E2A-PBX1 Interacts Directly with the KIX Domain of CBP/p300 in the Induction of Proliferation in Primary Hematopoietic Cells*
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Richard A. Currie | David P. LeBrun | D. LeBrun | G. Blobel | Brandy D. Hyndman | R. Currie | Gerd A. Blobel | R. Casselman | Richard Bayly | Luan Chuen | Richard Casselman | Richard Bayly | L. Chuen
[1] Kevin S. Smith,et al. Bmi-1 regulation of INK4A-ARF is a downstream requirement for transformation of hematopoietic progenitors by E2a-Pbx1. , 2003, Molecular cell.
[2] D. LeBrun,et al. A BASIC HELIX-LOOP-HELIX TRANSCRIPTION FACTORS IN HUMAN LEUKEMIA , 2002 .
[3] T. Yao,et al. Regulation of E2A Activities by Histone Acetyltransferases in B Lymphocyte Development* , 2003, The Journal of Biological Chemistry.
[4] M. Groudine,et al. Controlling the double helix , 2003, Nature.
[5] K. Lumb,et al. Structurally distinct modes of recognition of the KIX domain of CBP by Jun and CREB. , 2002, Biochemistry.
[6] Peter E Wright,et al. Cooperativity in Transcription Factor Binding to the Coactivator CREB-binding Protein (CBP) , 2002, The Journal of Biological Chemistry.
[7] J. Rehg,et al. A transcription-factor-binding surface of coactivator p300 is required for haematopoiesis , 2002, Nature.
[8] R. St.Onge,et al. DNA Damage-dependent and -independent Phosphorylation of the hRad9 Checkpoint Protein* , 2001, The Journal of Biological Chemistry.
[9] H. Kondoh,et al. The leukemogenic transcription factor E2a-Pbx1 induces expression of the putative N-myc and p53 target gene NDRG1 in Ba/F3 cells , 2001, Leukemia.
[10] R. Tjian,et al. Transcriptional coactivator complexes. , 2001, Annual review of biochemistry.
[11] L. Cantley,et al. Minimal activators that bind to the KIX domain of p300/CBP identified by phage display screening , 2000, Nature Biotechnology.
[12] D. LeBrun,et al. Role for Homodimerization in Growth Deregulation by E2a Fusion Proteins , 2000, Molecular and Cellular Biology.
[13] R. Goodman,et al. CBP/p300 in cell growth, transformation, and development. , 2000, Genes & development.
[14] L. Gazzolo,et al. The Human T Cell Leukemia/Lymphotropic Virus Type 1 Tax Protein Represses MyoD-dependent Transcription by Inhibiting MyoD-binding to the KIX Domain of p300 , 2000, The Journal of Biological Chemistry.
[15] C. Murre,et al. Helix-Loop-Helix Proteins: Regulators of Transcription in Eucaryotic Organisms , 2000, Molecular and Cellular Biology.
[16] H. Giebler,et al. Binding of p53 to the KIX Domain of CREB Binding Protein , 1999, The Journal of Biological Chemistry.
[17] P. Wright,et al. Role of Secondary Structure in Discrimination between Constitutive and Inducible Activators , 1999, Molecular and Cellular Biology.
[18] S. Berger,et al. A conserved motif present in a class of helix-loop-helix proteins activates transcription by direct recruitment of the SAGA complex. , 1999, Molecular cell.
[19] C. Glass,et al. Determinants of coactivator LXXLL motif specificity in nuclear receptor transcriptional activation. , 1998, Genes & development.
[20] D. LeBrun,et al. Restricted expression of E2A protein in primary human tissues correlates with proliferation and differentiation. , 1998, The American journal of pathology.
[21] Arun J Sharma,et al. p300 Mediates Transcriptional Stimulation by the Basic Helix-Loop-Helix Activators of the Insulin Gene , 1998, Molecular and Cellular Biology.
[22] Peter E Wright,et al. Solution Structure of the KIX Domain of CBP Bound to the Transactivation Domain of CREB: A Model for Activator:Coactivator Interactions , 1997, Cell.
[23] D. Pinkel,et al. E2A deficiency leads to abnormalities in alphabeta T-cell development and to rapid development of T-cell lymphomas , 1997, Molecular and cellular biology.
[24] M. Cleary,et al. Immortalization and leukemic transformation of a myelomonocytic precursor by retrovirally transduced HRX–ENL , 1997, The EMBO journal.
[25] L. Kedes,et al. Molecular mechanisms of myogenic coactivation by p300: direct interaction with the activation domain of MyoD and with the MADS box of MEF2C , 1997, Molecular and cellular biology.
[26] D. Livingston,et al. Interaction and functional collaboration of p300/CBP and bHLH proteins in muscle and B-cell differentiation. , 1996, Genes & development.
[27] R. Evans,et al. Phosphorylation of CREB at Ser-133 induces complex formation with CREB-binding protein via a direct mechanism , 1996, Molecular and cellular biology.
[28] Q. Lu,et al. DNA-binding by oncoprotein E2a-Pbx1 is important for blocking differentiation but dispensable for fibroblast transformation. , 1996, Oncogene.
[29] P. Jennings,et al. The AD1 transactivation domain of E2A contains a highly conserved helix which is required for its activity in both Saccharomyces cerevisiae and mammalian cells , 1996, Molecular and cellular biology.
[30] Ian Krop,et al. E2A proteins are required for proper B cell development and initiation of immunoglobulin gene rearrangements , 1994, Cell.
[31] H. Weintraub,et al. The helix-loop-helix gene E2A is required for B cell formation , 1994, Cell.
[32] D. LeBrun,et al. Transformation properties of the E2a-Pbx1 chimeric oncoprotein: fusion with E2a is essential, but the Pbx1 homeodomain is dispensable , 1994, Molecular and cellular biology.
[33] M. Kamps,et al. Oncoprotein E2A-Pbx1 immortalizes a myeloid progenitor in primary marrow cultures without abrogating its factor-dependence. , 1994, Oncogene.
[34] Q. Lu,et al. Fusion with E2A converts the Pbx1 homeodomain protein into a constitutive transcriptional activator in human leukemias carrying the t(1;19) translocation , 1994, Molecular and cellular biology.
[35] D. LeBrun,et al. Fusion with E2A alters the transcriptional properties of the homeodomain protein PBX1 in t(1;19) leukemias. , 1994, Oncogene.
[36] Masatoshi Hagiwara,et al. Phosphorylated CREB binds specifically to the nuclear protein CBP , 1993, Nature.
[37] A. Aronheim,et al. The E2A gene product contains two separable and functionally distinct transcription activation domains. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[38] C. Murre,et al. Pbx1 is converted into a transcriptional activator upon acquiring the N-terminal region of E2A in pre-B-cell acute lymphoblastoid leukemia. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[39] A. Aronheim,et al. Cell-specific expression of helix-loop-helix transcription factors encoded by the E2A gene. , 1993, Nucleic acids research.
[40] C. Murre,et al. A new transcriptional-activation motif restricted to a class of helix-loop-helix proteins is functionally conserved in both yeast and mammalian cells , 1993, Molecular and cellular biology.
[41] T. Kadesch,et al. Two distinct transcription factors that bind the immunoglobulin enhancer microE5/kappa 2 motif. , 1990, Science.
[42] R. Roeder,et al. Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei. , 1983, Nucleic acids research.