2004, Annals of neurology.
[5] Y. Imai,et al. Human CD34+ cells differentiate into microglia and express recombinant therapeutic protein. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[6] P. Scott,et al. MHC Class II Expression Restricted to CD8α+ and CD11b+ Dendritic Cells Is Sufficient for Control of Leishmania major , 2004, The Journal of experimental medicine.
[7] D. Lo,et al. CD8α+ and CD11b+ Dendritic Cell-Restricted MHC Class II Controls Th1 CD4+ T Cell Immunity 1 , 2003, The Journal of Immunology.
[8] B. Becher,et al. IL-23 produced by CNS-resident cells controls T cell encephalitogenicity during the effector phase of experimental autoimmune encephalomyelitis. , 2003, The Journal of clinical investigation.
[9] R. Kastelein,et al. Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain , 2003, Nature.
[10] S. Youssef,et al. The Role of the MHC Class II Transactivator in Class II Expression and Antigen Presentation by Astrocytes and in Susceptibility to Central Nervous System Autoimmune Disease1 , 2002, The Journal of Immunology.
[11] Meredith O'Keeffe,et al. Effects of administration of progenipoietin 1, Flt-3 ligand, granulocyte colony-stimulating factor, and pegylated granulocyte-macrophage colony-stimulating factor on dendritic cell subsets in mice. , 2002, Blood.
[12] Mark M. Davis,et al. Dynamics of the immunological synapse: finding, establishing and solidifying a connection. , 2002, Current opinion in immunology.
[13] H. Weiner,et al. Requirement for endocytic antigen processing and influence of invariant chain and H-2M deficiencies in CNS autoimmunity. , 2001, The Journal of clinical investigation.
[14] H. Lassmann,et al. Migratory activity and functional changes of green fluorescent effector cells before and during experimental autoimmune encephalomyelitis. , 2001, Immunity.
[15] B. Becher,et al. The Clinical Course of Experimental Autoimmune Encephalomyelitis and Inflammation Is Controlled by the Expression of Cd40 within the Central Nervous System , 2001, The Journal of experimental medicine.
[16] A. Juedes,et al. Resident and Infiltrating Central Nervous System APCs Regulate the Emergence and Resolution of Experimental Autoimmune Encephalomyelitis1 , 2001, The Journal of Immunology.
[17] B. Serafini,et al. Intracerebral recruitment and maturation of dendritic cells in the onset and progression of experimental autoimmune encephalomyelitis. , 2000, The American journal of pathology.
[18] C. Figdor,et al. Identification of DC-SIGN, a Novel Dendritic Cell–Specific ICAM-3 Receptor that Supports Primary Immune Responses , 2000, Cell.
[19] Burkhard Becher,et al. Brain‐immune connection: Immuno‐regulatory properties of CNS‐resident cells , 2000, Glia.
[20] P. Ricciardi-Castagnoli,et al. Microglia induce myelin basic protein‐specific T cell anergy or T cell activation, according to their state of activation , 1999, European journal of immunology.
[21] T. Serikawa,et al. Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-κb-inducing kinase , 1999, Nature Genetics.
[22] P. McMenamin,et al. Distribution and phenotype of dendritic cells and resident tissue macrophages in the dura mater, leptomeninges, and choroid plexus of the rat brain as demonstrated in wholemount preparations , 1999, The Journal of comparative neurology.
[23] L. Adorini,et al. Microglia are more efficient than astrocytes in antigen processing and in Th1 but not Th2 cell activation. , 1998, Journal of immunology.
[24] J. Sutcliffe,et al. Mature microglia resemble immature antigen‐presenting cells , 1998, Glia.
[25] E. Maraskovsky,et al. Dramatic increase in the numbers of functionally mature dendritic cells in Flt3 ligand-treated mice: multiple dendritic cell subpopulations identified , 1996, The Journal of experimental medicine.
[26] J. Sedgwick,et al. Microglia induce CD4 T lymphocyte final effector function and death , 1996, The Journal of experimental medicine.
[27] B. Becher,et al. Comparison of phenotypic and functional properties of immediately ex vivo and cultured human adult microglia , 1996, Glia.
[28] V. Perry,et al. The potential role of dendritic cells in immune-mediated inflammatory diseases in the central nervous system , 1996, Neuroscience.
[29] L Steinman,et al. Multiple Sclerosis: A Coordinated Immunological Attack against Myelin in the Central Nervous System , 1996, Cell.
[30] W. Hickey,et al. Normal adult ramified microglia separated from other central nervous system macrophages by flow cytometric sorting. Phenotypic differences defined and direct ex vivo antigen presentation to myelin basic protein-reactive CD4+ T cells compared. , 1995, Journal of immunology.
[31] R. Bjerkvig,et al. Human microglial cells have phenotypic and functional characteristics in common with both macrophages and dendritic antigen‐presenting cells , 1994, Journal of leukocyte biology.
[32] H. Lassmann,et al. Bone marrow derived elements and resident microglia in brain inflammation , 1993, Glia.
[33] H. Lassmann,et al. Bone Marrow-derived Elements in the Central Nervous System: An Immunohistochemical and Ultrastructural Survey of Rat Chimeras , 1992, Journal of neuropathology and experimental neurology.
[34] W. Hickey. Migration of Hematogenous Cells Through the Blood‐Brain Barrier and the Initiation of CNS Inflammation , 1991, Brain pathology.
[35] Sunhee C. Lee,et al. Multiple sclerosis: Oligodendrocytes in active lesions do not express class II major histocompatibility complex molecules , 1989, Journal of Neuroimmunology.
[36] W. Hickey,et al. Perivascular microglial cells of the CNS are bone marrow-derived and present antigen in vivo. , 1988, Science.
[37] W. Fierz,et al. Astrocytes as antigen-presenting cells. I. Induction of Ia antigen expression on astrocytes by T cells via immune interferon and its effect on antigen presentation. , 1985, Journal of immunology.
[38] D. McFarlin,et al. Adoptively transferred chronic relapsing experimental autoimmune encephalomyelitis in the mouse. Neuropathologic analysis. , 1984, Laboratory investigation; a journal of technical methods and pathology.
[39] J. Prineas. Multiple sclerosis: presence of lymphatic capillaries and lymphoid tissue in the brain and spinal cord. , 1979, Science.
[40] T. Serikawa,et al. Alymphoplasia is caused by a point mutation in the mouse gene encoding Nf-kappa b-inducing kinase. , 1999, Nature genetics.
[41] V. ter meulen,et al. Astrocytes as antigen presenting cells for primary and secondary T cell responses: effect of astrocyte infection by murine hepatitis virus. , 1990, Advances in experimental medicine and biology.
[42] G. Wong,et al. Inducible expression of H–2 and Ia antigens on brain cells , 1984, Nature.