Sex differences in caspase activation after stroke.
暂无分享,去创建一个
Jun Li | Fudong Liu | Louise D McCullough | L. McCullough | C. Siegel | Fudong Liu | Zhong Li | Zhong Li | Jun Li | Chad Siegel | Rongwen Yuan | Rongwen Yuan
[1] Ruedi Aebersold,et al. Molecular characterization of mitochondrial apoptosis-inducing factor , 1999, Nature.
[2] N. Plesnila,et al. Caspase activation and neuroprotection in caspase-3- deficient mice after in vivo cerebral ischemia and in vitro oxygen glucose deprivation , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[3] T. Kadar,et al. Aging has a complex effect on a rat model of ischemic stroke , 2002, Brain Research.
[4] R. Ratcheson,et al. Caspase-9 Inhibition after Focal Cerebral Ischemia Improves Outcome following Reversible Focal Ischemia , 2002, Metabolic Brain Disease.
[5] T. Dawson,et al. Mediation of Poly(ADP-Ribose) Polymerase-1-Dependent Cell Death by Apoptosis-Inducing Factor , 2002, Science.
[6] K. Furie,et al. Heart disease and stroke statistics--2007 update: a report from the American Heart Association Statistics Committee and Stroke Statistics Subcommittee. , 2008, Circulation.
[7] L. McCullough,et al. Pharmacological Inhibition of AMP-activated Protein Kinase Provides Neuroprotection in Stroke* , 2005, Journal of Biological Chemistry.
[8] P. Hurn,et al. Estrogen as a Neuroprotectant in Stroke , 2000, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[9] Simon C Watkins,et al. Innate Gender-based Proclivity in Response to Cytotoxicity and Programmed Cell Death Pathway* , 2004, Journal of Biological Chemistry.
[10] L. Stefanis. Caspase-Dependent and -Independent Neuronal Death: Two Distinct Pathways to Neuronal Injury , 2005, The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry.
[11] L. McCullough,et al. Ischemic Nitric Oxide and Poly (ADP-Ribose) Polymerase-1 in Cerebral Ischemia: Male Toxicity, Female Protection , 2005, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[12] N. Plesnila,et al. Apoptosis-Inducing Factor Triggered by Poly(ADP-Ribose) Polymerase and Bid Mediates Neuronal Cell Death after Oxygen-Glucose Deprivation and Focal Cerebral Ischemia , 2005, The Journal of Neuroscience.
[13] Michael M. Wang,et al. Sex differences in cell death , 2005, Annals of neurology.
[14] D. Mozaffarian,et al. Heart disease and stroke statistics--2009 update: a report from the American Heart Association Statistics Committee and Stroke Statistics Subcommittee. , 2009, Circulation.
[15] Junying Yuan,et al. Cleavage of BID by Caspase 8 Mediates the Mitochondrial Damage in the Fas Pathway of Apoptosis , 1998, Cell.
[16] D. Duverger,et al. The Quantification of Cerebral Infarction following Focal Ischemia in the Rat: Influence of Strain, Arterial Pressure, Blood Glucose Concentration, and Age , 1988, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[17] F. Moroni. Poly(ADP-ribose)polymerase 1 (PARP-1) and postischemic brain damage. , 2008, Current opinion in pharmacology.
[18] I. Ferrer. Signaling of Cell Death and Cell Survival Following Focal Cerebral Ischemia: Life and Death Struggle in the Penumbra , 2003, Journal of neuropathology and experimental neurology.
[19] M. Johnston,et al. Sex and the pathogenesis of cerebral palsy , 2006, Developmental medicine and child neurology.
[20] R. Traystman,et al. Differential Effect of PARP-2 Deletion on Brain Injury after Focal and Global Cerebral Ischemia , 2006, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[21] C. Bushnell. Stroke and the female brain , 2008, Nature Clinical Practice Neurology.
[22] John Robbins,et al. National cross sectional survey to determine whether the decision to delivery interval is critical in emergency caesarean section , 2004, BMJ : British Medical Journal.
[23] M. Moskowitz,et al. Attenuation of Delayed Neuronal Death after Mild Focal Ischemia in Mice by Inhibition of the Caspase Family , 1998, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[24] J. Mariani,et al. Specific caspase inhibitor Q‐VD‐OPh prevents neonatal stroke in P7 rat: a role for gender , 2007, Journal of neurochemistry.
[25] M. Moskowitz,et al. Effects of cerebral ischemia in mice deficient in neuronal nitric oxide synthase. , 1994, Science.
[26] C. Iadecola,et al. Caspase inhibitors reduce neuronal injury after focal but not global cerebral ischemia in rats. , 2000, Stroke.
[27] L. McCullough,et al. Pathways to ischemic neuronal cell death: are sex differences relevant? , 2008, Journal of Translational Medicine.
[28] J. Kemp,et al. Is caspase-3 inhibition a valid therapeutic strategy in cerebral ischemia? , 2001, Drug discovery today.
[29] J. Blenis,et al. Caspase-8 Is Required for Cell Death Induced by Expanded Polyglutamine Repeats , 1999, Neuron.
[30] S. Snyder,et al. Poly(ADP-ribose) polymerase gene disruption renders mice resistant to cerebral ischemia , 1997, Nature Medicine.
[31] S. Snyder,et al. Nitric oxide mediates glutamate neurotoxicity in primary cortical cultures. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[32] Changlian Zhu,et al. Different apoptotic mechanisms are activated in male and female brains after neonatal hypoxia–ischaemia , 2006, Journal of neurochemistry.
[33] L. McCullough,et al. Estrogen and ischemic neuroprotection: an integrated view , 2003, Trends in Endocrinology & Metabolism.
[34] P. Chan. Mitochondria and Neuronal Death/Survival Signaling Pathways in Cerebral Ischemia , 2004, Neurochemical Research.
[35] Kimberly A. Houser,et al. Early disruptions of the blood–brain barrier may contribute to exacerbated neuronal damage and prolonged functional recovery following stroke in aged rats , 2008, Neurobiology of Aging.
[36] J. Ménissier-de murcia,et al. Active Caspase-8 Translocates into the Nucleus of Apoptotic Cells to Inactivate Poly(ADP-ribose) Polymerase-2* , 2002, The Journal of Biological Chemistry.
[37] Yea-Ru Yang,et al. Effect of Age in Rats following Middle Cerebral Artery Occlusion , 2002, Gerontology.
[38] T. Joh,et al. A novel systemically active caspase inhibitor attenuates the toxicities of MPTP, malonate, and 3NP in vivo , 2004, Neurobiology of Disease.
[39] R. Traystman,et al. Gender-linked brain injury in experimental stroke. , 1998, Stroke.
[40] A. Anagnostopoulos,et al. Peripheral blood stem cell transplantation in the treatment of progressive multiple sclerosis: first results of a pilot study , 1997, Bone Marrow Transplantation.
[41] R. Sacco,et al. Stroke incidence among white, black, and Hispanic residents of an urban community: the Northern Manhattan Stroke Study. , 1998, American journal of epidemiology.
[42] G. Salvesen,et al. The apoptosome: signalling platform of cell death , 2007, Nature Reviews Molecular Cell Biology.