Neutrophil extracellular trap formation and circulating nucleosomes in patients with chronic myeloproliferative neoplasms

[1]  C. Toh,et al.  The role of extracellular histones in haematological disorders , 2016, British journal of haematology.

[2]  M. Gutiérrez,et al.  Long‐term follow‐up of essential thrombocythemia patients treated with anagrelide: subgroup analysis according to JAK2/CALR/MPL mutational status , 2016, European journal of haematology.

[3]  R. Gómez,et al.  Mediators and molecular pathways involved in the regulation of neutrophil extracellular trap formation mediated by activated platelets , 2016, Journal of leukocyte biology.

[4]  D. Wagner,et al.  PHAGOCYTES , GRANULOCYTES , AND MYELOPOIESIS P-selectin promotes neutrophil extracellular trap formation in mice , 2015 .

[5]  C. Kahn,et al.  Diabetes primes neutrophils to undergo NETosis, which impairs wound healing , 2015, Nature Medicine.

[6]  T. Gould,et al.  Extracellular DNA and histones: double‐edged swords in immunothrombosis , 2015, Journal of thrombosis and haemostasis : JTH.

[7]  Ayalew Tefferi,et al.  Myeloproliferative Neoplasms A Contemporary Review , 2015 .

[8]  N. Takeyama,et al.  Neutrophil extracellular traps in patients with sepsis. , 2015, The Journal of surgical research.

[9]  O. Tasaki,et al.  Presence of Neutrophil Extracellular Traps and Citrullinated Histone H3 in the Bloodstream of Critically Ill Patients , 2014, PloS one.

[10]  S. Akira,et al.  JAK2V617F+ myeloproliferative neoplasm clones evoke paracrine DNA damage to adjacent normal cells through secretion of lipocalin-2. , 2014, Blood.

[11]  D. Tang,et al.  Release and activity of histone in diseases , 2014, Cell Death and Disease.

[12]  A. Buser,et al.  Enhanced neutrophil extracellular trap generation in rheumatoid arthritis: analysis of underlying signal transduction pathways and potential diagnostic utility , 2014, Arthritis Research & Therapy.

[13]  P. Libby,et al.  Neutrophil extracellular traps form predominantly during the organizing stage of human venous thromboembolism development , 2014, Journal of thrombosis and haemostasis : JTH.

[14]  T. Golub,et al.  Integrated genomic analysis illustrates the central role of JAK-STAT pathway activation in myeloproliferative neoplasm pathogenesis. , 2014, Blood.

[15]  D. Wagner,et al.  Thrombosis: tangled up in NETs. , 2014, Blood.

[16]  D. Wagner,et al.  VWF-mediated leukocyte recruitment with chromatin decondensation by PAD4 increases myocardial ischemia/reperfusion injury in mice. , 2014, Blood.

[17]  T. Fuchs,et al.  Neutrophils release extracellular DNA traps during storage of red blood cell units , 2013, Transfusion.

[18]  M. Gougerot-Pocidalo,et al.  Increased reactive oxygen species production and p47phox phosphorylation in neutrophils from myeloproliferative disorders patients with JAK2 (V617F) mutation , 2013, Haematologica.

[19]  T. Barbui,et al.  Myeloproliferative neoplasms and thrombosis. , 2013, Blood.

[20]  H. Crijns,et al.  Elevated Levels of Circulating DNA and Chromatin Are Independently Associated With Severe Coronary Atherosclerosis and a Prothrombotic State , 2013, Arteriosclerosis, thrombosis, and vascular biology.

[21]  S. Negrotto,et al.  Regulation of Neutrophil Extracellular Trap Formation by Anti-Inflammatory Drugs , 2013, The Journal of Pharmacology and Experimental Therapeutics.

[22]  E. Solary,et al.  A role for reactive oxygen species in JAK2V617F myeloproliferative neoplasm progression , 2013, Leukemia.

[23]  S. Middeldorp,et al.  Circulating Nucleosomes and Neutrophil Activation as Risk Factors for Deep Vein Thrombosis , 2012, Arteriosclerosis, thrombosis, and vascular biology.

[24]  R. D. de Winter,et al.  Neutrophils, neutrophil extracellular traps and interleukin-17 associate with the organisation of thrombi in acute myocardial infarction , 2012, Thrombosis and Haemostasis.

[25]  B. Engelmann,et al.  Thrombosis as an intravascular effector of innate immunity , 2012, Nature Reviews Immunology.

[26]  A. Falanga,et al.  Thrombotic disease in the myeloproliferative neoplasms. , 2012, Hematology. American Society of Hematology. Education Program.

[27]  A. Tefferi,et al.  Plasma cytokines in polycythemia vera: Phenotypic correlates, prognostic relevance, and comparison with myelofibrosis , 2012, American journal of hematology.

[28]  M. Dikshit,et al.  Cytokines Induced Neutrophil Extracellular Traps Formation: Implication for the Inflammatory Disease Condition , 2012, PloS one.

[29]  D. Scadden,et al.  Cancers predispose neutrophils to release extracellular DNA traps that contribute to cancer-associated thrombosis , 2012, Proceedings of the National Academy of Sciences.

[30]  T. Barbui,et al.  Disease characteristics and clinical outcome in young adults with essential thrombocythemia versus early/prefibrotic primary myelofibrosis. , 2012, Blood.

[31]  Z. Werb,et al.  Platelets induce neutrophil extracellular traps in transfusion-related acute lung injury. , 2012, The Journal of clinical investigation.

[32]  H. Anders,et al.  Histones from dying renal cells aggravate kidney injury via TLR2 and TLR4. , 2012, Journal of the American Society of Nephrology : JASN.

[33]  D. Wagner,et al.  Neutrophil extracellular traps promote deep vein thrombosis in mice , 2012, Journal of thrombosis and haemostasis : JTH.

[34]  S. Negrotto,et al.  Acidosis downregulates platelet haemostatic functions and promotes neutrophil proinflammatory responses mediated by platelets , 2011, Thrombosis and Haemostasis.

[35]  J. Hartwig,et al.  Extracellular DNA traps are associated with the pathogenesis of TRALI in humans and mice. , 2011, Blood.

[36]  A. Tefferi,et al.  Circulating interleukin (IL)-8, IL-2R, IL-12, and IL-15 levels are independently prognostic in primary myelofibrosis: a comprehensive cytokine profiling study. , 2011, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.

[37]  H. Waldmann,et al.  Activation of the Raf-MEK-ERK pathway is required for neutrophil extracellular trap formation. , 2011, Nature chemical biology.

[38]  F. Passamonti,et al.  DIPSS plus: a refined Dynamic International Prognostic Scoring System for primary myelofibrosis that incorporates prognostic information from karyotype, platelet count, and transfusion status. , 2011, Journal of clinical oncology : official journal of the American Society of Clinical Oncology.

[39]  P. Vandenabeele,et al.  Neutrophil extracellular trap cell death requires both autophagy and superoxide generation , 2011, Cell Research.

[40]  Ayalew Tefferi,et al.  Safety and efficacy of INCB018424, a JAK1 and JAK2 inhibitor, in myelofibrosis. , 2010, The New England journal of medicine.

[41]  J. Hartwig,et al.  Extracellular DNA traps promote thrombosis , 2010, Proceedings of the National Academy of Sciences.

[42]  P. Lev,et al.  Monocyte IL-2Ralpha expression is associated with thrombosis and the JAK2V617F mutation in myeloproliferative neoplasms. , 2010, Cytokine.

[43]  P. Lev,et al.  Screening for MPL mutations in essential thrombocythemia and primary myelofibrosis: normal Mpl expression and absence of constitutive STAT3 and STAT5 activation in MPLW515L‐positive platelets , 2010, European journal of haematology.

[44]  C. Bloomfield,et al.  The 2008 revision of the World Health Organization (WHO) classification of myeloid neoplasms and acute leukemia: rationale and important changes. , 2009, Blood.

[45]  Z. Werb,et al.  Netting neutrophils in autoimmune small-vessel vasculitis , 2009, Nature Medicine.

[46]  T. Barbui,et al.  Leukocytosis is a risk factor for thrombosis in essential thrombocythemia: interaction with treatment, standard risk factors, and Jak2 mutation status. , 2007, Blood.

[47]  V. Wahn,et al.  Novel cell death program leads to neutrophil extracellular traps , 2007, The Journal of Cell Biology.

[48]  C. Cerletti,et al.  Inhibition of tissue factor expression by hydroxyurea in polymorphonuclear leukocytes from patients with myeloproliferative disorders: a new effect for an old drug? , 2006, Journal of thrombosis and haemostasis : JTH.

[49]  P. Lev,et al.  JAK2V617F mutation in platelets from essential thrombocythemia patients: correlation with clinical features and analysis of STAT5 phosphorylation status , 2006, European journal of haematology.

[50]  N. Villamor,et al.  Increased platelet and leukocyte activation as contributing mechanisms for thrombosis in essential thrombocythemia and correlation with the JAK2 mutational status. , 2006, Haematologica.

[51]  A. Zychlinsky,et al.  Neutrophil Extracellular Traps Kill Bacteria , 2004, Science.

[52]  M. Untch,et al.  Nucleosomes in serum of patients with benign and malignant diseases , 2001, International journal of cancer.

[53]  T. Barbui,et al.  Polymorphonuclear leukocyte activation and hemostasis in patients with essential thrombocythemia and polycythemia vera , 2000 .

[54]  M. Lishner,et al.  Leukocyte function in chronic myeloproliferative disorders. , 1998, Blood cells, molecules & diseases.