Effects of Opsonization and Gamma Interferon on Growth of Brucella , melitensis 16 M in Mouse Peritoneal

[1]  R. Parton,et al.  Brucella abortus Transits through the Autophagic Pathway and Replicates in the Endoplasmic Reticulum of Nonprofessional Phagocytes , 1998, Infection and Immunity.

[2]  E. Caron,et al.  Expression and bactericidal activity of nitric oxide synthase in Brucella suis-infected murine macrophages. , 1997, Infection and immunity.

[3]  R. Roop,et al.  The HtrA stress response protease contributes to resistance of Brucella abortus to killing by murine phagocytes , 1996, Infection and immunity.

[4]  R. van Furth,et al.  Acquired resistance against a secondary infection with Listeria monocytogenes in mice is not dependent on reactive nitrogen intermediates , 1996, Infection and immunity.

[5]  T. Hadfield,et al.  Deletion of purE attenuates Brucella melitensis 16M for growth in human monocyte-derived macrophages , 1995, Infection and immunity.

[6]  A. Vázquez-Torres,et al.  Nitric oxide production does not directly increase macrophage candidacidal activity , 1995, Infection and immunity.

[7]  P. Elzer,et al.  Antibody-mediated protection against Brucella abortus in BALB/c mice at successive periods after infection: variation between virulent strain 2308 and attenuated vaccine strain 19. , 1994, Immunology.

[8]  P. Campbell,et al.  Gentamicin kills intracellular Listeria monocytogenes , 1994, Infection and immunity.

[9]  C. Cheers,et al.  Endogenous gamma interferon mediates resistance to Brucella abortus infection , 1993, Infection and immunity.

[10]  X. Jiang,et al.  Macrophage control of Brucella abortus: role of reactive oxygen intermediates and nitric oxide. , 1993, Cellular immunology.

[11]  F. Enright,et al.  Phagocytosis, killing, and oxidant production by bovine monocyte-derived macrophages upon exposure to Brucella abortus strain 2308. , 1993, Veterinary immunology and immunopathology.

[12]  L. Schlesinger.  Macrophage phagocytosis of virulent but not attenuated strains of Mycobacterium tuberculosis is mediated by mannose receptors in addition to complement receptors. , 1993, Journal of immunology.

[13]  X. Jiang,et al.  Effects of cytokines on intracellular growth of Brucella abortus , 1993, Infection and immunity.

[14]  J S Beckman,et al.  Peroxynitrite formation from macrophage-derived nitric oxide. , 1992, Archives of biochemistry and biophysics.

[15]  A. Winter,et al.  Survival of virulent and attenuated strains of Brucella abortus in normal and gamma interferon-activated murine peritoneal macrophages , 1992, Infection and immunity.

[16]  A. Fortier,et al.  Activation of macrophages for destruction of Francisella tularensis: identification of cytokines, effector cells, and effector molecules , 1992, Infection and immunity.

[17]  S. Levitz,et al.  Binding of Cryptococcus neoformans by human cultured macrophages. Requirements for multiple complement receptors and actin. , 1991, The Journal of clinical investigation.

[18]  R. Price,et al.  Ability of mononuclear phagocytes from cattle naturally resistant or susceptible to brucellosis to control in vitro intracellular survival of Brucella abortus , 1990, Infection and immunity.

[19]  M. Horwitz,et al.  Phagocytosis of Mycobacterium tuberculosis is mediated by human monocyte complement receptors and complement component C3. , 1990, Journal of immunology.

[20]  L. G. Adams,et al.  Capacity of passively administered antibody to prevent establishment of Brucella abortus infection in mice , 1989, Infection and immunity.

[21]  J. Roth,et al.  Opsonin-dependent stimulation of bovine neutrophil oxidative metabolism by Brucella abortus. , 1988, American journal of veterinary research.

[22]  M. Horwitz,et al.  Phagocytosis of Legionella pneumophila is mediated by human monocyte complement receptors , 1987, The Journal of experimental medicine.

[23]  A. Winter,et al.  Comparison of living and nonliving vaccines for Brucella abortus in BALB/c mice , 1986, Infection and immunity.

[24]  E. J. Young,et al.  Antimicrobial susceptibility of clinical isolates of Brucella. , 1986, Diagnostic microbiology and infectious disease.

[25]  Robert B Sim,et al.  Macrophage complement and lectin-like receptors bind Leishmania in the absence of serum , 1985, The Journal of experimental medicine.

[26]  K. Yamamoto,et al.  Dissociation of phagocytosis from stimulation of the oxidative metabolic burst in macrophages , 1984, The Journal of experimental medicine.

[27]  S. Wright,et al.  Receptors for C3b and C3bi promote phagocytosis but not the release of toxic oxygen from human phagocytes , 1983, The Journal of experimental medicine.

[28]  C. Cheers,et al.  Resistance and susceptibility of mice to bacterial infection. IV. Functional specificity in natural resistance to facultative intracellular bacteria. , 1983, Journal of the Reticuloendothelial Society.

[29]  M. Plommet,et al.  Immune serum-mediated effects on brucellosis evolution in mice , 1983, Infection and immunity.

[30]  D. Musher,et al.  Opsonizing and bactericidal effects of normal human serum on nontypable Haemophilus influenzae , 1983, Infection and immunity.

[31]  R. Johnston,et al.  Role of binding through C3b and IgG in polymorphonuclear neutrophil function: studies with trypsin-generated C3b. , 1979, Journal of immunology.

[32]  G. Mackaness.  Resistance to intracellular infection. , 1971, Journal of Infectious Diseases.

[33]  G. Mackaness.  THE IMMUNOLOGICAL BASIS OF ACQUIRED CELLULAR RESISTANCE , 1964, The Journal of experimental medicine.

[34]  A. Winter,et al.  Macrophages and Brucella. , 1994, Immunology series.

[35]  M. Perry,et al.  The lipopolysaccharides of Brucella abortus and B. melitensis. , 1987, Annales de l'Institut Pasteur. Microbiology.