A role for retinoblastoma protein in potentiating transcriptional activation by the glucocorticoid receptor
暂无分享,去创建一个
[1] T. Smith,et al. Prediction of similar transforming regions in simian virus 40 large T, adenovirus E1A, and myc oncoproteins , 1988, Journal of virology.
[2] Thomas C. Kaufman,et al. brahma: A regulator of Drosophila homeotic genes structurally related to the yeast transcriptional activator SNF2 SWI2 , 1992, Cell.
[3] C. Kedinger,et al. Rb may act as a transcriptional co-activator in undifferentiated F9 cells. , 1993, Oncogene.
[4] K. Münger,et al. The state of the p53 and retinoblastoma genes in human cervical carcinoma cell lines. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[5] W. Lee,et al. C-terminal truncation of the retinoblastoma gene product leads to functional inactivation. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[6] Phang-lang Chen,et al. Phosphorylation of the retinoblastoma gene product is modulated during the cell cycle and cellular differentiation , 1989, Cell.
[7] P. Humphrey,et al. Sp-1 binds promoter elements regulated by the RB protein and Sp-1-mediated transcription is stimulated by RB coexpression. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[8] C. Anderson,et al. The retinoblastoma protein is phosphorylated on multiple sites by human cdc2. , 1991, The EMBO journal.
[9] Paul A. Khavari,et al. BRG1 contains a conserved domain of the SWI2/SNF2 family necessary for normal mitotic growth and transcription , 1993, Nature.
[10] E. Harlow,et al. Cellular targets for transformation by the adenovirus E1A proteins , 1989, Cell.
[11] Xinyang Zheng,et al. Cell transformation and activation of pp60c-src by overexpression of a protein tyrosine phosphatase , 1992, Nature.
[12] Chun-Ming Huang,et al. SV40 large T antigen binds preferentially to an underphosphorylated member of the retinoblastoma susceptibility gene product family , 1989, Cell.
[13] I. Herskowitz,et al. Characterization of the yeast SWI1, SWI2, and SWI3 genes, which encode a global activator of transcription , 1992, Cell.
[14] David M. Livingston,et al. The product of the retinoblastoma susceptibility gene has properties of a cell cycle regulatory element , 1989, Cell.
[15] S. Fields,et al. A novel genetic system to detect proteinprotein interactions , 1989, Nature.
[16] Michael R. Green,et al. Retinoblastoma gene product activates expression of the human TGF-β2 gene through transcription factor ATF-2 , 1992, Nature.
[17] I. Herskowitz,et al. Roles of SWI1, SWI2, and SWI3 proteins for transcriptional enhancement by steroid receptors. , 1992, Science.
[18] E. Harlow,et al. The retinoblastoma protein is phosphorylated during specific phases of the cell cycle , 1989, Cell.
[19] S. Elledge,et al. The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases , 1993, Cell.
[20] Pearl S Huang,et al. Cloning of cDNAs for cellular proteins that bind to the retinoblastoma gene product , 1991, Nature.
[21] M. Sporn,et al. Regulation of transforming growth factor beta 1 gene expression by the product of the retinoblastoma-susceptibility gene. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[22] B. Gallie,et al. Evidence for retinoblastoma protein (RB) dependent and independent IFN-gamma responses: RB coordinately rescues IFN-gamma induction of MHC class II gene transcription in noninducible breast carcinoma cells. , 1994, Oncogene.
[23] M. Yaniv,et al. A human homologue of Saccharomyces cerevisiae SNF2/SWI2 and Drosophila brm genes potentiates transcriptional activation by the glucocorticoid receptor. , 1993, The EMBO journal.
[24] M. Botchan,et al. The retinoblastoma gene product regulates Sp1-mediated transcription , 1992, Molecular and cellular biology.
[25] Michael R. Green,et al. Promoter targeting by adenovirus E1a through interaction with different cellular DNA-binding domains , 1994, Nature.