Switch from Myc/Max to Mad1/Max binding and decrease in histone acetylation at the telomerase reverse transcriptase promoter during differentiation of HL60 cells
暂无分享,去创建一个
Nikita Popov | Qian Wang | M. Henriksson | M. Björkholm | Dawei Xu | M. Hou | A. Gruber | N. Popov | A. Menkel | Dawei Xu | Mi Hou | Magnus Björkholm | Astrid Gruber | Annette R. Menkel | Marie Henriksson | Qian Wang
[1] A. Zetterberg,et al. Frequent amplification of the telomerase reverse transcriptase gene in human tumors. , 2000, Cancer research.
[2] Gregory J. Hannon,et al. Cell biology: Risky immortalization by telomerase , 2000, Nature.
[3] Y. Cong,et al. Histone Deacetylation Is Involved in the Transcriptional Repression of hTERT in Normal Human Cells* , 2000, The Journal of Biological Chemistry.
[4] L. Chin,et al. Role for N-CoR and histone deacetylase in Sin3-mediated transcriptional repression , 1997, nature.
[5] C. Glass,et al. A complex containing N-CoR, mSln3 and histone deacetylase mediates transcriptional repression , 1997, nature.
[6] C. Englert,et al. Expression of the hTERT gene is regulated at the level of transcriptional initiation and repressed by Mad1. , 2000, Cancer research.
[7] R. DePinho,et al. Repression by the Mad(Mxi1)‐Sin3 complex , 1998, BioEssays : news and reviews in molecular, cellular and developmental biology.
[8] B. Lüscher,et al. Inhibition of Proliferation and Apoptosis by the Transcriptional Repressor Mad1 , 2000, The Journal of Biological Chemistry.
[9] Stuart L Schreiber,et al. Histone Deacetylase Activity Is Required for Full Transcriptional Repression by mSin3A , 1997, Cell.
[10] R. Roeder,et al. The adenovirus major late transcription factor USF is a member of the helix-loop-helix group of regulatory proteins and binds to DNA as a dimer. , 1990, Genes & development.
[11] L. Larsson,et al. Expression of mad, mxi1, max and c-myc during induced differentiation of hematopoietic cells: opposite regulation of mad and c-myc. , 1994, Oncogene.
[12] K. Struhl. Histone acetylation and transcriptional regulatory mechanisms. , 1998, Genes & development.
[13] V. Lundblad,et al. The telomerase reverse transcriptase: components and regulation. , 1998, Genes & development.
[14] C. Harley,et al. Extension of life-span by introduction of telomerase into normal human cells. , 1998, Science.
[15] C. Asker,et al. Inhibition of cell growth and apoptosis by inducible expression of the transcriptional repressor Mad1. , 2000, Experimental cell research.
[16] M. Spitz,et al. Associations among telomerase activity, p53 protein overexpression, and genetic instability in lung cancer , 1999, British Journal of Cancer.
[17] M. Henriksson,et al. Proteins of the Myc network: essential regulators of cell growth and differentiation. , 1996, Advances in cancer research.
[18] R. Weinberg,et al. hEST2, the Putative Human Telomerase Catalytic Subunit Gene, Is Up-Regulated in Tumor Cells and during Immortalization , 1997, Cell.
[19] P. Marks,et al. Histone deacetylase inhibitors: inducers of differentiation or apoptosis of transformed cells. , 2000, Journal of the National Cancer Institute.
[20] R. Paro,et al. Analysis of chromatin structure by in vivo formaldehyde cross-linking. , 1997, Methods.
[21] G. Roos,et al. Telomeres and telomerase in normal and malignant haematopoietic cells. , 1997, European journal of cancer.
[22] P. Pisa,et al. Telomerase activity and the expression of telomerase components in acute myelogenous leukaemia , 1998, British journal of haematology.
[23] R. Dalla‐Favera,et al. Direct activation of TERT transcription by c-MYC , 1999, Nature Genetics.
[24] M. Yutsudo,et al. Cloning of human telomerase catalytic subunit (hTERT) gene promoter and identification of proximal core promoter sequences essential for transcriptional activation in immortalized and cancer cells. , 1999, Cancer research.
[25] R. Eisenman,et al. Mad-max transcriptional repression is mediated by ternary complex formation with mammalian homologs of yeast repressor Sin3 , 1995, Cell.
[26] W. Klapper,et al. Regulation of telomerase activity in quiescent immortalized human cells. , 1998, Biochimica et biophysica acta.
[27] D. Ledbetter,et al. Rox, a novel bHLHZip protein expressed in quiescent cells that heterodimerizes with Max, binds a non‐canonical E box and acts as a transcriptional repressor , 1997, The EMBO journal.
[28] L. Chin,et al. Telomerase reverse transcriptase gene is a direct target of c-Myc but is not functionally equivalent in cellular transformation , 1999, Oncogene.
[29] M. Cole,et al. The Essential Cofactor TRRAP Recruits the Histone Acetyltransferase hGCN5 to c-Myc , 2000, Molecular and Cellular Biology.
[30] J F Barrett,et al. Identification of CDK4 as a target of c-MYC. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[31] E. Kremmer,et al. Cell growth inhibition by the Mad/Max complex through recruitment of histone deacetylase activity , 1997, Current Biology.
[32] Wen‐Ming Yang,et al. Histone Deacetylases Associated with the mSin3 Corepressor Mediate Mad Transcriptional Repression , 1997, Cell.
[33] R. Eisenman,et al. Mnt, a novel Max-interacting protein is coexpressed with Myc in proliferating cells and mediates repression at Myc binding sites. , 1997, Genes & development.
[34] C B Harley,et al. Telomerase catalytic subunit homologs from fission yeast and human. , 1997, Science.
[35] A. Wolffe. Sinful repression , 1997, nature.
[36] Young-Hwa Song,et al. Identification of Mad as a repressor of the human telomerase (hTERT) gene , 2000, Oncogene.
[37] P. Farnham,et al. Direct Examination of Histone Acetylation on Myc Target Genes Using Chromatin Immunoprecipitation* , 2000, The Journal of Biological Chemistry.
[38] C. Harley,et al. Telomerase, cell immortality, and cancer. , 1994, Cold Spring Harbor symposia on quantitative biology.
[39] W. Ansorge,et al. Direct induction of cyclin D2 by Myc contributes to cell cycle progression and sequestration of p27 , 1999, The EMBO journal.
[40] L. Chin,et al. An amino-terminal domain of Mxi1 mediates anti-myc oncogenic activity and interacts with a homolog of the Yeast Transcriptional Repressor SIN3 , 1995, Cell.
[41] Y. Cong,et al. The human telomerase catalytic subunit hTERT: organization of the gene and characterization of the promoter. , 1999, Human molecular genetics.
[42] G. Hannon,et al. Myc activates telomerase. , 1998, Genes & development.
[43] C B Harley,et al. Specific association of human telomerase activity with immortal cells and cancer. , 1994, Science.
[44] M. Cole,et al. The Myc oncoprotein: a critical evaluation of transactivation and target gene regulation , 1999, Oncogene.