Lack ofp53 mutations and loss of heterozygosity in non-cultured human melanocytic lesions
暂无分享,去创建一个
[1] J. N. Newton Bishop,et al. The genetics of melanoma. , 2006, British journal of hospital medicine.
[2] G. Burg,et al. Analysis ofras mutations in human melanocytic lesions: activation of theras gene seems to be associated with the nodular type of human malignant melanoma , 2005, Journal of Cancer Research and Clinical Oncology.
[3] David Lane,et al. p53 tumour suppressor , 1998, Current Biology.
[4] Ian Jackson,et al. Variants of the melanocyte–stimulating hormone receptor gene are associated with red hair and fair skin in humans , 1995, Nature Genetics.
[5] D. Barker,et al. Comparison of the responses of human melanocytes with different melanin contents to ultraviolet B irradiation. , 1995, Cancer research.
[6] R. Eeles,et al. Genetic evidence in melanoma and bladder cancers that p16 and p53 function in separate pathways of tumor suppression. , 1995, The American journal of pathology.
[7] T. Leslie Youd,et al. Structural Aspects , 1995 .
[8] G. Burg,et al. Analysis of ras mutations in human melanocytic lesions: activation of the ras gene seems to be associated with the nodular type of human malignant melanoma. , 1995, Journal of cancer research and clinical oncology.
[9] S. Rauth,et al. Establishment of a human melanoma cell line lacking p53 expression and spontaneously metastasizing in nude mice. , 1994, Anticancer research.
[10] R Montesano,et al. Database of p53 gene somatic mutations in human tumors and cell lines. , 1994, Nucleic acids research.
[11] X. Montano,et al. Analysis of p53 in human cutaneous melanoma cell lines. , 1994, Oncogene.
[12] P. Kleihues,et al. Absence of p53 gene mutations in cutaneous melanoma. , 1994, The Journal of investigative dermatology.
[13] T. Soussi,et al. Can we predict solar ultraviolet radiation as the causal event in human tumours by analysing the mutation spectra of the p53 gene? , 1994, Mutation research.
[14] N. Hayward,et al. Mutation and expression of the p53 gene in human malignant melanoma , 1994, Melanoma research.
[15] J. Nesland,et al. TP53 allele loss, mutations and expression in malignant melanoma. , 1994, British Journal of Cancer.
[16] D. Bishop,et al. p53 protein detected by immunohistochemical staining is not always mutant. , 1993, Disease markers.
[17] A. Sober,et al. Lack of allelic deletion and point mutation as mechanisms of p53 activation in human malignant melanoma , 1993, International journal of cancer.
[18] R. Tyrrell,et al. Singlet oxygen: a primary effector in the ultraviolet A/near-visible light induction of the human heme oxygenase gene. , 1993, Cancer research.
[19] W. Cavenee,et al. Mutation and expression of the p53 gene in malignant melanoma cell lines , 1993, International journal of cancer.
[20] Elwood Jm. Recent developments in melanoma epidemiology, 1993. , 1993 .
[21] M. Barbareschi,et al. p53 Protein expression in nevi and melanomas. , 1993, Archives of dermatology.
[22] B. Epe,et al. DNA damage induced by photosensitizers in cellular and cell-free systems. , 1993, Mutation research.
[23] N. S. Mcnutt,et al. Oncogenes in Melanomas , 1992, The Journal of dermatology.
[24] M. Kulesz-Martin,et al. Formation of 8-hydroxydeoxyguanosine within DNA of mouse keratinocytes exposed in culture to UVB and H2O2. , 1992, Carcinogenesis.
[25] G. Wahl,et al. Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 alleles , 1992, Cell.
[26] S. Nishimura,et al. Photosensitized formation of 8-hydroxydeoxyguanosine in cellular DNA by riboflavin. , 1992, Biochemical and biophysical research communications.
[27] Bert Vogelstein,et al. p53 function and dysfunction , 1992, Cell.
[28] P. Friedman,et al. Wild-type p53 activates transcription in vitro , 1992, Nature.
[29] D. Lane,et al. p53, guardian of the genome , 1992, Nature.
[30] P. Meltzer,et al. Amplification of a gene encoding a p53-associated protein in human sarcomas , 1992, Nature.
[31] L. Donehower,et al. Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours , 1992, Nature.
[32] L. Loeb,et al. 8-Hydroxyguanine, an abundant form of oxidative DNA damage, causes G----T and A----C substitutions. , 1992, The Journal of biological chemistry.
[33] B. Vogelstein,et al. Participation of p53 protein in the cellular response to DNA damage. , 1991, Cancer research.
[34] J. Simon,et al. A role for sunlight in skin cancer: UV-induced p53 mutations in squamous cell carcinoma. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[35] C. Harris,et al. Chemical and physical carcinogenesis: advances and perspectives for the 1990s. , 1991, Cancer research.
[36] J. Bartek,et al. Aberrant expression of the p53 oncoprotein is a common feature of a wide spectrum of human malignancies. , 1991, Oncogene.
[37] A. Kimchi,et al. Wild-type p53 induces apoptosis of myeloid leukaemic cells that is inhibited by interleukin-6 , 1991, Nature.
[38] B. Vogelstein,et al. p53 mutations in human cancers. , 1991, Science.
[39] K. Kinzler,et al. Identification of p53 as a sequence-specific DNA-binding protein , 1991, Science.
[40] G. Gaidano,et al. p53 mutations in human lymphoid malignancies: association with Burkitt lymphoma and chronic lymphocytic leukemia. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[41] A. Levine,et al. The p53 tumour suppressor gene , 1991, Nature.
[42] D. George,et al. Tumorigenic potential associated with enhanced expression of a gene that is amplified in a mouse tumor cell line. , 1991, The EMBO journal.
[43] U. Schlegel,et al. Mutational analysis of the human p53 gene in malignant melanoma. , 1991, Pigment cell research.
[44] B. Epe. Genotoxicity of singlet oxygen. , 1991, Chemico-biological interactions.
[45] B. Vogelstein. A deadly inheritance , 1990, Nature.
[46] Arnold J. Levine,et al. The E6 oncoprotein encoded by human papillomavirus types 16 and 18 promotes the degradation of p53 , 1990, Cell.
[47] L. Strong,et al. Germ line p53 mutations in a familial syndrome of breast cancer, sarcomas, and other neoplasms. , 1990, Science.
[48] J. Yagüe,et al. AccII polymorphism of the p53 gene. , 1990, Nucleic acids research.
[49] T. Soussi,et al. Structural aspects of the p53 protein in relation to gene evolution. , 1990, Oncogene.
[50] F. Collins,et al. Mutations in the p53 gene occur in diverse human tumour types , 1989, Nature.
[51] Henry A. Erlich,et al. PCR Technology: Principles and Applications for DNA Amplification , 1989 .
[52] W. Blattner,et al. A cancer family syndrome in twenty-four kindreds. , 1988, Cancer research.
[53] A. Levine,et al. Activating mutations for transformation by p53 produce a gene product that forms an hsc70-p53 complex with an altered half-life , 1988, Molecular and cellular biology.
[54] T. Soussi,et al. Cloning and characterization of a cDNA from Xenopus laevis coding for a protein homologous to human and murine p53. , 1987, Oncogene.
[55] S. Bülow,et al. [The cancer family syndrome]. , 1981, Ugeskrift for laeger.
[56] D. Lane,et al. T antigen is bound to a host protein in SY40-transformed cells , 1979, Nature.
[57] A. Griffiths. Introduction to Genetic Analysis , 1976 .