Changes in pro-inflammatory cytokine levels and late-life depression: A two year population based longitudinal study
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R. Stewart | Sung-Wan Kim | Hee-Ju Kang | Ju-Wan Kim | Jae-Min Kim | I. Shin | Jin-Sang Yoon | K. Bae
[1] Sophie E. Holmes,et al. Elevated Translocator Protein in Anterior Cingulate in Major Depression and a Role for Inflammation in Suicidal Thinking: A Positron Emission Tomography Study , 2018, Biological Psychiatry.
[2] Charles F. Reynolds,et al. Management of Depression in Older Adults: A Review , 2017, JAMA.
[3] A. Carvalho,et al. Peripheral cytokine and chemokine alterations in depression: a meta‐analysis of 82 studies , 2017, Acta psychiatrica Scandinavica.
[4] Proteesh Rana,et al. Comparison of fluoxetine and 1-methyl-L-tryptophan in treatment of depression-like illness in Bacillus Calmette-Guerin-induced inflammatory model of depression in mice , 2016, Journal of basic and clinical physiology and pharmacology.
[5] Young T. Hong,et al. Neuroinflammatory and morphological changes in late-life depression: the NIMROD study , 2016, British Journal of Psychiatry.
[6] Sung-Wan Kim,et al. Associations of cytokine genes with Alzheimer's disease and depression in an elderly Korean population , 2014, Journal of Neurology, Neurosurgery & Psychiatry.
[7] C. Mady,et al. Plasma Cytokine Profile in Tropical Endomyocardial Fibrosis: Predominance of TNF-a, IL-4 and IL-10 , 2014, PloS one.
[8] W. Taylor,et al. The vascular depression hypothesis: mechanisms linking vascular disease with depression , 2013, Molecular Psychiatry.
[9] P. Sachdev,et al. Inflammatory biomarkers predict depressive, but not anxiety symptoms during aging: The prospective Sydney Memory and Aging Study , 2012, Psychoneuroendocrinology.
[10] M. Maes,et al. Mechanistic explanations how cell-mediated immune activation, inflammation and oxidative and nitrosative stress pathways and their sequels and concomitants play a role in the pathophysiology of unipolar depression , 2012, Neuroscience & Biobehavioral Reviews.
[11] Michael Maes. Depression is an inflammatory disease, but cell-mediated immune activation is the key component of depression , 2011, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[12] F. Licastro,et al. Blood Inflammatory Proteins and Risk of Incident Depression in the Elderly , 2010, Dementia and Geriatric Cognitive Disorders.
[13] T. Kamarck,et al. A prospective evaluation of the directionality of the depression–inflammation relationship , 2009, Brain, Behavior, and Immunity.
[14] S. Bandinelli,et al. Interleukin-1 Receptor Antagonist and Incident Depressive Symptoms Over 6 Years in Older Persons: The InCHIANTI Study , 2009, Biological Psychiatry.
[15] Shuran Li,et al. Prevalence of dementia in Latin America, India, and China: a population-based cross-sectional survey , 2008, The Lancet.
[16] R. Stewart,et al. Predictive value of folate, vitamin B12 and homocysteine levels in late-life depression , 2008, British Journal of Psychiatry.
[17] A. Beekman,et al. Inflammatory markers in late-life depression: results from a population-based study. , 2008, Journal of affective disorders.
[18] David Loewenstein,et al. Depression and risk for Alzheimer disease: systematic review, meta-analysis, and metaregression analysis. , 2006, Archives of general psychiatry.
[19] Kenneth E Freedland,et al. Depression, the Autonomic Nervous System, and Coronary Heart Disease , 2005, Psychosomatic medicine.
[20] J. Gottdiener,et al. The role of immune system parameters in the relationship between depression and coronary artery disease. , 2005, Psychosomatic medicine.
[21] Karina W Davidson,et al. The epidemiology, pathophysiology, and management of psychosocial risk factors in cardiac practice: the emerging field of behavioral cardiology. , 2005, Journal of the American College of Cardiology.
[22] D. Posthuma,et al. Heritability estimates of innate immunity: an extended twin study , 2005, Genes and Immunity.
[23] R. Stewart,et al. Physical health, depression and cognitive function as correlates of disability in an older Korean population , 2005, International journal of geriatric psychiatry.
[24] M. Wichers,et al. Cytokines and major depression , 2005, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[25] Ruoling Chen,et al. A community‐based study of depression in older people in Hefei, China—the GMS‐AGECAT prevalence, case validation and socio‐economic correlates , 2004, International journal of geriatric psychiatry.
[26] L. Ferrucci,et al. Inflammatory markers and depressed mood in older persons: results from the health, aging and body composition study , 2003, Biological Psychiatry.
[27] R. Stewart,et al. Diagnosing dementia in a developing nation: an evaluation of the GMS‐AGECAT algorithm in an older Korean population , 2003, International journal of geriatric psychiatry.
[28] M. Prince,et al. Dementia diagnosis in developing countries: a cross-cultural validation study , 2003, The Lancet.
[29] R. Dantzer,et al. Cytokine-induced sickness behaviour: mechanisms and implications , 2002, Trends in Neurosciences.
[30] J. Maccluer,et al. A population-based, cross-sectional survey of the Zuni Pueblo: a collaborative approach to an epidemic of kidney disease. , 2002, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[31] R. Dantzer. Cytokine‐Induced Sickness Behavior: Mechanisms and Implications , 2001, Annals of the New York Academy of Sciences.
[32] T. Tang,et al. Community study of depression in old age in Taiwan , 2001, British Journal of Psychiatry.
[33] P. Mottram,et al. Validation of the Hamilton Depression Rating Scale and Montgommery and Asberg Rating Scales in terms of AGECAT depression cases , 2000, International journal of geriatric psychiatry.
[34] M. Wong,et al. The role of inflammatory mediators in the biology of major depression: central nervous system cytokines modulate the biological substrate of depressive symptoms, regulate stress-responsive systems, and contribute to neurotoxicity and neuroprotection , 1999, Molecular Psychiatry.
[35] C. Nemeroff,et al. Psychoneuroendocrinology of depression. Hypothalamic-pituitary-adrenal axis. , 1998, The Psychiatric clinics of North America.
[36] E. Kua. A Community Study of Mental Disorders in Elderly Singaporean Chinese Using the Gms-Agecat Package , 1992, The Australian and New Zealand journal of psychiatry.
[37] J. Copeland,et al. The epidemiology of dementia: GMS-AGECAT studies of prevalence and incidence, including studies in progress , 1991, European Archives of Psychiatry and Clinical Neuroscience.
[38] J. Copeland,et al. A computerized psychiatric diagnostic system and case nomenclature for elderly subjects: GMS and AGECAT , 1986, Psychological Medicine.
[39] S. Folstein,et al. "Mini-mental state". A practical method for grading the cognitive state of patients for the clinician. , 1975, Journal of psychiatric research.
[40] R. Dantzer,et al. From inflammation to sickness and depression: when the immune system subjugates the brain , 2008, Nature Reviews Neuroscience.
[41] A. Hofman,et al. Inflammatory proteins and depression in the elderly. , 2003, Epidemiology.
[42] V. Leirer,et al. Development and validation of a geriatric depression screening scale: a preliminary report. , 1982, Journal of psychiatric research.
[43] L. Radloff. The CES-D Scale: A Self-Report Depression Scale for Research in the General Population — Source link , 2022 .