Macrophage roles following myocardial infarction.

[1]  Hanna Björk,et al.  ADAMTS-4 and -8 are inflammatory regulated enzymes expressed in macrophage-rich areas of human atherosclerotic plaques. , 2008, Atherosclerosis.

[2]  E. Solary,et al.  A role for caspases in the differentiation of erythroid cells and macrophages. , 2008, Biochimie.

[3]  X. Zhang,et al.  Macrophage activation by endogenous danger signals , 2008, The Journal of pathology.

[4]  D. Mukherjee,et al.  Acute coronary syndromes: unstable angina/non-ST elevation myocardial infarction. , 2007, Critical care clinics.

[5]  G. Sanz [Risk stratification in acute coronary syndromes: an unresolved issue]. , 2007, Revista espanola de cardiologia.

[6]  Y. Saijo,et al.  Macrophage colony-stimulating factor improves cardiac function after ischemic injury by inducing vascular endothelial growth factor production and survival of cardiomyocytes. , 2007, The American journal of pathology.

[7]  D. Greenhalgh,et al.  Cutaneous Wound Healing , 2007, Journal of burn care & research : official publication of the American Burn Association.

[8]  M. Lindsey,et al.  Mechanisms to inhibit matrix metalloproteinase activity: where are we in the development of clinically relevant inhibitors? , 2007, Recent patents on anti-cancer drug discovery.

[9]  N. Frangogiannis Chemokines in ischemia and reperfusion , 2007, Thrombosis and Haemostasis.

[10]  T. Kodama,et al.  Targeted Deletion of Class A Macrophage Scavenger Receptor Increases the Risk of Cardiac Rupture After Experimental Myocardial Infarction , 2007, Circulation.

[11]  M. Harmsen,et al.  Macrophage depletion impairs wound healing and increases left ventricular remodeling after myocardial injury in mice. , 2007, The American journal of pathology.

[12]  Ajai Kumar Jain,et al.  PexeLizumab for Acute ST-elevation myocardial infarction in patients undergoing primary percutaneous coronary intervention: A randomized controlled trial , 2007 .

[13]  R. Strieter,et al.  Inflammation and angiogenesis in fibrotic lung disease. , 2006, Seminars in respiratory and critical care medicine.

[14]  R. Gomer,et al.  Bone marrow-derived fibroblast precursors mediate ischemic cardiomyopathy in mice , 2006, Proceedings of the National Academy of Sciences.

[15]  N. Frangogiannis The mechanistic basis of infarct healing. , 2006, Antioxidants & redox signaling.

[16]  Geert Raes,et al.  Classical and alternative activation of mononuclear phagocytes: picking the best of both worlds for tumor promotion. , 2006, Immunobiology.

[17]  F. Dunn,et al.  Serological evidence of altered collagen homeostasis reflects early ventricular remodeling following acute myocardial infarction. , 2006, International journal of cardiology.

[18]  J. Leor,et al.  Ex Vivo Activated Human Macrophages Improve Healing, Remodeling, and Function of the Infarcted Heart , 2006, Circulation.

[19]  E. Solary,et al.  Identification of Proteins Cleaved Downstream of Caspase Activation in Monocytes Undergoing Macrophage Differentiation* , 2006, Journal of Biological Chemistry.

[20]  B. Willis,et al.  Epithelial origin of myofibroblasts during fibrosis in the lung. , 2006, Proceedings of the American Thoracic Society.

[21]  V. Chopra,et al.  Tissue inhibitor of metalloproteinase-1 (TIMP-1) is an independent predictor of all-cause mortality, cardiac mortality, and myocardial infarction. , 2006, American heart journal.

[22]  C. Overall,et al.  Towards third generation matrix metalloproteinase inhibitors for cancer therapy , 2006, British Journal of Cancer.

[23]  Xu-wen Liu,et al.  Novel functions of TIMPs in cell signaling , 2006, Cancer and Metastasis Reviews.

[24]  M. Caldwell,et al.  Mactinin treatment promotes wound‐healing‐associated inflammation in urokinase knockout mice , 2006, Wound repair and regeneration : official publication of the Wound Healing Society [and] the European Tissue Repair Society.

[25]  J. Peterson The importance of estimating the therapeutic index in the development of matrix metalloproteinase inhibitors. , 2006, Cardiovascular research.

[26]  Gillian Murphy,et al.  Structure and function of matrix metalloproteinases and TIMPs. , 2006, Cardiovascular research.

[27]  K. Shimamoto,et al.  Macrophage colony-stimulating factor treatment after myocardial infarction attenuates left ventricular dysfunction by accelerating infarct repair. , 2006, Journal of the American College of Cardiology.

[28]  Marc A Pfeffer,et al.  Controversies in ventricular remodelling , 2006, The Lancet.

[29]  John Condeelis,et al.  Macrophages: Obligate Partners for Tumor Cell Migration, Invasion, and Metastasis , 2006, Cell.

[30]  J. Pollard,et al.  Distinct role of macrophages in different tumor microenvironments. , 2006, Cancer research.

[31]  F. Antohe Endothelial cells and macrophages, partners in atherosclerotic plaque progression , 2006, Archives of physiology and biochemistry.

[32]  Richard T. Lee,et al.  Matrix metalloproteinase-9 gene deletion facilitates angiogenesis after myocardial infarction. , 2006, American journal of physiology. Heart and circulatory physiology.

[33]  M. Entman,et al.  Extracellular matrix remodeling in canine and mouse myocardial infarcts , 2006, Cell and Tissue Research.

[34]  Peter Carmeliet,et al.  Angiogenesis in life, disease and medicine , 2005, Nature.

[35]  Craig Murdoch,et al.  Macrophage migration and gene expression in response to tumor hypoxia , 2005, International journal of cancer.

[36]  D. Dormont,et al.  Macrophage activation switching: an asset for the resolution of inflammation , 2005, Clinical and experimental immunology.

[37]  J. Duffield,et al.  Conditional ablation of macrophages halts progression of crescentic glomerulonephritis. , 2005, The American journal of pathology.

[38]  W. Burns,et al.  Connective Tissue Growth Factor and Cardiac Fibrosis after Myocardial Infarction , 2005, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[39]  Kazuo Kobayashi,et al.  Macrophages in inflammation. , 2005, Current drug targets. Inflammation and allergy.

[40]  B. Rollins,et al.  CCL2/Monocyte Chemoattractant Protein-1 Regulates Inflammatory Responses Critical to Healing Myocardial Infarcts , 2005, Circulation research.

[41]  L. Edelstein-Keshet,et al.  Quantifying macrophage defects in type 1 diabetes. , 2005, Journal of theoretical biology.

[42]  G. Mazur,et al.  Kinetics of chemokines in acute myocardial infarction. , 2005, Kardiologia polska.

[43]  M. Simons Angiogenesis: where do we stand now? , 2005, Circulation.

[44]  S. Forbes,et al.  Selective depletion of macrophages reveals distinct, opposing roles during liver injury and repair. , 2005, The Journal of clinical investigation.

[45]  V. Roger,et al.  Heart failure after myocardial infarction: clinical presentation and survival , 2005, European journal of heart failure.

[46]  N. Moldovan,et al.  Role of monocytes and macrophages in angiogenesis. , 2005, EXS.

[47]  T. Borg,et al.  Structural and functional characterisation of cardiac fibroblasts. , 2005, Cardiovascular research.

[48]  J. Boyle,et al.  Macrophage activation in atherosclerosis: pathogenesis and pharmacology of plaque rupture. , 2005, Current vascular pharmacology.

[49]  M. Schellings,et al.  Matricellular proteins in the heart: possible role during stress and remodeling. , 2004, Cardiovascular research.

[50]  Robert M Califf,et al.  Relation between renal dysfunction and cardiovascular outcomes after myocardial infarction. , 2004, The New England journal of medicine.

[51]  M. Burdick,et al.  Circulating fibrocytes traffic to the lungs in response to CXCL12 and mediate fibrosis. , 2004, The Journal of clinical investigation.

[52]  Shawn Cowper,et al.  Circulating fibrocytes: collagen-secreting cells of the peripheral blood. , 2004, The international journal of biochemistry & cell biology.

[53]  S. Bijlsma,et al.  A combination of proteomics, principal component analysis and transcriptomics is a powerful tool for the identification of biomarkers for macrophage maturation in the U937 cell line , 2004, Proteomics.

[54]  W. Hornebeck,et al.  An introduction to matrikines: extracellular matrix-derived peptides which regulate cell activity. Implication in tumor invasion. , 2004, Critical reviews in oncology/hematology.

[55]  A. Yndestad,et al.  Connective tissue growth factor--a novel mediator of angiotensin II-stimulated cardiac fibroblast activation in heart failure in rats. , 2004, Journal of molecular and cellular cardiology.

[56]  L. Martiny,et al.  Matrix metalloproteinases and matrikines in angiogenesis. , 2004, Critical reviews in oncology/hematology.

[57]  M. Fini,et al.  Matrix Metalloproteinase-9 Is Required for Adequate Angiogenic Revascularization of Ischemic Tissues: Potential Role in Capillary Branching , 2004, Circulation research.

[58]  K. Weber,et al.  Tissue repair and angiotensin II generated at sites of healing , 1997, Basic Research in Cardiology.

[59]  S. Solomon,et al.  The decreasing incidence of left ventricular remodeling following myocardial infarction , 1997, Basic Research in Cardiology.

[60]  M. Simons,et al.  Re-evaluating therapeutic neovascularization. , 2004, Journal of molecular and cellular cardiology.

[61]  J. Peterson Matrix Metalloproteinase Inhibitor Development and the Remodeling of Drug Discovery , 2004, Heart Failure Reviews.

[62]  Merry L. Lindsey,et al.  MMP Induction and Inhibition in Myocardial Infarction , 2004, Heart Failure Reviews.

[63]  Douglas L Mann,et al.  Stress-activated cytokines and the heart: from adaptation to maladaptation. , 2003, Annual review of physiology.

[64]  S. Lavine Prediction of Heart Failure Post Myocardial Infarction: Comparison of Ejection Fraction, Transmitral Filling Parameters, and the Index of Myocardial Performance , 2003, Echocardiography.

[65]  M. Pfeffer,et al.  Predictors of late development of heart failure in stable survivors of myocardial infarction: the CARE study. , 2003, Journal of the American College of Cardiology.

[66]  U. Ikeda,et al.  Inflammatory cytokines and cardiovascular disease. , 2003, Current drug targets. Inflammation and allergy.

[67]  O. Dewald,et al.  Inflammatory mechanisms in myocardial infarction. , 2003, Current drug targets. Inflammation and allergy.

[68]  F. Sellebjerg,et al.  Chemokines and matrix metalloproteinase-9 in leukocyte recruitment to the central nervous system , 2003, Brain Research Bulletin.

[69]  K. Csaky,et al.  Macrophage depletion diminishes lesion size and severity in experimental choroidal neovascularization. , 2003, Investigative ophthalmology & visual science.

[70]  M. Entman,et al.  MCSF expression is induced in healing myocardial infarcts and may regulate monocyte and endothelial cell phenotype. , 2003, American journal of physiology. Heart and circulatory physiology.

[71]  J. Ambati,et al.  Macrophage depletion inhibits experimental choroidal neovascularization. , 2003, Investigative ophthalmology & visual science.

[72]  D. Hsu,et al.  Critical role of galectin-3 in phagocytosis by macrophages. , 2003, The Journal of clinical investigation.

[73]  V. Quaranta,et al.  Tales from the crypt[ic] sites of the extracellular matrix. , 2003, Trends in cell biology.

[74]  B. Yawn,et al.  Incidence of heart failure after myocardial infarction: is it changing over time? , 2003, American journal of epidemiology.

[75]  R. Visse,et al.  This Review Is Part of a Thematic Series on Matrix Metalloproteinases, Which Includes the following Articles: Matrix Metalloproteinase Inhibition after Myocardial Infarction: a New Approach to Prevent Heart Failure? Matrix Metalloproteinases in Vascular Remodeling and Atherogenesis: the Good, the Ba , 2022 .

[76]  R. Marfella,et al.  Ventricular remodeling does not accompany the development of heart failure in diabetic patients after myocardial infarction. , 2003, Circulation.

[77]  Armin Helisch,et al.  Arteriogenesis The Development and Growth of Collateral Arteries , 2003, Microcirculation.

[78]  D. Mosser,et al.  The many faces of macrophage activation , 2003, Journal of leukocyte biology.

[79]  William C. Parks,et al.  Matrilysin Shedding of Syndecan-1 Regulates Chemokine Mobilization and Transepithelial Efflux of Neutrophils in Acute Lung Injury , 2002, Cell.

[80]  Masanori Hangai,et al.  Matrix metalloproteinase-9-dependent exposure of a cryptic migratory control site in collagen is required before retinal angiogenesis. , 2002, The American journal of pathology.

[81]  I. Fishbein,et al.  Macrophage Depletion by Clodronate-Containing Liposomes Reduces Neointimal Formation After Balloon Injury in Rats and Rabbits , 2002, Circulation.

[82]  Z. Werb,et al.  New functions for the matrix metalloproteinases in cancer progression , 2002, Nature Reviews Cancer.

[83]  G. Laurie,et al.  Exposure of Cryptic Domains in the α1-chain of Laminin-1 by Elastase Stimulates Macrophages Urokinase and Matrix Metalloproteinase-9 Expression* , 2002, The Journal of Biological Chemistry.

[84]  G. Thibault,et al.  Is angiotensin II a proliferative factor of cardiac fibroblasts? , 2002, Cardiovascular research.

[85]  J. Peterson,et al.  Matrix metalloproteinase inhibitor development for the treatment of heart failure , 2002 .

[86]  M. Entman,et al.  The inflammatory response in myocardial infarction. , 2002, Cardiovascular research.

[87]  G. Horstick C1-esterase inhibitor in ischemia and reperfusion. , 2002, Immunobiology.

[88]  Noam Brown,et al.  Microenvironmental influence on macrophage regulation of angiogenesis in wounds and malignant tumors , 2001, Journal of leukocyte biology.

[89]  Paul J. McMillan,et al.  Angiogenesis after Stroke is Correlated with Increased Numbers of Macrophages: The Clean-up Hypothesis , 2001, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.

[90]  Jingsong Xu,et al.  Proteolytic exposure of a cryptic site within collagen type IV is required for angiogenesis and tumor growth in vivo , 2001, The Journal of cell biology.

[91]  M. Entman,et al.  Regeneration of ischemic cardiac muscle and vascular endothelium by adult stem cells. , 2001, The Journal of clinical investigation.

[92]  I. Eue,et al.  Growth inhibition of human mammary carcinoma by liposomal hexadecylphosphocholine: Participation of activated macrophages in the antitumor mechanism , 2001, International journal of cancer.

[93]  Merry Lindsey,et al.  Matrix-Dependent Mechanism of Neutrophil-Mediated Release and Activation of Matrix Metalloproteinase 9 in Myocardial Ischemia/Reperfusion , 2001, Circulation.

[94]  S. Solomon,et al.  Recovery of Ventricular Function after Myocardial Infarction in the Reperfusion Era: The Healing and Early Afterload Reducing Therapy Study , 2001, Annals of Internal Medicine.

[95]  S. Tyagi,et al.  Tissue inhibitor of metalloproteinase‐4 instigates apoptosis in transformed cardiac fibroblasts , 2001, Journal of cellular biochemistry.

[96]  P. Libby,et al.  An HMG-CoA Reductase Inhibitor, Cerivastatin, Suppresses Growth of Macrophages Expressing Matrix Metalloproteinases and Tissue Factor In Vivo and In Vitro , 2001, Circulation.

[97]  W. Colucci,et al.  Oxidative stress regulates collagen synthesis and matrix metalloproteinase activity in cardiac fibroblasts. , 2001, American journal of physiology. Cell physiology.

[98]  K. Matsushima,et al.  Comprehensive gene expression profile of LPS-stimulated human monocytes by SAGE. , 2000, Blood.

[99]  B Cao,et al.  The potential role of PDGF, IGF-1, TGF-beta expression in idiopathic pulmonary fibrosis. , 2000, Chinese medical journal.

[100]  M. Bendeck Mining the myocardium with macrophage drills: A novel mechanism for revascularization. , 2000, Circulation research.

[101]  S. Shapiro,et al.  Contribution of Monocytes/Macrophages to Compensatory Neovascularization: The Drilling of Metalloelastase-Positive Tunnels in Ischemic Myocardium , 2000, Circulation research.

[102]  B. Antus,et al.  Influence of alternatively and classically activated macrophages on fibrogenic activities of human fibroblasts. , 2000, Cellular immunology.

[103]  N. Ohkohchi,et al.  Macrophage depletion prevents accelerated rejection and results in long-term survival in hamster to rat cardiac xenotransplantation. , 2000, Transplantation proceedings.

[104]  A. Matsumori,et al.  Roles and Relationship of Macrophages and Monocyte Chemotactic and Activating Factor/Monocyte Chemoattractant Protein-1 in the Ischemic and Reperfused Rat Heart , 2000, Laboratory Investigation.

[105]  P. Libby,et al.  Targeted deletion of matrix metalloproteinase-9 attenuates left ventricular enlargement and collagen accumulation after experimental myocardial infarction. , 2000, The Journal of clinical investigation.

[106]  N. Sharpe,et al.  Left ventricular remodeling after myocardial infarction: pathophysiology and therapy. , 2000, Circulation.

[107]  R. Rezzonico,et al.  Th2 Cell Membrane Factors in Association with IL-4 Enhance Matrix Metalloproteinase-1 (MMP-1) While Decreasing MMP-9 Production by Granulocyte-Macrophage Colony-Stimulating Factor-Differentiated Human Monocytes1 , 2000, The Journal of Immunology.

[108]  D. Tracey,et al.  Depletion of macrophages reduces axonal degeneration and hyperalgesia following nerve injury , 2000, Pain.

[109]  K. Weber,et al.  Infarct scar: a dynamic tissue. , 2000, Cardiovascular research.

[110]  D. Mackenna,et al.  Role of mechanical factors in modulating cardiac fibroblast function and extracellular matrix synthesis. , 2000, Cardiovascular research.

[111]  G. Davis,et al.  Regulation of tissue injury responses by the exposure of matricryptic sites within extracellular matrix molecules. , 2000, The American journal of pathology.

[112]  J. Bryant,et al.  Evolution of matrix metalloprotease and tissue inhibitor expression during heart failure progression in the infarcted rat. , 2000, Cardiovascular research.

[113]  E. Tremoli,et al.  Angiotensin-converting enzyme inhibitors downregulate tissue factor synthesis in monocytes. , 2000, Circulation research.

[114]  Constantin E. Orfanos,et al.  Alternative versus Classical Activation of Macrophages , 2000, Pathobiology.

[115]  D. Cucinotta,et al.  Cardioprotection by the phytoestrogen genistein in experimental myocardial ischaemia‐reperfusion injury , 1999, British journal of pharmacology.

[116]  B. Greenberg,et al.  Tumor Necrosis Factor- (cid:97) Upregulates Angiotensin II Type 1 Receptors on Cardiac Fibroblasts , 1999 .

[117]  S. Shapiro Diverse Roles of Macrophage Matrix Metalloproteinases in Tissue Destruction and Tumor Growth , 1999, Thrombosis and Haemostasis.

[118]  M. Kuwano,et al.  Biological implications of macrophage infiltration in human tumor angiogenesis , 1999, Cancer Chemotherapy and Pharmacology.

[119]  J. Varani,et al.  Role of matrix metalloproteinases in models of macrophage-dependent acute lung injury. Evidence for alveolar macrophage as source of proteinases. , 1999, American journal of respiratory cell and molecular biology.

[120]  S. Weiss,et al.  Characterization of matrix metalloproteinases produced by rat alveolar macrophages. , 1999, American journal of respiratory cell and molecular biology.

[121]  B. Greenberg,et al.  Tumor necrosis factor-alpha upregulates angiotensin II type 1 receptors on cardiac fibroblasts. , 1999, Circulation research.

[122]  D. Falcone,et al.  Macrophage Formation of Angiostatin during Inflammation , 1998, The Journal of Biological Chemistry.

[123]  A. Rees,et al.  Initial cytokine exposure determines function of macrophages and renders them unresponsive to other cytokines. , 1998, Journal of immunology.

[124]  S. Tyagi,et al.  Stretch‐induced membrane type matrix metalloproteinase and tissue plasminogen activator in cardiac fibroblast cells , 1998, Journal of cellular physiology.

[125]  U. Shankavaram,et al.  Lipopolysaccharide induction of monocyte matrix metalloproteinases is regulated by the tyrosine phosphorylation of cytosolic phospholipase A2 , 1998, Journal of leukocyte biology.

[126]  P. Anversa,et al.  Ventricular myocytes are not terminally differentiated in the adult mammalian heart. , 1998, Circulation research.

[127]  R. Ziegler,et al.  Molecular cloning of genes differentially regulated by TNF-alpha in bovine aortic endothelial cells, fibroblasts and smooth muscle cells. , 1998, Cardiovascular research.

[128]  S. Goerdt,et al.  Alternative macrophage activation-associated CC-chemokine-1, a novel structural homologue of macrophage inflammatory protein-1 alpha with a Th2-associated expression pattern. , 1998, Journal of immunology.

[129]  R. Bucala,et al.  Regulated production of type I collagen and inflammatory cytokines by peripheral blood fibrocytes. , 1998, Journal of immunology.

[130]  K. Weber,et al.  Extracellular matrix remodeling in heart failure: a role for de novo angiotensin II generation. , 1997, Circulation.

[131]  G. Lamas,et al.  Cardiovascular death and left ventricular remodeling two years after myocardial infarction: baseline predictors and impact of long-term use of captopril: information from the Survival and Ventricular Enlargement (SAVE) trial. , 1997, Circulation.

[132]  B. C. Patterson,et al.  Angiostatin-converting Enzyme Activities of Human Matrilysin (MMP-7) and Gelatinase B/Type IV Collagenase (MMP-9)* , 1997, The Journal of Biological Chemistry.

[133]  L. Minutoli,et al.  17β-oestradiol reduces cardiac leukocyte accumulation in myocardial ischaemia reperfusion injury in rat , 1997 .

[134]  K. Weber,et al.  Fibrous tissue and angiotensin II. , 1997, Journal of molecular and cellular cardiology.

[135]  J. Faul,et al.  T‐cell cytokines may control the balance of functionally distinct macrophage populations , 1997, Immunology.

[136]  C. Mackay,et al.  Induction of monocyte chemoattractant protein-1 in the small veins of the ischemic and reperfused canine myocardium. , 1997, Circulation.

[137]  L. Minutoli,et al.  17Beta-oestradiol reduces cardiac leukocyte accumulation in myocardial ischaemia reperfusion injury in rat. , 1997, European journal of pharmacology.

[138]  K. Weber,et al.  In situ production of angiotensin II by fibrosed rat pericardium. , 1996, Journal of molecular and cellular cardiology.

[139]  G. Booz,et al.  Molecular signalling mechanisms controlling growth and function of cardiac fibroblasts. , 1995, Cardiovascular research.

[140]  E. Miyamoto,et al.  Increased angiotensin converting enzyme activity in left ventricular aneurysm of patients after myocardial infarction. , 1995, Cardiovascular research.

[141]  R. Bohle,et al.  Cellular distribution of angiotensin-converting enzyme after myocardial infarction. , 1995, Hypertension.

[142]  G. Kingsley The handbook of immunopharmacology , 1995 .

[143]  M. Pfeffer,et al.  Angiotensin-converting enzyme inhibition and ventricular remodeling after myocardial infarction. , 1995, Annual review of physiology.

[144]  K. Weber,et al.  Cardiac angiotensin converting enzyme and myocardial fibrosis in the rat. , 1994, Cardiovascular research.

[145]  Cord Sunderkötter,et al.  Macrophages and angiogenesis , 1994, Journal of leukocyte biology.

[146]  I. Kimura,et al.  Interferon-γ-activated macrophages enhance angiogenesis from endothelial cells of rat aorta , 1994 .

[147]  S. Kobayashi,et al.  Interferon-gamma-activated macrophages enhance angiogenesis from endothelial cells of rat aorta. , 1994, Immunopharmacology.

[148]  K. Weber,et al.  Effects of endothelins on collagen turnover in cardiac fibroblasts. , 1993, Cardiovascular research.

[149]  A. Desmoulière,et al.  Transforming growth factor-beta 1 induces alpha-smooth muscle actin expression in granulation tissue myofibroblasts and in quiescent and growing cultured fibroblasts , 1993, The Journal of cell biology.

[150]  S. Goerdt,et al.  Inducible expression of MS-1 high-molecular-weight protein by endothelial cells of continuous origin and by dendritic cells/macrophages in vivo and in vitro. , 1993, The American journal of pathology.

[151]  G. Lamas,et al.  Ventricular remodeling after myocardial infarction. , 1993, Advances in experimental medicine and biology.

[152]  T. Ganz Macrophage function. , 1993, New horizons.

[153]  E. J. Brown,et al.  Effect of captopril on mortality and morbidity in patients with left ventricular dysfunction after myocardial infarction. Results of the survival and ventricular enlargement trial. The SAVE Investigators. , 1992, The New England journal of medicine.

[154]  S Gordon,et al.  Interleukin 4 potently enhances murine macrophage mannose receptor activity: a marker of alternative immunologic macrophage activation , 1992, The Journal of experimental medicine.

[155]  H. Schunkert,et al.  Tissue-specific activation of cardiac angiotensin converting enzyme in experimental heart failure. , 1991, Circulation research.

[156]  K. Schulze-Osthoff,et al.  Macrophage-derived angiogenesis factors. , 1991, Pharmacology & therapeutics.

[157]  M. Pfeffer,et al.  Ventricular Remodeling After Myocardial Infarction: Experimental Observations and Clinical Implications , 1990, Circulation.

[158]  P. Hart,et al.  Augmentation of glucocorticoid action on human monocytes by interleukin-4. , 1990, Lymphokine research.

[159]  Donald E. Ingber,et al.  How does extracellular matrix control capillary morphogenesis? , 1989, Cell.

[160]  P. Hart,et al.  Potential antiinflammatory effects of interleukin 4: suppression of human monocyte tumor necrosis factor alpha, interleukin 1, and prostaglandin E2. , 1989, Proceedings of the National Academy of Sciences of the United States of America.

[161]  D. Rifkin,et al.  In vitro angiogenesis on the human amniotic membrane: requirement for basic fibroblast growth factor-induced proteinases , 1989, The Journal of cell biology.

[162]  D. Rappolee,et al.  Secretory products of phagocytes. , 1988, Current opinion in immunology.

[163]  R. Clark,et al.  Cryptic chemotactic activity of fibronectin for human monocytes resides in the 120-kDa fibroblastic cell-binding fragment. , 1988, The Journal of biological chemistry.

[164]  M. Entman,et al.  Mechanism of Complement Activation After Coronary Artery Occlusion: Evidence That Myocardial Ischemia in Dogs Causes Release of Constituents of Myocardial Subcellular Origin That Complex With Human C1q In Vivo , 1988, Circulation research.

[165]  H. Weisman,et al.  Steroid administration after myocardial infarction promotes early infarct expansion. A study in the rat. , 1987, The Journal of clinical investigation.

[166]  C. Nathan,et al.  Secretory products of macrophages. , 1987, The Journal of clinical investigation.

[167]  R. Voegeli,et al.  A monoclonal antibody to a novel differentiation antigen on human macrophages associated with the down-regulatory phase of the inflammatory process. , 1987, Experimental cell biology.

[168]  M. Mori,et al.  Healing of focal injury in the rat liver. , 1985, The American journal of pathology.

[169]  E. Braunwald,et al.  Dose-dependent effects of short-term methylprednisolone on myocardial infarct extent, scar formation, and ventricular function. , 1983, Circulation.

[170]  M. Fishbein,et al.  The histopathologic evolution of myocardial infarction. , 1978, Chest.

[171]  J. Folkman,et al.  Migration and proliferation of endothelial cells in preformed and newly formed blood vessels during tumor angiogenesis. , 1977, Microvascular research.