The Poly(A)-Binding Protein Nuclear 1 Suppresses Alternative Cleavage and Polyadenylation Sites
暂无分享,去创建一个
R. Elkon | R. Agami | D. Rubinsztein | F. Menzies | J. Drost | M. Jenal | F. Loayza-Puch | G. Haaften | U. Kühn | J. O. Vrielink | Arnold J. Bos | K. Rooijers | Fabricio Loayza-Puch
[1] Reuven Agami,et al. 3′UTR-Mediated Gene Silencing of the Mixed Lineage Leukemia (MLL) Gene , 2011, PloS one.
[2] K. Nishida,et al. Mechanisms and consequences of alternative polyadenylation. , 2011, Molecules and Cells.
[3] R. Agami,et al. MicroRNA regulation by RNA-binding proteins and its implications for cancer , 2011, Nature Reviews Cancer.
[4] Chong-Jian Chen,et al. Differential genome-wide profiling of tandem 3' UTRs among human breast cancer and normal cells by high-throughput sequencing. , 2011, Genome research.
[5] Peter J. Shepard,et al. Complex and dynamic landscape of RNA polyadenylation revealed by PAS-Seq. , 2011, RNA.
[6] D. Rubinsztein,et al. Over-expression of BCL2 rescues muscle weakness in a mouse model of oculopharyngeal muscular dystrophy , 2011, Human molecular genetics.
[7] D. Bartel,et al. Formation, Regulation and Evolution of Caenorhabditis elegans 3′UTRs , 2010, Nature.
[8] P. Kapranov,et al. Comprehensive Polyadenylation Site Maps in Yeast and Human Reveal Pervasive Alternative Polyadenylation , 2010, Cell.
[9] A. Virtanen,et al. To polyadenylate or to deadenylate , 2010, Cell cycle.
[10] R. Elkon,et al. A Pumilio-induced RNA structure switch in p27-3′ UTR controls miR-221 and miR-222 accessibility , 2010, Nature Cell Biology.
[11] W. Filipowicz,et al. Regulation of mRNA translation and stability by microRNAs. , 2010, Annual review of biochemistry.
[12] J. Lemay,et al. Crossing the borders: Poly(A)-binding proteins working on both sides of the fence , 2010, RNA biology.
[13] Bin Tian,et al. A functional human Poly(A) site requires only a potent DSE and an A-rich upstream sequence , 2010, The EMBO journal.
[14] Andrew H. Beck,et al. 3′-End Sequencing for Expression Quantification (3SEQ) from Archival Tumor Samples , 2010, PloS one.
[15] David Haussler,et al. The UCSC Genome Browser database: update 2010 , 2009, Nucleic Acids Res..
[16] Peter M Woollard. Asking complex questions of the genome without programming. , 2010, Methods in molecular biology.
[17] M. Schatz,et al. Searching for SNPs with cloud computing , 2009, Genome Biology.
[18] A. Riccio,et al. To localize or not to localize: mRNA fate is in 3'UTR ends. , 2009, Trends in cell biology.
[19] C. Mayr,et al. Widespread Shortening of 3′UTRs by Alternative Cleavage and Polyadenylation Activates Oncogenes in Cancer Cells , 2009, Cell.
[20] E. Wahle,et al. Poly(A) Tail Length Is Controlled by the Nuclear Poly(A)-binding Protein Regulating the Interaction between Poly(A) Polymerase and the Cleavage and Polyadenylation Specificity Factor* , 2009, The Journal of Biological Chemistry.
[21] B. Tian,et al. Progressive lengthening of 3′ untranslated regions of mRNAs by alternative polyadenylation during mouse embryonic development , 2009, Proceedings of the National Academy of Sciences.
[22] J. Yates,et al. Molecular architecture of the human pre-mRNA 3' processing complex. , 2009, Molecular cell.
[23] C. Lutz,et al. Alternative polyadenylation: a twist on mRNA 3' end formation. , 2008, ACS chemical biology.
[24] R. Shamir,et al. Transcription factor and microRNA motif discovery: the Amadeus platform and a compendium of metazoan target sets. , 2008, Genome research.
[25] P. Sharp,et al. Proliferating Cells Express mRNAs with Shortened 3' Untranslated Regions and Fewer MicroRNA Target Sites , 2008, Science.
[26] R. Agami,et al. Interplay between microRNAs and RNA-binding proteins determines developmental processes , 2008, Cell cycle.
[27] W. Filipowicz,et al. Mechanisms of post-transcriptional regulation by microRNAs: are the answers in sight? , 2008, Nature Reviews Genetics.
[28] Reuven Agami,et al. RNA-Binding Protein Dnd1 Inhibits MicroRNA Access to Target mRNA , 2007, Cell.
[29] Reuven Agami,et al. Regulation of the p27Kip1 tumor suppressor by miR‐221 and miR‐222 promotes cancer cell proliferation , 2007 .
[30] L. Lim,et al. MicroRNA targeting specificity in mammals: determinants beyond seed pairing. , 2007, Molecular cell.
[31] Bin Tian,et al. PolyA_DB 2: mRNA polyadenylation sites in vertebrate genes , 2007, Nucleic Acids Res..
[32] Reuven Agami,et al. Regulation of the p27(Kip1) tumor suppressor by miR-221 and miR-222 promotes cancer cell proliferation. , 2007, The EMBO journal.
[33] Mariette Schrier,et al. A Genetic Screen Implicates miRNA-372 and miRNA-373 As Oncogenes in Testicular Germ Cell Tumors , 2006, Cell.
[34] L. Kappos,et al. Oculopharyngeal muscular dystrophy - an under-diagnosed disorder? , 2005, Swiss medical weekly.
[35] B. Tian,et al. Bioinformatic identification of candidate cis-regulatory elements involved in human mRNA polyadenylation. , 2005, RNA.
[36] D. Rubinsztein,et al. Doxycycline attenuates and delays toxicity of the oculopharyngeal muscular dystrophy mutation in transgenic mice , 2005, Nature Medicine.
[37] D. Bentley,et al. Rules of engagement: co-transcriptional recruitment of pre-mRNA processing factors. , 2005, Current opinion in cell biology.
[38] M. Carmo-Fonseca,et al. In vivo aggregation properties of the nuclear poly(A)-binding protein PABPN1. , 2005, RNA.
[39] Bin Tian,et al. A large-scale analysis of mRNA polyadenylation of human and mouse genes , 2005, Nucleic acids research.
[40] S. Masich,et al. Nuclear poly(A)-binding protein PABPN1 is associated with RNA polymerase II during transcription and accompanies the released transcript to the nuclear pore. , 2003, Experimental cell research.
[41] D. Rubinsztein,et al. Mammalian, Yeast, Bacterial, and Chemical Chaperones Reduce Aggregate Formation and Death in a Cell Model of Oculopharyngeal Muscular Dystrophy* , 2002, The Journal of Biological Chemistry.
[42] D. Gautheret,et al. Patterns of variant polyadenylation signal usage in human genes. , 2000, Genome research.
[43] U. Kutay,et al. Deciphering the cellular pathway for transport of poly(A)-binding protein II. , 2000, RNA.
[44] J. Manley,et al. Levels of polyadenylation factor CstF-64 control IgM heavy chain mRNA accumulation and other events associated with B cell differentiation. , 1998, Molecular cell.
[45] J. Rommens,et al. Short GCG expansions in the PABP2 gene cause oculopharyngeal muscular dystrophy , 1998, Nature Genetics.
[46] S. Blumen,et al. Epidemiology and inheritance of oculopharyngeal muscular dystrophy in Israel , 1997, Neuromuscular Disorders.
[47] F. Thomé,et al. CAPD in southern Brazil: an epidemiological study. , 1997, Advances in peritoneal dialysis. Conference on Peritoneal Dialysis.
[48] J. Manley,et al. The Polyadenylation Factor CstF-64 Regulates Alternative Processing of IgM Heavy Chain Pre-mRNA during B Cell Differentiation , 1996, Cell.
[49] E. Wahle,et al. Poly(A) Tail Length Control Is Caused by Termination of Processive Synthesis (*) , 1995, The Journal of Biological Chemistry.
[50] B. Hames,et al. RNA processing : a practical approach , 1994 .
[51] J. Bonner,et al. Differentiation , 1968, Nature.