Increased cellular reaction to damage caused by angiotensin in arterioles of normal recipient rats after transfer of lymphocytes from hypertensive rats.

The presence of an immunological factor in vascular disease in renal hypertensive rats is demonstrated by showing, that transfer of thoracic duct cells from these rats to normal syngeneic recipients is followed by a change in the reactivity to injections of angiotensin with the result that the normal recipients react with a secondary cellular response in the damaged arterioles.

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