Disease progression among 446 children with newly diagnosed type 1 diabetes located in Scandinavia, Europe, and North America during the last 27 yr
暂无分享,去创建一个
P. Hougaard | C. Beam | H. Mortensen | D. Becker | J. Petersen | C. Greenbaum | J. Svensson | Sven Pörksen | L. Hansen | L. Nielsen | M. M. Max Andersen | J. Petersen | Marie Louise C Max Andersen
[1] R. Hamman,et al. Clinical evolution of beta cell function in youth with diabetes: the SEARCH for Diabetes in Youth study , 2012, Diabetologia.
[2] Å. Lernmark,et al. Association between autoantibodies to the Arginine variant of the Zinc transporter 8 (ZnT8) and stimulated C‐peptide levels in Danish children and adolescents with newly diagnosed type 1 diabetes , 2012, Pediatric diabetes.
[3] M. Atkinson,et al. Formation of a human β-cell population within pancreatic islets is set early in life. , 2012, The Journal of clinical endocrinology and metabolism.
[4] P. Bingley,et al. Trends in childhood type 1 diabetes incidence in Europe during 1989–2008: evidence of non-uniformity over time in rates of increase , 2012, Diabetologia.
[5] M. Knip. Descriptive epidemiology of type 1 diabetes—is it still in? , 2012, Diabetologia.
[6] M. Rewers,et al. The interplay of autoimmunity and insulin resistance in type 1 diabetes. , 2012, Discovery medicine.
[7] Darrell M. Wilson,et al. Co-stimulation modulation with abatacept in patients with recent-onset type 1 diabetes: a randomised, double-blind, placebo-controlled trial , 2011, The Lancet.
[8] L. Nyström,et al. Incidence of Type 1 Diabetes in Sweden Among Individuals Aged 0–34 Years, 1983–2007 , 2011, Diabetes Care.
[9] M. Blössner,et al. Global prevalence and trends of overweight and obesity among preschool children. , 2010, The American journal of clinical nutrition.
[10] Darrell M. Wilson,et al. Failure to Preserve β-Cell Function With Mycophenolate Mofetil and Daclizumab Combined Therapy in Patients With New- Onset Type 1 Diabetes , 2010, Diabetes Care.
[11] Darrell M. Wilson,et al. Rituximab, B-lymphocyte depletion, and preservation of beta-cell function. , 2009, The New England journal of medicine.
[12] C. de Beaufort,et al. Multinational study in children and adolescents with newly diagnosed type 1 diabetes: association of age, ketoacidosis, HLA status, and autoantibodies on residual beta‐cell function and glycemic control 12 months after diagnosis , 2009, Pediatric diabetes.
[13] H. Mortensen,et al. Long‐term trends in the incidence of type 1 diabetes in Denmark: the seasonal variation changes over time , 2009, Pediatric diabetes.
[14] J. Krischer,et al. Glucose and C-Peptide Changes in the Perionset Period of Type 1 Diabetes in the Diabetes Prevention Trial–Type 1 , 2008, Diabetes Care.
[15] M. Nordwall,et al. Clinical manifestations and beta cell function in Swedish diabetic children have remained unchanged during the last 25 years , 2008, Diabetes/metabolism research and reviews.
[16] R. Rizza,et al. β-Cell Replication Is the Primary Mechanism Subserving the Postnatal Expansion of β-Cell Mass in Humans , 2008, Diabetes.
[17] R. Mirmira. Faculty Opinions recommendation of Beta-cell replication is the primary mechanism subserving the postnatal expansion of beta-cell mass in humans. , 2008 .
[18] J. Tuomilehto,et al. Incidence and trends of childhood Type 1 diabetes worldwide 1990–1999 , 2006, Diabetic medicine : a journal of the British Diabetic Association.
[19] J. Krischer,et al. Patterns of metabolic progression to type 1 diabetes in the Diabetes Prevention Trial-Type 1. , 2006, Diabetes care.
[20] G. Dahlquist,et al. Time trend of childhood type 1 diabetes incidence in Lithuania and Sweden, 1983–2000 , 2004, Acta paediatrica.
[21] C. Berne,et al. Temporary Preservation of β-Cell Function by Diazoxide Treatment in Childhood Type 1 Diabetes , 2004 .
[22] J. She,et al. Preservation of C‐peptide secretion in subjects at high risk of developing type 1 diabetes mellitus – a new surrogate measure of non‐progression? , 2004, Pediatric diabetes.
[23] M. Steffes,et al. Beta-cell function and the development of diabetes-related complications in the diabetes control and complications trial. , 2003, Diabetes care.
[24] T. Wilkin. The accelerator hypothesis: weight gain as the missing link between Type I and Type II diabetes , 2001, Diabetologia.
[25] C. Mølgaard,et al. Body mass index of 0 to 45-y-old Danes: reference values and comparison with published European reference values , 2001, International Journal of Obesity.
[26] T. Cole,et al. Establishing a standard definition for child overweight and obesity worldwide: international survey , 2000, BMJ : British Medical Journal.
[27] J. Ilonen,et al. Autoantibodies associated with Type I diabetes mellitus persist after diagnosis in children , 1998, Diabetologia.
[28] E. Bognetti,et al. Residual beta-cell function and spontaneous clinical remission in type 1 diabetes mellitus: the role of puberty , 1998, Acta Diabetologica.
[29] Å. Lernmark,et al. Glutamate decarboxylase antibody levels predict rate of beta-cell decline in adult-onset diabetes. , 1995, Diabetes research and clinical practice.
[30] J. Jeppsson,et al. Improved Method for Analysis of Glycated Haemoglobin by Ion Exchange Chromatography , 1994, Annals of clinical biochemistry.
[31] Å. Lernmark,et al. Improved Specificity of ICA Assays in the Fourth International Immunology of Diabetes Serum Exchange Workshop , 1992, Diabetes.
[32] Å. Lernmark,et al. Factors influencing the magnitude, duration, and rate of fall of B-cell function in Type 1 (insulin-dependent) diabetic children followed for two years from their clinical diagnosis , 1988, Diabetologia.
[33] E. Andersen,et al. [Heights and weights of Danish children]. , 1982, Ugeskrift for laeger.
[34] Darrell M. Wilson,et al. Failure to Preserve beta-cell function with Mycophenolate Mofetil and Daclizumab Combined Therapy in patients with new onset Type 1 Diabetes Running title: MMF and DZB in New Onset T1D , 2010 .
[35] G. Dahlquist. Can we slow the rising incidence of childhood-onset autoimmune diabetes? The overload hypothesis , 2005, Diabetologia.
[36] C. Berne,et al. Temporary preservation of beta-cell function by diazoxide treatment in childhood type 1 diabetes. , 2004, Diabetes care.
[37] Effect of intensive therapy on residual beta-cell function in patients with type 1 diabetes in the diabetes control and complications trial. A randomized, controlled trial. The Diabetes Control and Complications Trial Research Group. , 1998, Annals of internal medicine.
[38] Effects of age, duration and treatment of insulin-dependent diabetes mellitus on residual beta-cell function: observations during eligibility testing for the Diabetes Control and Complications Trial (DCCT). The DCCT Research Group. , 1987, The Journal of clinical endocrinology and metabolism.