APOE DNA methylation is altered in Lewy body dementia

[1]  J. Iga,et al.  DNA methylation changes at SNCA intron 1 in patients with dementia with Lewy bodies , 2017, Psychiatry and clinical neurosciences.

[2]  J. O'Brien,et al.  Exome sequencing in dementia with Lewy bodies , 2016, Translational Psychiatry.

[3]  J R Ecker,et al.  Human DNA methylomes of neurodegenerative diseases show common epigenomic patterns , 2016, Translational psychiatry.

[4]  S. Leurgans,et al.  Association of DNA methylation in the brain with age in older persons is confounded by common neuropathologies. , 2015, The international journal of biochemistry & cell biology.

[5]  J. Morris,et al.  Clinical features of Alzheimer disease with and without lewy bodies , 2015, Alzheimer's & Dementia.

[6]  Chang-En Yu,et al.  Epigenetic considerations of the APOE gene , 2015, Biomolecular concepts.

[7]  E. Mukaetova-Ladinska.  Molecular imaging biomarkers for dementia with Lewy bodies: an update , 2014, International Psychogeriatrics.

[8]  D. Bennett,et al.  Methylomic profiling implicates cortical deregulation of ANK1 in Alzheimer's disease , 2014, Nature Neuroscience.

[9]  Manolis Kellis,et al.  Alzheimer's disease: early alterations in brain DNA methylation at ANK1, BIN1, RHBDF2 and other loci , 2014 .

[10]  Manolis Kellis,et al.  Alzheimery's disease pathology is associated with early alterations in brain DNA methylation at ANK1, BIN1, RHBDF2 and other loci , 2014, Nature Neuroscience.

[11]  W. M. van der Flier,et al.  Genetic analysis implicates APOE, SNCA and suggests lysosomal dysfunction in the etiology of dementia with Lewy bodies , 2014, Human molecular genetics.

[12]  T. Montine,et al.  Epigenetic signature and enhancer activity of the human APOE gene. , 2013, Human molecular genetics.

[13]  Thomas Lengauer,et al.  A DNA methylation fingerprint of 1628 human samples. , 2011, Genome research.

[14]  E. Masliah,et al.  Alpha-synuclein sequesters Dnmt1 from the nucleus: a novel mechanism for epigenetic alterations in Lewy body diseases. , 2011, The Journal of biological chemistry.

[15]  S. Tsuji,et al.  CpG Demethylation Enhances Alpha-Synuclein Expression and Affects the Pathogenesis of Parkinson's Disease , 2010, PloS one.

[16]  Oliver Kaut,et al.  Methylation Regulates Alpha-Synuclein Expression and Is Decreased in Parkinson's Disease Patients' Brains , 2010, The Journal of Neuroscience.

[17]  C. Morris,et al.  BuChE‐K and APOE ϵ4 allele frequencies in Lewy body dementias, and influence of genotype and hyperhomocysteinemia on cognitive decline , 2009, Movement disorders : official journal of the Movement Disorder Society.

[18]  T. Montine,et al.  RESEARCH ARTICLE: Empiric Refinement of the Pathologic Assessment of Lewy‐Related Pathology in the Dementia Patient , 2008, Brain pathology.

[19]  E B Larson,et al.  Cognitive differences in dementia patients with autopsy-verified AD, Lewy body pathology, or both , 2005, Neurology.

[20]  O. Lopez,et al.  Genetic association between the APOE*4 allele and Lewy bodies in Alzheimer disease , 2005, Neurology.

[21]  Masanori Kato,et al.  Pathological entity of dementia with Lewy bodies and its differentiation from Alzheimer’s disease , 2004, Acta Neuropathologica.

[22]  J. Nutt,et al.  Age at onset of Parkinson disease and apolipoprotein E genotypes. , 2002, American journal of medical genetics.

[23]  Chang-En Yu,et al.  The APOE Gene is Differentially Methylated in Alzheimer's Disease. , 2015, Journal of Alzheimer's disease : JAD.