CD4+ and CD8+ cytotoxic T lymphocytes may induce mesenchymal cell apoptosis in IgG4-related disease.
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N. Yosef | J. Stone | Xiuwei Zhang | S. Pillai | J. Kers | M. Bolster | M. Moriyama | Seiji Nakamura | Z. Wallace | S. Montesi | N. Kaneko | C. Perugino | E. Della-Torre | V. Mahajan | H. Mattoo | M. Ghebremichael | G. Mylvaganam | L. Liang | T. Maehara | H. Allard-Chamard | Mizuki Sakamoto | Samuel J H Murphy | Yesim Tuncay | Hang Liu | Liam Harvey | Akira Tinju | Keita Mochizuki | Ryusuke Munemura | Samuel J. H. Murphy
[1] Simona Baghai Sain,et al. B lymphocytes directly contribute to tissue fibrosis in IgG4-Related Disease. , 2020, The Journal of allergy and clinical immunology.
[2] J. Stone,et al. Cytotoxic CD4+ T lymphocytes may induce endothelial cell apoptosis in systemic sclerosis. , 2020, The Journal of clinical investigation.
[3] Amita Sharma,et al. The 2019 American College of Rheumatology/European League Against Rheumatism classification criteria for IgG4-related disease , 2019, Annals of the rheumatic diseases.
[4] Amita Sharma,et al. The 2019 American College of Rheumatology/European League Against Rheumatism classification criteria for IgG4-related disease , 2019, Arthritis & rheumatology.
[5] M. Kubo,et al. Synergistic effect of IgG4 antibody and CTLs causes tissue inflammation in IgG4-related disease. , 2019, International immunology.
[6] J. Stone,et al. Identification of galectin‐3 as an autoantigen in patients with IgG4‐related disease , 2019, The Journal of allergy and clinical immunology.
[7] J. Stone,et al. An International Multispecialty Validation Study of the IgG4‐Related Disease Responder Index , 2018, Arthritis care & research.
[8] Kamil Slowikowski,et al. Mixed-effects association of single cells identifies an expanded effector CD4+ T cell subset in rheumatoid arthritis , 2018, Science Translational Medicine.
[9] H. Ueno,et al. A CD4+ T cell population expanded in Lupus blood provides B cell help through IL10 and succinate , 2018, Nature Medicine.
[10] Toshimasa Shimizu,et al. Modulation of Apoptosis by Cytotoxic Mediators and Cell-Survival Molecules in Sjögren’s Syndrome , 2018, International journal of molecular sciences.
[11] J. Stone,et al. A CD8α− Subset of CD4+SLAMF7+ Cytotoxic T Cells Is Expanded in Patients With IgG4‐Related Disease and Decreases Following Glucocorticoid Treatment , 2018, Arthritis & rheumatology.
[12] Christoph Ziegenhain,et al. zUMIs - A fast and flexible pipeline to process RNA sequencing data with UMIs , 2017, bioRxiv.
[13] Bjoern Peters,et al. Precursors of human CD4+ cytotoxic T lymphocytes identified by single-cell transcriptome analysis , 2018, Science Immunology.
[14] Julie G. Burel,et al. Unique phenotypes and clonal expansions of human CD4 effector memory T cells re-expressing CD45RA , 2017, Nature Communications.
[15] Michael R. Elliott,et al. Efferocytosis Signaling in the Regulation of Macrophage Inflammatory Responses , 2017, The Journal of Immunology.
[16] Jefte M. Drijvers,et al. Clonal expansion of CD4(+) cytotoxic T lymphocytes in patients with IgG4-related disease. , 2016, The Journal of allergy and clinical immunology.
[17] Jefte M. Drijvers,et al. Lesional CD4+ IFN-γ+ cytotoxic T lymphocytes in IgG4-related dacryoadenitis and sialoadenitis , 2016, Annals of the rheumatic diseases.
[18] Ravi V. Kolla,et al. Dengue virus infection elicits highly polarized CX3CR1+ cytotoxic CD4+ T cells associated with protective immunity , 2015, Proceedings of the National Academy of Sciences.
[19] J. Stone,et al. IgG4-related disease , 2015, The Lancet.
[20] Ash A. Alizadeh,et al. Robust enumeration of cell subsets from tissue expression profiles , 2015, Nature Methods.
[21] J. Stone,et al. Rituximab for IgG4-related disease: a prospective, open-label trial , 2015, Annals of the rheumatic diseases.
[22] K. Imai,et al. The role of cytotoxic T cells in IgG4-related dacryoadenitis and sialadenitis, the so-called Mikulicz's disease , 2014, Modern rheumatology.
[23] J. Stone,et al. De novo oligoclonal expansions of circulating plasmablasts in active and relapsing IgG4-related disease. , 2014, The Journal of allergy and clinical immunology.
[24] A. Regev,et al. Preparation of Single‐Cell RNA‐Seq Libraries for Next Generation Sequencing , 2014, Current protocols in molecular biology.
[25] Christopher Chang,et al. Diagnostic criteria for sarcoidosis. , 2014, Autoimmunity reviews.
[26] Olga V. Britanova,et al. Preparing Unbiased T-Cell Receptor and Antibody cDNA Libraries for the Deep Next Generation Sequencing Profiling , 2013, Front. Immunol..
[27] D. Sahani,et al. Consensus statement on the pathology of IgG4-related disease , 2012, Modern Pathology.
[28] J. Stone,et al. IgG4-related disease. , 2012, The New England journal of medicine.
[29] M. Strioga,et al. CD8+ CD28− and CD8+ CD57+ T cells and their role in health and disease , 2011, Immunology.
[30] S. Kaneko,et al. Fractalkine and its receptor, CX3CR1, promote hypertensive interstitial fibrosis in the kidney , 2011, Hypertension Research.
[31] Michael N. Alonso,et al. B cells promote insulin resistance through modulation of T cells and production of pathogenic IgG antibodies , 2011, Nature Medicine.
[32] N. Kaminski,et al. CD28 Down-Regulation on Circulating CD4 T-Cells Is Associated with Poor Prognoses of Patients with Idiopathic Pulmonary Fibrosis , 2010, PloS one.
[33] K. Malmberg,et al. T Cell Infiltrates in the Muscles of Patients with Dermatomyositis and Polymyositis Are Dominated by CD28null T Cells1 , 2009, The Journal of Immunology.
[34] M. Betts,et al. The cytolytic enzymes granyzme A, granzyme B, and perforin: expression patterns, cell distribution, and their relationship to cell maturity and bright CD57 expression , 2008, Journal of leukocyte biology.
[35] Xue-guang Zhang,et al. Association of Graves’ Disease and Prevalence of Circulating IFN-γ-producing CD28− T Cells , 2008, Journal of Clinical Immunology.
[36] N. Hellings,et al. CD4+CD28null T Cells in Autoimmune Disease: Pathogenic Features and Decreased Susceptibility to Immunoregulation1 , 2007, The Journal of Immunology.
[37] T. Boon,et al. CD45RA on human CD8 T cells is sensitive to the time elapsed since the last antigenic stimulation. , 2006, Blood.
[38] P. Murphy,et al. Chemokine receptor CX3CR1 regulates renal interstitial fibrosis after ischemia-reperfusion injury. , 2006, The American journal of pathology.
[39] E. Remmerswaal,et al. Emergence of a CD4+CD28− Granzyme B+, Cytomegalovirus-Specific T Cell Subset after Recovery of Primary Cytomegalovirus Infection , 2004, The Journal of Immunology.
[40] R. Ecker,et al. Microscopy‐based multicolor tissue cytometry at the single‐cell level , 2004, Cytometry. Part A : the journal of the International Society for Analytical Cytology.
[41] K. Elkon,et al. Fibroblast and endothelial apoptosis in systemic sclerosis , 2003, Current opinion in rheumatology.
[42] D. Green,et al. Activation‐induced cell death in T cells , 2003, Immunological reviews.
[43] B. Uhal. Apoptosis in lung fibrosis and repair. , 2002, Chest.
[44] R. Jonsson,et al. Classification criteria for Sjögren's syndrome: a revised version of the European criteria proposed by the American-European Consensus Group , 2002, Annals of the rheumatic diseases.
[45] Anthony D. Kelleher,et al. Characterization of CD4+ CTLs Ex Vivo1 , 2002, The Journal of Immunology.
[46] J. Brayer,et al. Elevated proapoptotic Bax and caspase 3 activation in the NOD.scid model of Sjögren's syndrome. , 2001, Arthritis and rheumatism.
[47] F. Sallusto,et al. Two subsets of memory T lymphocytes with distinct homing potentials and effector functions , 1999, Nature.
[48] M. Gershwin,et al. Endothelial cell apoptosis is a primary pathogenetic event underlying skin lesions in avian and human scleroderma. , 1996, The Journal of clinical investigation.
[49] C. Weyand,et al. CD4+ CD7- CD28- T cells are expanded in rheumatoid arthritis and are characterized by autoreactivity. , 1996, The Journal of clinical investigation.
[50] J. Stone,et al. IgG4-related disease. , 2014, Annual review of pathology.