Commitment to the B-lymphoid lineage depends on the transcription factor Pax5
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Stephen L. Nutt | M. Busslinger | S. Nutt | A. Rolink | Meinrad Busslinger | B. Heavey | Antonius G. Rolink | Barry Heavey
[1] T. Enver,et al. Do stem cells play dice? , 1998, Blood.
[2] K. Ohmura,et al. Direct evidence for the commitment of hematopoietic stem cells to T, B and myeloid lineages in murine fetal liver. , 1997, International immunology.
[3] D. Raulet. Development and tolerance of natural killer cells. , 1999, Current opinion in immunology.
[4] E. Wagner,et al. Bone and haematopoietic defects in mice lacking c-fos , 1992, Nature.
[5] H. Karasuyama,et al. Establishment of mouse cell lines which constitutively secrete large quantities of interleukin 2, 3, 4 or 5, using modified cDNA expression vectors , 1988, European journal of immunology.
[6] A. Rolink,et al. The SCID but not the RAG-2 gene product is required for S mu-S epsilon heavy chain class switching. , 1996, Immunity.
[7] R. Grosschedl,et al. Failure of B-cell differentiation in mice lacking the transcription factor EBF , 1995, Nature.
[8] T. Waldmann,et al. Requirement for IRF-1 in the microenvironment supporting development of natural killer cells , 1998, Nature.
[9] M. Sigvardsson,et al. EBF and E47 collaborate to induce expression of the endogenous immunoglobulin surrogate light chain genes. , 1997, Immunity.
[10] T. Era,et al. Development of osteoclasts from embryonic stem cells through a pathway that is c-fms but not c-kit dependent. , 1997, Blood.
[11] A. Elefanty,et al. Progenitor tumours from Emu‐bcl‐2‐myc transgenic mice have lymphomyeloid differentiation potential and reveal developmental differences in cell survival. , 1996, The EMBO journal.
[12] A. Rolink,et al. Long‐term proliferating early pre B cell lines and clones with the potential to develop to surface Ig‐positive, mitogen reactive B cells in vitro and in vivo. , 1991, The EMBO journal.
[13] R. Steinman,et al. Generation of large numbers of dendritic cells from mouse bone marrow cultures supplemented with granulocyte/macrophage colony-stimulating factor , 1992, The Journal of experimental medicine.
[14] A. Cumano,et al. Bipotential precursors of B cells and macrophages in murine fetal liver , 1992, Nature.
[15] M. Greaves,et al. Loops, Lineage, and Leukemia , 1998, Cell.
[16] D. Metcalf. Lineage commitment and maturation in hematopoietic cells: the case for extrinsic regulation. , 1998, Blood.
[17] M. Greaves,et al. Multilineage gene expression precedes commitment in the hemopoietic system. , 1997, Genes & development.
[18] E. Wagner,et al. Complete block of early B cell differentiation and altered patterning of the posterior midbrain in mice lacking Pax5 BSAP , 1994, Cell.
[19] J D Kemp,et al. Resolution and characterization of pro-B and pre-pro-B cell stages in normal mouse bone marrow , 1991, The Journal of experimental medicine.
[20] S. Morony,et al. OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis , 1999, Nature.
[21] R. Hardy,et al. Identification of the earliest B lineage stage in mouse bone marrow. , 1996, Immunity.
[22] J. Cleveland,et al. Transformation of murine bone marrow cells with combined v-raf-v-myc oncogenes yields clonally related mature B cells and macrophages , 1990, Molecular and cellular biology.
[23] M. Busslinger,et al. Independent regulation of the two Pax5 alleles during B-cell development , 1999, Nature Genetics.
[24] R. Steinman,et al. Dendritic cells and the control of immunity , 1998, Nature.
[25] C. Sherr,et al. Macrophage lineage switching of murine early pre-B lymphoid cells expressing transduced fms genes , 1990, Molecular and cellular biology.
[26] M. Sigvardsson,et al. The B29 (Immunoglobulin β-Chain) Gene Is a Genetic Target for Early B-Cell Factor , 1999, Molecular and Cellular Biology.
[27] D. Fearon,et al. A subpopulation of B220+ cells in murine bone marrow does not express CD19 and contains natural killer cell progenitors , 1996, The Journal of experimental medicine.
[28] K. Akashi,et al. Simultaneous occurrence of myelomonocytic leukemia and multiple myeloma: Involvement of common leukemic progenitors and their developmental abnormality of “lineage infidelity” , 1991, Journal of cellular physiology.
[29] M. Busslinger,et al. Long-term in vivo reconstitution of T-cell development by Pax5-deficient B-cell progenitors , 1999, Nature.
[30] S. Burdach,et al. Lymphopenia in interleukin (IL)-7 gene-deleted mice identifies IL-7 as a nonredundant cytokine , 1995, The Journal of experimental medicine.
[31] J. Monroe,et al. Molecular Biology of B-Cell and T-Cell Development , 1998, Contemporary Immunology.
[32] M. Ogawa,et al. Development of natural killer cells, B lymphocytes, macrophages, and mast cells from single hematopoietic progenitors in culture of murine fetal liver cells. , 1997, Blood.
[33] A. Galy,et al. Human T, B, natural killer, and dendritic cells arise from a common bone marrow progenitor cell subset. , 1995, Immunity.
[34] I. Weissman,et al. Identification of Clonogenic Common Lymphoid Progenitors in Mouse Bone Marrow , 1997, Cell.
[35] C. Murre,et al. Induction of Early B Cell Factor (EBF) and Multiple B Lineage Genes by the Basic Helix-Loop-Helix Transcription Factor E12 , 1998, The Journal of experimental medicine.
[36] G. Lacaud,et al. Identification of a fetal hematopoietic precursor with B cell, T cell, and macrophage potential. , 1998, Immunity.
[37] T. Winkler,et al. IL-2 receptor alpha chain (CD25, TAC) expression defines a crucial stage in pre-B cell development. , 1994, International immunology.
[38] V. Stewart,et al. RAG-2-deficient mice lack mature lymphocytes owing to inability to initiate V(D)J rearrangement , 1992, Cell.
[39] S. Rudikoff,et al. Relationships between B cell and myeloid differentiation. Studies with a B lymphocyte progenitor line, HAFTL-1 , 1988, The Journal of experimental medicine.
[40] H. Weintraub,et al. The helix-loop-helix gene E2A is required for B cell formation , 1994, Cell.
[41] Ian Krop,et al. E2A proteins are required for proper B cell development and initiation of immunoglobulin gene rearrangements , 1994, Cell.
[42] M. Busslinger,et al. Essential functions of Pax5 (BSAP) in pro-B cell development: difference between fetal and adult B lymphopoiesis and reduced V-to-DJ recombination at the IgH locus. , 1997, Genes & development.
[43] R. Jaenisch,et al. MHC class I deficiency: susceptibility to natural killer (NK) cells and impaired NK activity. , 1991, Science.
[44] A. M. Morrison,et al. Identification of BSAP (Pax‐5) target genes in early B‐cell development by loss‐ and gain‐of‐function experiments , 1998, The EMBO journal.
[45] W. Alexander,et al. Hemopoietic lineage switch: v-raf oncogene converts Eμmyc transgenic B cells into macrophages , 1988, Cell.
[46] M. Busslinger,et al. Early Function of Pax5 (BSAP) before the Pre-B Cell Receptor Stage of B Lymphopoiesis , 1998, The Journal of experimental medicine.
[47] A. Aguzzi,et al. Pax-5 encodes the transcription factor BSAP and is expressed in B lymphocytes, the developing CNS, and adult testis. , 1992, Genes & development.
[48] D. Link,et al. Interleukin-6 and the granulocyte colony-stimulating factor receptor are major independent regulators of granulopoiesis in vivo but are not required for lineage commitment or terminal differentiation. , 1997, Blood.
[49] M. A. Borrello,et al. The B/macrophage cell: an elusive link between CD5+ B lymphocytes and macrophages. , 1996, Immunology today.
[50] E. Wagner,et al. c-Fos: a key regulator of osteoclast-macrophage lineage determination and bone remodeling. , 1994, Science.