A sensitive array-based assay for identifying multiple TMPRSS2:ERG fusion gene variants
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Jessica Wang-Rodriguez | Tracy Downs | J. Wang-Rodriguez | Yu-Tseung Liu | D. Carson | T. Downs | K. Christensen | Yu-Tsueng Liu | Dennis A. Carson | Esperanza Nunez | Qing Lu | Chunrun Lin | Kimberly Christensen | Esperanza Núñez | Qing Lu | Chu-Fang Lin
[1] T. Tammela,et al. TMPRSS2:ERG Fusion Identifies a Subgroup of Prostate Cancers with a Favorable Prognosis , 2008, Clinical Cancer Research.
[2] M. Rubin. Targeted therapy of cancer: new roles for pathologists—prostate cancer , 2008, Modern Pathology.
[3] R. Shah,et al. Role of the TMPRSS2-ERG gene fusion in prostate cancer. , 2008, Neoplasia.
[4] S. Dhanasekaran,et al. Distinct classes of chromosomal rearrangements create oncogenic ETS gene fusions in prostate cancer , 2007, Nature.
[5] Gratien G. Prefontaine,et al. Developmentally Regulated Activation of a SINE B2 Repeat as a Domain Boundary in Organogenesis , 2007, Science.
[6] Y Pawitan,et al. TMPRSS2:ERG gene fusion associated with lethal prostate cancer in a watchful waiting cohort , 2007, Oncogene.
[7] B. Jordan. DNA microarrays in the clinic: how soon, how extensively? , 2007, BioEssays : news and reviews in molecular, cellular and developmental biology.
[8] Junia V. Melo,et al. Chronic myeloid leukaemia as a model of disease evolution in human cancer , 2007, Nature Reviews Cancer.
[9] Debashis Ghosh,et al. Comprehensive assessment of TMPRSS2 and ETS family gene aberrations in clinically localized prostate cancer , 2007, Modern Pathology.
[10] R. Eeles,et al. Diversity of TMPRSS2-ERG fusion transcripts in the human prostate , 2007, Oncogene.
[11] Yu-Tseung Liu,et al. A Novel Approach for Determining Cancer Genomic Breakpoints in the Presence of Normal DNA , 2007, PloS one.
[12] T. Szczepański. Why and how to quantify minimal residual disease in acute lymphoblastic leukemia? , 2007, Leukemia.
[13] B. Johansson,et al. The impact of translocations and gene fusions on cancer causation , 2007, Nature Reviews Cancer.
[14] J. Brooks,et al. A variant TMPRSS2 isoform and ERG fusion product in prostate cancer with implications for molecular diagnosis , 2007, Modern Pathology.
[15] S. Leung,et al. Frequency of the TMPRSS2:ERG gene fusion is increased in moderate to poorly differentiated prostate cancers , 2007, Journal of Clinical Pathology.
[16] Yang Liu,et al. Panmicrobial Oligonucleotide Array for Diagnosis of Infectious Diseases , 2007, Emerging infectious diseases.
[17] L. Klotz,et al. Expression of TMPRSS2:ERG gene fusion in prostate cancer cells is an important prognostic factor for cancer progression , 2007, Cancer biology & therapy.
[18] G. Jenster,et al. TMPRSS2:ERG fusion by translocation or interstitial deletion is highly relevant in androgen-dependent prostate cancer, but is bypassed in late-stage androgen receptor-negative prostate cancer. , 2006, Cancer research.
[19] O. Kallioniemi,et al. TMPRSS2 fusions with oncogenic ETS factors in prostate cancer involve unbalanced genomic rearrangements and are associated with HDAC1 and epigenetic reprogramming. , 2006, Cancer research.
[20] Lei Wang,et al. Noninvasive detection of TMPRSS2:ERG fusion transcripts in the urine of men with prostate cancer. , 2006, Neoplasia.
[21] R. Henrique,et al. TMPRSS2-ERG gene fusion causing ERG overexpression precedes chromosome copy number changes in prostate carcinomas and paired HGPIN lesions. , 2006, Neoplasia.
[22] Michael Ittmann,et al. Expression of variant TMPRSS2/ERG fusion messenger RNAs is associated with aggressive prostate cancer. , 2006, Cancer research.
[23] J. Tchinda,et al. TMPRSS2:ERG fusion-associated deletions provide insight into the heterogeneity of prostate cancer. , 2006, Cancer research.
[24] J. Schalken,et al. THE TMPRSS2-ERG FUSION GENE CAN BE FOUND IN URINARY SEDIMENTS AFTER DRE , 2006 .
[25] I. Panagopoulos,et al. Confirmation of the high frequency of the TMPRSS2/ERG fusion gene in prostate cancer. , 2006, Genes, chromosomes & cancer.
[26] A. Scott,et al. TARGETING OF TNF TO CLEAR CELL RENAL CELL CARCINOMA , 2006 .
[27] Simona Soverini,et al. Monitoring CML patients responding to treatment with tyrosine kinase inhibitors: review and recommendations for harmonizing current methodology for detecting BCR-ABL transcripts and kinase domain mutations and for expressing results. , 2006, Blood.
[28] A. Evans,et al. Three-color FISH analysis of TMPRSS2/ERG fusions in prostate cancer indicates that genomic microdeletion of chromosome 21 is associated with rearrangement. , 2006, Neoplasia.
[29] J. Tchinda,et al. Recurrent fusion of TMPRSS2 and ETS transcription factor genes in prostate cancer. , 2006, Science.
[30] Arul M Chinnaiyan,et al. TMPRSS2:ETV4 gene fusions define a third molecular subtype of prostate cancer. , 2006, Cancer research.
[31] J. Cayuela,et al. A diagnostic biochip for the comprehensive analysis of MLL translocations in acute leukemia , 2004, Leukemia.
[32] J. Lieb,et al. ChIP-chip: considerations for the design, analysis, and application of genome-wide chromatin immunoprecipitation experiments. , 2004, Genomics.
[33] J. Wang-Rodriguez,et al. In silico dissection of cell-type-associated patterns of gene expression in prostate cancer. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[34] A. Mirzabekov,et al. Clinical screening of gene rearrangements in childhood leukemia by using a multiplex polymerase chain reaction-microarray approach. , 2003, Clinical cancer research : an official journal of the American Association for Cancer Research.
[35] James M. Eldred,et al. Viral Discovery and Sequence Recovery Using DNA Microarrays , 2003, PLoS biology.
[36] J. Rowley,et al. Screening and quantification of multiple chromosome translocations in human leukemia. , 2003, Clinical chemistry.
[37] David Botstein,et al. Promoter-specific binding of Rap1 revealed by genome-wide maps of protein–DNA association , 2001, Nature Genetics.
[38] K. Pienta,et al. VCaP, a cell-based model system of human prostate cancer. , 2001, In vivo.
[39] M. Ladanyi,et al. Differential transactivation by alternative EWS-FLI1 fusion proteins correlates with clinical heterogeneity in Ewing's sarcoma. , 1999, Cancer research.
[40] D. Botstein,et al. Cluster analysis and display of genome-wide expression patterns. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[41] W. Gerald,et al. EWS-FLI1 fusion transcript structure is an independent determinant of prognosis in Ewing's sarcoma. , 1998, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.
[42] J. Rowley,et al. A method for the rapid sequence-independent amplification of microdissected chromosomal material. , 1992, Genomics.
[43] A. Chinnaiyan,et al. Recurrent gene fusions in prostate cancer , 2008, Nature Reviews Cancer.
[44] K. Garner,et al. Rapid detection of leukemia-associated translocation fusion genes using a novel combined RT-PCR and flow cytometric method , 2002, Leukemia.
[45] A. Mirzabekov,et al. Identification of chromosomal translocations in leukemias by hybridization with oligonucleotide microarrays. , 2002, Haematologica.
[46] P. Brown,et al. DNA arrays for analysis of gene expression. , 1999, Methods in enzymology.