The coronary circulation in diabetes: influence of reactive oxygen species on K+ channel-mediated vasodilation.
暂无分享,去创建一个
[1] D. Gutterman,et al. High Glucose Impairs Voltage-Gated K+ Channel Current in Rat Small Coronary Arteries , 2001, Circulation research.
[2] S. J. Elliott,et al. Peroxynitrite reversibly inhibits Ca(2+)-activated K(+) channels in rat cerebral artery smooth muscle cells. , 2000, American journal of physiology. Heart and circulatory physiology.
[3] P. Vallance,et al. Nitric oxide and vascular responses in Type I diabetes , 2000, Diabetologia.
[4] D. Lamontagne,et al. Modification of vasodilator response in streptozotocin-induced diabetic rat , 1999 .
[5] S. Petrou,et al. Ca2+ influx inhibits voltage-dependent and augments Ca2+-dependent K+ currents in arterial myocytes. , 1999, American journal of physiology. Cell physiology.
[6] H. Miura,et al. Human coronary arteriolar dilation to bradykinin depends on membrane hyperpolarization: contribution of nitric oxide and Ca2+-activated K+ channels. , 1999, Circulation.
[7] P. Menasché,et al. Opening of potassium channels: the common cardioprotective link between preconditioning and natural hibernation? , 1999, Circulation.
[8] N. Brot,et al. Regulation of voltage‐dependent K+ channels by methionine oxidation: effect of nitric oxide and vitamin C , 1999, FEBS letters.
[9] Effects of hydroxyl radicals on KATP channels in guinea-pig ventricular myocytes , 1998, Pflügers Archiv.
[10] T. Rabelink,et al. Nitric oxide availability in diabetes mellitus. , 1998, Diabetes/metabolism reviews.
[11] Wen‐Hui Wang,et al. Reaction of nitric oxide with superoxide inhibits basolateral K+ channels in the rat CCD. , 1998, American journal of physiology. Cell physiology.
[12] G. Pieper. Review of alterations in endothelial nitric oxide production in diabetes: protective role of arginine on endothelial dysfunction. , 1998, Hypertension.
[13] G. King,et al. Protein kinase C activation and its role in the development of vascular complications in diabetes mellitus , 1997, Journal of Molecular Medicine.
[14] T. Lüscher,et al. Endothelial dysfunction in diabetes mellitus. , 1998, Journal of cardiovascular pharmacology.
[15] G. Santostasi,et al. Impairment of endothelium‐dependent but not of endothelium‐independent dilatation in guinea‐pig aorta rings incubated in the presence of elevated glucose , 1997, British journal of pharmacology.
[16] C. Kilo,et al. Cytosolic NADH/NAD+ , free radicals, and vascular dysfunction in early diabetes mellitus , 1997, Diabetologia.
[17] H. Kinoshita,et al. Role of potassium channels in relaxations of isolated canine basilar arteries to acidosis. , 1997, Stroke.
[18] J S Beckman,et al. Nitric oxide, superoxide, and peroxynitrite: the good, the bad, and ugly. , 1996, The American journal of physiology.
[19] K. Kamata,et al. Changes in superoxide dismutase mRNA expression by streptozotocin‐induced diabetes , 1996, British journal of pharmacology.
[20] W. Meng,et al. Global ischemia impairs ATP-sensitive K+ channel function in cerebral arterioles in piglets. , 1996, Stroke.
[21] J S Beckman,et al. Mechanisms of cerebral vasodilation by superoxide, hydrogen peroxide, and peroxynitrite. , 1996, The American journal of physiology.
[22] M. Kakei,et al. Direct activation of the ATP-sensitive potassium channel by oxygen free radicals in guinea-pig ventricular cells: its potentiation by MgADP. , 1996, Journal of molecular and cellular cardiology.
[23] J. Yoshikawa,et al. Possible involvement of phospholipase D and protein kinase C in vascular growth induced by elevated glucose concentration. , 1996, Hypertension.
[24] M. Arita,et al. Openings of cardiac KATP channel by oxygen free radicals produced by xanthine oxidase reaction. , 1996, The American journal of physiology.
[25] E. Aiello,et al. Regulation of 4-aminopyridine-sensitive, delayed rectifier K+ channels in vascular smooth muscle by phosphorylation. , 1996, Biochemistry and cell biology = Biochimie et biologie cellulaire.
[26] K. Heygate,et al. Impaired endothelium‐dependent relaxation in isolated resistance arteries of spontaneously diabetic rats , 1995, British journal of pharmacology.
[27] J. Kersten,et al. Impaired microvascular response to graded coronary occlusion in diabetic and hyperglycemic dogs. , 1995, The American journal of physiology.
[28] F. Cosentino,et al. Tetrahydrobiopterin and dysfunction of endothelial nitric oxide synthase in coronary arteries. , 1995, Circulation.
[29] J. Lombard,et al. Role of endothelium and arterial K+ channels in mediating hypoxic dilation of middle cerebral arteries. , 1994, The American journal of physiology.
[30] W. Mayhan. Effect of diabetes mellitus on response of the basilar artery to activation of ATP-sensitive potassium channels , 1994, Brain Research.
[31] F. Dai,et al. Diabetes-induced endothelial dysfunction in streptozotocin-treated rats: role of prostaglandin endoperoxides and free radicals. , 1993, Journal of the American Society of Nephrology : JASN.
[32] F. Faraci,et al. Responses of cerebral arterioles in diabetic rats to activation of ATP-sensitive potassium channels. , 1993, The American journal of physiology.
[33] J. Nyengaard,et al. Hyperglycemic Pseudohypoxia and Diabetic Complications , 1993, Diabetes.
[34] T. Lyons,et al. Glycation and oxidation: a role in the pathogenesis of atherosclerosis. , 1993, The American journal of cardiology.
[35] R. Cohen,et al. Free radicals mediate endothelial cell dysfunction caused by elevated glucose. , 1992, The American journal of physiology.
[36] M. Chiariello,et al. Cellular Electrophysiological Basis for Oxygen Radical‐Induced Arrhythmias: A Patch‐Clamp Study in Guinea Pig Ventricular Myocytes , 1991, Circulation.
[37] S. Wolff,et al. Oxidative glycation and free radical production: a causal mechanism of diabetic complications. , 1991, Free radical research communications.
[38] P. Rorsman,et al. ATP-Regulated K+ Channels and Diabetes Mellitus , 1990 .
[39] J. Daut,et al. Hypoxic dilation of coronary arteries is mediated by ATP-sensitive potassium channels. , 1990, Science.
[40] K. Kamata,et al. Functional changes in potassium channels in aortas from rats with streptozotocin-induced diabetes. , 1989, European journal of pharmacology.