CD4+ and CD8+ cytotoxic T lymphocytes may induce mesenchymal cell apoptosis in IgG4-related disease.

[1]  Simona Baghai Sain,et al.  B lymphocytes directly contribute to tissue fibrosis in IgG4-Related Disease. , 2020, The Journal of allergy and clinical immunology.

[2]  J. Stone,et al.  Cytotoxic CD4+ T lymphocytes may induce endothelial cell apoptosis in systemic sclerosis. , 2020, The Journal of clinical investigation.

[3]  Amita Sharma,et al.  The 2019 American College of Rheumatology/European League Against Rheumatism classification criteria for IgG4-related disease , 2019, Annals of the rheumatic diseases.

[4]  Amita Sharma,et al.  The 2019 American College of Rheumatology/European League Against Rheumatism classification criteria for IgG4-related disease , 2019, Arthritis & rheumatology.

[5]  M. Kubo,et al.  Synergistic effect of IgG4 antibody and CTLs causes tissue inflammation in IgG4-related disease. , 2019, International immunology.

[6]  J. Stone,et al.  Identification of galectin‐3 as an autoantigen in patients with IgG4‐related disease , 2019, The Journal of allergy and clinical immunology.

[7]  J. Stone,et al.  An International Multispecialty Validation Study of the IgG4‐Related Disease Responder Index , 2018, Arthritis care & research.

[8]  Kamil Slowikowski,et al.  Mixed-effects association of single cells identifies an expanded effector CD4+ T cell subset in rheumatoid arthritis , 2018, Science Translational Medicine.

[9]  H. Ueno,et al.  A CD4+ T cell population expanded in Lupus blood provides B cell help through IL10 and succinate , 2018, Nature Medicine.

[10]  Toshimasa Shimizu,et al.  Modulation of Apoptosis by Cytotoxic Mediators and Cell-Survival Molecules in Sjögren’s Syndrome , 2018, International journal of molecular sciences.

[11]  J. Stone,et al.  A CD8α− Subset of CD4+SLAMF7+ Cytotoxic T Cells Is Expanded in Patients With IgG4‐Related Disease and Decreases Following Glucocorticoid Treatment , 2018, Arthritis & rheumatology.

[12]  Christoph Ziegenhain,et al.  zUMIs - A fast and flexible pipeline to process RNA sequencing data with UMIs , 2017, bioRxiv.

[13]  Bjoern Peters,et al.  Precursors of human CD4+ cytotoxic T lymphocytes identified by single-cell transcriptome analysis , 2018, Science Immunology.

[14]  Julie G. Burel,et al.  Unique phenotypes and clonal expansions of human CD4 effector memory T cells re-expressing CD45RA , 2017, Nature Communications.

[15]  Michael R. Elliott,et al.  Efferocytosis Signaling in the Regulation of Macrophage Inflammatory Responses , 2017, The Journal of Immunology.

[16]  Jefte M. Drijvers,et al.  Clonal expansion of CD4(+) cytotoxic T lymphocytes in patients with IgG4-related disease. , 2016, The Journal of allergy and clinical immunology.

[17]  Jefte M. Drijvers,et al.  Lesional CD4+ IFN-γ+ cytotoxic T lymphocytes in IgG4-related dacryoadenitis and sialoadenitis , 2016, Annals of the rheumatic diseases.

[18]  Ravi V. Kolla,et al.  Dengue virus infection elicits highly polarized CX3CR1+ cytotoxic CD4+ T cells associated with protective immunity , 2015, Proceedings of the National Academy of Sciences.

[19]  J. Stone,et al.  IgG4-related disease , 2015, The Lancet.

[20]  Ash A. Alizadeh,et al.  Robust enumeration of cell subsets from tissue expression profiles , 2015, Nature Methods.

[21]  J. Stone,et al.  Rituximab for IgG4-related disease: a prospective, open-label trial , 2015, Annals of the rheumatic diseases.

[22]  K. Imai,et al.  The role of cytotoxic T cells in IgG4-related dacryoadenitis and sialadenitis, the so-called Mikulicz's disease , 2014, Modern rheumatology.

[23]  J. Stone,et al.  De novo oligoclonal expansions of circulating plasmablasts in active and relapsing IgG4-related disease. , 2014, The Journal of allergy and clinical immunology.

[24]  A. Regev,et al.  Preparation of Single‐Cell RNA‐Seq Libraries for Next Generation Sequencing , 2014, Current protocols in molecular biology.

[25]  Christopher Chang,et al.  Diagnostic criteria for sarcoidosis. , 2014, Autoimmunity reviews.

[26]  Olga V. Britanova,et al.  Preparing Unbiased T-Cell Receptor and Antibody cDNA Libraries for the Deep Next Generation Sequencing Profiling , 2013, Front. Immunol..

[27]  D. Sahani,et al.  Consensus statement on the pathology of IgG4-related disease , 2012, Modern Pathology.

[28]  J. Stone,et al.  IgG4-related disease. , 2012, The New England journal of medicine.

[29]  M. Strioga,et al.  CD8+ CD28− and CD8+ CD57+ T cells and their role in health and disease , 2011, Immunology.

[30]  S. Kaneko,et al.  Fractalkine and its receptor, CX3CR1, promote hypertensive interstitial fibrosis in the kidney , 2011, Hypertension Research.

[31]  Michael N. Alonso,et al.  B cells promote insulin resistance through modulation of T cells and production of pathogenic IgG antibodies , 2011, Nature Medicine.

[32]  N. Kaminski,et al.  CD28 Down-Regulation on Circulating CD4 T-Cells Is Associated with Poor Prognoses of Patients with Idiopathic Pulmonary Fibrosis , 2010, PloS one.

[33]  K. Malmberg,et al.  T Cell Infiltrates in the Muscles of Patients with Dermatomyositis and Polymyositis Are Dominated by CD28null T Cells1 , 2009, The Journal of Immunology.

[34]  M. Betts,et al.  The cytolytic enzymes granyzme A, granzyme B, and perforin: expression patterns, cell distribution, and their relationship to cell maturity and bright CD57 expression , 2008, Journal of leukocyte biology.

[35]  Xue-guang Zhang,et al.  Association of Graves’ Disease and Prevalence of Circulating IFN-γ-producing CD28− T Cells , 2008, Journal of Clinical Immunology.

[36]  N. Hellings,et al.  CD4+CD28null T Cells in Autoimmune Disease: Pathogenic Features and Decreased Susceptibility to Immunoregulation1 , 2007, The Journal of Immunology.

[37]  T. Boon,et al.  CD45RA on human CD8 T cells is sensitive to the time elapsed since the last antigenic stimulation. , 2006, Blood.

[38]  P. Murphy,et al.  Chemokine receptor CX3CR1 regulates renal interstitial fibrosis after ischemia-reperfusion injury. , 2006, The American journal of pathology.

[39]  E. Remmerswaal,et al.  Emergence of a CD4+CD28− Granzyme B+, Cytomegalovirus-Specific T Cell Subset after Recovery of Primary Cytomegalovirus Infection , 2004, The Journal of Immunology.

[40]  R. Ecker,et al.  Microscopy‐based multicolor tissue cytometry at the single‐cell level , 2004, Cytometry. Part A : the journal of the International Society for Analytical Cytology.

[41]  K. Elkon,et al.  Fibroblast and endothelial apoptosis in systemic sclerosis , 2003, Current opinion in rheumatology.

[42]  D. Green,et al.  Activation‐induced cell death in T cells , 2003, Immunological reviews.

[43]  B. Uhal Apoptosis in lung fibrosis and repair. , 2002, Chest.

[44]  R. Jonsson,et al.  Classification criteria for Sjögren's syndrome: a revised version of the European criteria proposed by the American-European Consensus Group , 2002, Annals of the rheumatic diseases.

[45]  Anthony D. Kelleher,et al.  Characterization of CD4+ CTLs Ex Vivo1 , 2002, The Journal of Immunology.

[46]  J. Brayer,et al.  Elevated proapoptotic Bax and caspase 3 activation in the NOD.scid model of Sjögren's syndrome. , 2001, Arthritis and rheumatism.

[47]  F. Sallusto,et al.  Two subsets of memory T lymphocytes with distinct homing potentials and effector functions , 1999, Nature.

[48]  M. Gershwin,et al.  Endothelial cell apoptosis is a primary pathogenetic event underlying skin lesions in avian and human scleroderma. , 1996, The Journal of clinical investigation.

[49]  C. Weyand,et al.  CD4+ CD7- CD28- T cells are expanded in rheumatoid arthritis and are characterized by autoreactivity. , 1996, The Journal of clinical investigation.

[50]  J. Stone,et al.  IgG4-related disease. , 2014, Annual review of pathology.