Molecular Characterization of a Mouse Genomic Element Mobilized by Advanced Glycation Endproduct Modified-DNA (AGE-DNA)

[1]  J. Sambrook,et al.  Molecular Cloning: A Laboratory Manual , 2001 .

[2]  M. Brenneman,et al.  Repair of site-specific double-strand breaks in a mammalian chromosome by homologous and illegitimate recombination , 1997, Molecular and cellular biology.

[3]  M. Seldin,et al.  Human/mouse homology relationships. , 1996, Genomics.

[4]  R. Bucala,et al.  Identification of N2-(1-carboxyethyl)guanine (CEG) as a guanine advanced glycosylation end product. , 1995, Biochemistry.

[5]  W. W. Nichols,et al.  Minisatellite DNA-binding proteins in mouse brain, liver, and kidney. , 1994, Experimental cell research.

[6]  R. Elliott,et al.  Characterization of murine middle repetitive DNA. , 1993, DNA and cell biology.

[7]  A. Lee,et al.  Transposition of an Alu-containing element induced by DNA-advanced glycosylation endproducts. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[8]  A. Cerami,et al.  Induction of gamma delta transposition in response to elevated glucose-6-phosphate levels. , 1991, Mutation research.

[9]  A. Cerami,et al.  Industion of γδ transposition in response to elevated glucose-6-phosphate levels , 1991 .

[10]  R. Berger,et al.  Structural alterations of the BCR and ABL genes in Ph1 positive acute leukemias with rearrangements in the BCR gene first intron: further evidence implicating Alu sequences in the chromosome translocation. , 1989, Nucleic acids research.

[11]  D. Lipman,et al.  Improved tools for biological sequence comparison. , 1988, Proceedings of the National Academy of Sciences of the United States of America.

[12]  M. Seldin,et al.  Genetic analysis of autoimmune gld mice. I. Identification of a restriction fragment length polymorphism closely linked to the gld mutation within a conserved linkage group , 1988, The Journal of experimental medicine.

[13]  A. Cerami,et al.  Elevated glucose 6-phosphate levels are associated with plasmid mutations in vivo. , 1987, Proceedings of the National Academy of Sciences of the United States of America.

[14]  N. Heisterkamp,et al.  Molecular analysis of both translocation products of a Philadelphia-positive CML patient. , 1986, Nucleic acids research.

[15]  M. Oishi,et al.  A repetitive DNA family (Sau3A family) in human chromosomes: extrachromosomal DNA and DNA polymorphism. , 1986, Proceedings of the National Academy of Sciences of the United States of America.

[16]  R. Bucala,et al.  Modification of DNA by glucose 6-phosphate induces DNA rearrangements in an Escherichia coli plasmid. , 1985, Proceedings of the National Academy of Sciences of the United States of America.

[17]  B. Calabretta,et al.  Genome instability in a region of human DNA enriched in Alu repeat sequences , 1982, Nature.

[18]  M. Oishi,et al.  Preference of the recombination sites involved in the formation of extrachromosomal copies of the human alphoid Sau3A repeat family. , 1995, Nucleic acids research.

[19]  R. Bucala,et al.  Advanced glycosylation: chemistry, biology, and implications for diabetes and aging. , 1992, Advances in pharmacology.

[20]  D. Bishop,et al.  The information content of phase-known matings for ordering genetic loci. , 1985, Genetic epidemiology.

[21]  C. Yanisch-Perron,et al.  Improved M13 phage cloning vectors and host strains: nucleotide sequences of the M13mp18 and pUC19 vectors. , 1985, Gene.

[22]  R. Bucala,et al.  Modification of DNA by reducing sugars: a possible mechanism for nucleic acid aging and age-related dysfunction in gene expression. , 1984, Proceedings of the National Academy of Sciences of the United States of America.

[23]  E. L. Green Linkage, recombination and mapping , 1981 .