Delayed minocycline but not delayed mild hypothermia protects against embolic stroke
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Tao Yang | A. Shuaib | C. X. Wang | Raza Noor
[1] S. Paul,et al. Minocycline prevents nigrostriatal dopaminergic neurodegeneration in the MPTP model of Parkinson's disease , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[2] S. Paul,et al. Minocycline blocks nitric oxide-induced neurotoxicity by inhibition p38 MAP kinase in rat cerebellar granule neurons , 2001, Neuroscience Letters.
[3] C. Wang,et al. A focal embolic model of cerebral ischemia in rats: introduction and evaluation. , 2001, Brain research. Brain research protocols.
[4] B. Fiebich,et al. Minocycline, a Tetracycline Derivative, Is Neuroprotective against Excitotoxicity by Inhibiting Activation and Proliferation of Microglia , 2001, The Journal of Neuroscience.
[5] K. Todd,et al. Patency of Cerebral Microvessels after Focal Embolic Stroke in the Rat , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[6] G K Steinberg,et al. Delayed induction and long-term effects of mild hypothermia in a focal model of transient cerebral ischemia: neurological outcome and infarct size. , 2001, Journal of neurosurgery.
[7] P. Chan,et al. A tetracycline derivative, minocycline, reduces inflammation and protects against focal cerebral ischemia with a wide therapeutic window. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[8] RobertSchmid-Elsaesser,et al. Combination Drug Therapy and Mild Hypothermia , 1999 .
[9] H. Reulen,et al. Combination drug therapy and mild hypothermia: a promising treatment strategy for reversible, focal cerebral ischemia. , 1999, Stroke.
[10] R. Busto,et al. Postischemic Hypothermia and IL-10 Treatment Provide Long-Lasting Neuroprotection of CA1 Hippocampus Following Transient Global Ischemia in Rats , 1999, Experimental Neurology.
[11] T. Hökfelt,et al. Tetracyclines inhibit microglial activation and are neuroprotective in global brain ischemia. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[12] A. Shuaib,et al. Quantification of infarct size on focal cerebral ischemia model of rats using a simple and economical method , 1998, Journal of Neuroscience Methods.
[13] G. Steinberg,et al. Optimal depth and duration of mild hypothermia in a focal model of transient cerebral ischemia: effects on neurologic outcome, infarct size, apoptosis, and inflammation. , 1998, Stroke.
[14] F. Barone,et al. The Role of Cytokines in the Neuropathology of Stroke and Neurotrauma , 1998, Neuroimmunomodulation.
[15] K. Becker. Inflammation and acute stroke. , 1998, Current opinion in neurology.
[16] D. Corbett,et al. Postischemic hypothermia. A critical appraisal with implications for clinical treatment. , 1997, Molecular neurobiology.
[17] CiceroCoimbra,et al. Long-lasting Neuroprotective Effect of Postischemic Hypothermia and Treatment With an Anti-inflammatory/Antipyretic Drug , 1996 .
[18] T. Wieloch,et al. Long-lasting neuroprotective effect of postischemic hypothermia and treatment with an anti-inflammatory/antipyretic drug. Evidence for chronic encephalopathic processes following ischemia. , 1996, Stroke.
[19] A. Shuaib,et al. CGS-19755 is neuroprotective during repetitive ischemia: This effect is significantly enhanced when combined with hypothermia , 1993, Neuroscience.
[20] A. Shuaib,et al. During Repetitive Forebrain Ischemia, Post-ischemic Hypothermia Protects Neurons from Damage , 1992, Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques.
[21] R. A. Solomon,et al. Reduction by delayed hypothermia of cerebral infarction following middle cerebral artery occlusion in the rat: a time-course study. , 1992, Journal of neurosurgery.
[22] L. Ng,et al. Massive Cerebral Infarction with Severe Brain Swelling: A CLINICOPATHOLOGICAL STUDY , 1970, Stroke.