Matrix metalloproteinase-9 is elevated in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced parkinsonism in mice
暂无分享,去创建一个
S. Lorenzl | T. Joh | M. Beal | H. Krell | D. S. Albers | Lichuan Yang | N. Calingasan | Shuei Shugama | J. Gregorio | J. Chirichigno | Jason W. Chirichigno
[1] D. Boyd,et al. Regulation of matrix metalloproteinase gene expression , 2007, Journal of cellular physiology.
[2] Susanna M. Grzeschik,et al. Neurodegeneration in Striatum Induced by the Mitochondrial Toxin 3-Nitropropionic Acid: Role of Matrix Metalloproteinase-9 in Early Blood-Brain Barrier Disruption? , 2003, The Journal of Neuroscience.
[3] Norman Relkin,et al. Increased plasma levels of matrix metalloproteinase-9 in patients with Alzheimer’s disease , 2003, Neurochemistry International.
[4] E. Hirsch,et al. The Role of Glial Reaction and Inflammation in Parkinson's Disease , 2003, Annals of the New York Academy of Sciences.
[5] Subramanian Rajagopalan,et al. Genetic or Pharmacological Iron Chelation Prevents MPTP-Induced Neurotoxicity In Vivo A Novel Therapy for Parkinson's Disease , 2003, Neuron.
[6] B. Volpe,et al. Age-related microglial activation in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-induced dopaminergic neurodegeneration in C57BL/6 mice , 2003, Brain Research.
[7] S. Loening,et al. Plasma matrix metalloproteinase 9 as biomarker of prostate cancer progression in Dunning (Copenhagen) rats , 2003, The Prostate.
[8] 猪原 匡史. Chronic cerebral hypoperfusion induces MMP-2 but not MMP-9 expression in the microglia and vascular endothelium of white matter , 2003 .
[9] S. Lorenzl,et al. Expression of MMP-2, MMP-9, and MMP-1 and Their Endogenous Counterregulators TIMP-1 and TIMP-2 in Postmortem Brain Tissue of Parkinson's Disease , 2002, Experimental Neurology.
[10] Jiankun Cui,et al. S-Nitrosylation of Matrix Metalloproteinases: Signaling Pathway to Neuronal Cell Death , 2002, Science.
[11] M. Fini,et al. Secretion of matrix metalloproteinase-2 and -9 after mechanical trauma injury in rat cortical cultures and involvement of MAP kinase. , 2002, Journal of neurotrauma.
[12] L. Kaczmarek,et al. Matrix Metalloproteinase-9 Undergoes Expression and Activation during Dendritic Remodeling in Adult Hippocampus , 2002, The Journal of Neuroscience.
[13] M. Beal,et al. Caspase-9 Activation Results in Downstream Caspase-8 Activation and Bid Cleavage in 1-Methyl-4-Phenyl-1,2,3,6-Tetrahydropyridine-Induced Parkinson's Disease , 2001, The Journal of Neuroscience.
[14] S. Itohara,et al. Matrix metalloproteinase 2 gene knockout has no effect on acute brain injury after focal ischemia , 2001, Neuroreport.
[15] E. Hirsch,et al. Caspase‐3 activation in 1‐methyl‐4‐phenyl‐1,2,3,6‐tetrahydropyridine (MPTP)‐treated mice , 2001, Movement disorders : official journal of the Movement Disorder Society.
[16] T. Hattori,et al. Expression of matrix metalloproteinase-9 and urinary-type plasminogen activator in Alzheimer's disease brain. , 2001, Clinical neuropathology.
[17] P. Gottschall,et al. Regional and age-related expression of gelatinases in the brains of young and old rats after treatment with kainic acid , 2000, Neuroscience Letters.
[18] F. Weber,et al. Matrix metalloproteinase‐9 is elevated in serum of patients with amyotrophic lateral sclerosis , 2000, Neuroreport.
[19] C. Morris,et al. Brain matrix metalloproteinase 1 levels are elevated in Alzheimer's disease , 2000, Neuroscience Letters.
[20] R. Scott,et al. MPTP Activates c‐Jun NH2‐Terminal Kinase (JNK) and Its Upstream Regulatory Kinase MKK4 in Nigrostriatal Neurons In Vivo , 2000, Journal of neurochemistry.
[21] T. Hattori,et al. Selective distribution of matrix metalloproteinase-3 (MMP-3) in Alzheimer’s disease brain , 2000, Acta Neuropathologica.
[22] M. Fujimura,et al. Early appearance of activated matrix metalloproteinase-9 and blood–brain barrier disruption in mice after focal cerebral ischemia and reperfusion , 1999, Brain Research.
[23] A. Członkowska,et al. The Inflammatory Reaction Following 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine Intoxication in Mouse , 1999, Experimental Neurology.
[24] P. Gottschall,et al. Regional and differential expression of gelatinases in rat brain after systemic kainic acid or bicuculline administration , 1998, The European journal of neuroscience.
[25] J. Penney,et al. 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyride Neurotoxicity Is Attenuated in Mice Overexpressing Bcl-2 , 1998, The Journal of Neuroscience.
[26] S. Lorenzl,et al. Matrix metalloproteinases contribute to the blood—brain barrier disruption during bacterial meningitis , 1998, Annals of neurology.
[27] M. Beal,et al. Excitotoxicity and nitric oxide in parkinson's disease pathogenesis , 1998, Annals of neurology.
[28] M. Mass,et al. The MAP Kinase Cascade Is Activated prior to the Induction of Gliosis in the 1‐Methyl‐4‐phenyl‐1,2,3,6‐tetrahydropyridine (MPTP) Model of Dopaminergic Neurotoxicity , 1998, Annals of the New York Academy of Sciences.
[29] M. Fini,et al. Regulation of Matrix Metalloproteinase Gene Expression , 1998 .
[30] J. O'Callaghan,et al. The MAP kinase cascade is activated prior to the induction of gliosis in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) model of dopaminergic neurotoxicity. , 1998, Annals of the New York Academy of Sciences.
[31] A. Clark,et al. Increased gelatinase A (MMP-2) and gelatinase B (MMP-9) activities in human brain after focal ischemia , 1997, Neuroscience Letters.
[32] K. Nishi. Expression of c-Jun in dopaminergic neurons of the substantia nigra in 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-treated mice , 1997, Brain Research.
[33] P. Gottschall,et al. Zymographic measurement of gelatinase activity in brain tissue after detergent extraction and affinity-support purification , 1997, Journal of Neuroscience Methods.
[34] C. Miller,et al. Matrix Metalloproteinases in the Neocortex and Spinal Cord of Amyotrophic Lateral Sclerosis Patients , 1996, Journal of neurochemistry.
[35] R. Sobel,et al. Matrix Metalloproteinases in the Normal Human Central Nervous System, Microglial Nodules, and Multiple Sclerosis Lesions , 1996, Journal of neuropathology and experimental neurology.
[36] M. Beal,et al. Aging, energy, and oxidative stress in neurodegenerative diseases , 1995, Annals of neurology.
[37] S. Zucker,et al. Localization of Tissue Inhibitor of Matrix Metalloproteinases in Alzheimer's Disease and Normal Brain , 1995, Journal of neuropathology and experimental neurology.
[38] W. Stetler-Stevenson,et al. Quantitative zymography: detection of picogram quantities of gelatinases. , 1994, Analytical biochemistry.
[39] H. Nagase. Matrix metalloproteinases. A mini-review. , 1994, Contributions to nephrology.
[40] G. Opdenakker,et al. Gelatinase B is present in the cerebrospinal fluid during experimental autoimmune encephalomyelitis and cleaves myelin basic protein , 1993, Journal of neuroscience research.
[41] K. Tipton,et al. Advances in Our Understanding of the Mechanisms of the Neurotoxicity of MPTP and Related Compounds , 1993, Journal of neurochemistry.
[42] Miki Hasegawa,et al. A metalloproteinase inhibitor domain in Alzheimer amyloid protein precursor , 1993, Nature.
[43] A. Duchemin,et al. c-fos mRNA in mouse brain after MPTP treatment , 1992, Neurochemistry International.
[44] Richard E. Coggeshall,et al. A consideration of neural counting methods , 1992, Trends in Neurosciences.
[45] R. Kurlan,et al. The time course and magnitude of spontaneous recovery of parkinsonism produced by intracarotid administration of 1‐Methyl‐4‐Phenyl‐1,2,3,6‐tetrahydropyridine to monkeys , 1991, Annals of neurology.
[46] I. Date,et al. Long-term effect of MPTP in the mouse brain in relation to aging: neurochemical and immunocytochemical analysis , 1990, Brain Research.
[47] J. Langston,et al. Chronic Parkinsonism in humans due to a product of meperidine-analog synthesis. , 1983, Science.