Alternative complement pathway activation by Salmonella O polysaccharide as a virulence determinant in the mouse.

[1]  S. Gordon,et al.  Interaction of human monocytes, macrophages, and polymorphonuclear leukocytes with zymosan in vitro. Role of type 3 complement receptors and macrophage-derived complement. , 1985, The Journal of clinical investigation.

[2]  Robert B Sim,et al.  Macrophage complement and lectin-like receptors bind Leishmania in the absence of serum , 1985, The Journal of experimental medicine.

[3]  E. Kellenberger,et al.  Capsule of Escherichia coli K29: ultrastructural preservation and immunoelectron microscopy , 1985, Journal of bacteriology.

[4]  R. van Furth,et al.  Differences in initial rate of intracellular killing of Salmonella typhimurium by resident peritoneal macrophages from various mouse strains. , 1985, Journal of immunology.

[5]  G. Barnickel,et al.  High state of order of isolated bacterial lipopolysaccharide and its possible contribution to the permeation barrier property of the outer membrane , 1985, Journal of bacteriology.

[6]  J. Blackwell,et al.  Role of macrophage complement and lectin-like receptors in binding Leishmania parasites to host macrophages. , 1985, Immunology letters.

[7]  M. Hovi,et al.  Lipopolysaccharide and mouse virulence of Salmonella: O antigen is important after intraperitoneal but not intravenous challenge , 1984 .

[8]  O. Mäkelä,et al.  Isotype of protective anti-Salmonella antibodies in experimental mouse salmonellosis , 1984, Infection and immunity.

[9]  E. Lehtonen,et al.  Cell relationships during aggregation between preimplantation embryos and teratocarcinoma-derived cells. , 1984, Journal of embryology and experimental morphology.

[10]  S. Gordon,et al.  Local opsonization by secreted macrophage complement components. Role of receptors for complement in uptake of zymosan , 1984, The Journal of experimental medicine.

[11]  K. Joiner,et al.  C3 binding but not its breakdown is affected by the structure of lipopolysaccharide (LPS) on the surface of Salmonellae , 1984 .

[12]  L. Leive,et al.  Complement activation via the alternative pathway by purified Salmonella lipopolysaccharide is affected by its structure but not its O-antigen length. , 1984, Journal of immunology.

[13]  E. Johnson,et al.  Complement (C3)‐Receptor‐Mediated Phagocytosis of Agarose Beads by Mouse Macrophages , 1983, Scandinavian journal of immunology.

[14]  P. Mäkelä,et al.  Complement activation by polysaccharide of lipopolysaccharide: an important virulence determinant of salmonellae , 1983, Infection and immunity.

[15]  L. Leive,et al.  Salmonellae activate complement differentially via the alternative pathway depending on the structure of their lipopolysaccharide O-antigen. , 1983, Journal of immunology.

[16]  O. Mäkelä,et al.  The protective capacity of immune sera in experimental mouse salmonellosis is mainly due to IgM antibodies. , 1982, Immunology letters.

[17]  M. Frank,et al.  Studies on the mechanism of bacterial resistance to complement-mediated killing. I. Terminal complement components are deposited and released from salmonella minnesota S218 without causing bacterial death , 1982, The Journal of experimental medicine.

[18]  P. Mäkelä,et al.  Phagocytosis of bacteria by macrophages: changing the carbohydrate of lipopolysaccharide alters interaction with complement and macrophages. , 1982, Journal of immunology.

[19]  D. Briles,et al.  Phagocytosis and killing of salmonella typhimurium by peritoneal exudate cells , 1981, Infection and immunity.

[20]  Mark J. Miller,et al.  Evidence for receptor-mediated binding of glycoproteins, glycoconjugates, and lysosomal glycosidases by alveolar macrophages. , 1978, Proceedings of the National Academy of Sciences of the United States of America.

[21]  M. Valtonen.  Role of phagocytosis in mouse virulence of Salmonella typhimurium recombinants with O antigen 6,7 or 4,12 , 1977, Infection and immunity.

[22]  M. Sarvas,et al.  Effect of polymyxin on the ultrastructure of the outer membrane of wild-type and polymyxin-resistant strain of Salmonella , 1976, Journal of bacteriology.

[23]  V. Valtonen,et al.  Effect of the quality of the lipopolysaccharide on mouse virulence of Salmonella enteritidis , 1975, Infection and immunity.

[24]  M. Listgarten,et al.  Serology of Streptococcus sanguis: Localization of Antigens with Unlabeled Antisera , 1973, Infection and immunity.

[25]  P. Gemski,et al.  Non-smooth mutants of Salmonella typhimurium: differentiation by phage sensitivity and genetic mapping. , 1972, Journal of general microbiology.

[26]  Jeffrey H. Miller.  Experiments in molecular genetics , 1972 .

[27]  Mäkelä Ph,et al.  The Effect of Lipopolysaccharide Modifications – Antigenic Factors 1, 5, 122 and 27-on the Virulence of Salmonella Strains for Mice , 1971 .

[28]  V. Valtonen,et al.  The smooth lipopolysaccharide character of 1,4,(5),12 and 1,9,12 transductants formed as hybrids between groups B and D of salmonella. , 1971, European journal of biochemistry.

[29]  J. Shands.  CHAPTER 3 – The Physical Structure of Bacterial Lipopolysaccharides , 1971 .

[30]  V. Valtonen.  Mouse virulence of Salmonella strains: the effect of different smooth-type O side-chains. , 1970, Journal of general microbiology.

[31]  S. Muñoz,et al.  Ability of Staphylococcus aureus Cells to Bind Normal Human Immunoglobulin G , 1970 .

[32]  M. Nakano,et al.  Chemical Components in the Cell Wall of Salmonella typhimurium affecting its Virulence and Immunogenicity in Mice , 1969, Nature.

[33]  F. Collins.  Effect of Specific Immune Mouse Serum on the Growth of Salmonella enteritidis in Nonvaccinated Mice Challenged by Various Routes , 1969, Journal of bacteriology.

[34]  R. J. Roantree.  Salmonella O antigens and virulence. , 1967, Annual Review of Microbiology.

[35]  K. Turner,et al.  Number of Molecules of Antibody Required to Promote Phagocytosis of One Bacterium , 1966, Nature.

[36]  C. Jenkin,et al.  BASIS FOR IMMUNITY TO TYPHOID IN MICE AND THE QUESTION OF "CELLULAR IMMUNITY". , 1963, Bacteriological reviews.

[37]  M. Nakano.  An Electron Microscopic Study of the Liver of Mice infected with Salmonella enteritidis , 1961 .

[38]  R. J. Roantree,et al.  A STUDY OF THE RELATIONSHIP OF THE NORMAL BACTERICIDAL ACTIVITY OF HUMAN SERUM TO BACTERIAL INFECTION. , 1960, The Journal of clinical investigation.

[39]  J. Whitby,et al.  The role of macrophages in the elimination of bacteria from the mouse peritoneum. , 1959, British journal of experimental pathology.

[40]  D. M. Donaldson,et al.  Lack of bactericidal effect of mouse serum on a number of common microorganisms. , 1954, Science.