Elevations in Circulating sST2 Levels Are Associated With In-hospital Mortality and Adverse Clinical Outcomes After Blunt Trauma.
暂无分享,去创建一个
[1] Y. Vodovotz,et al. A conceptual time window‐based model for the early stratification of trauma patients , 2019, Journal of internal medicine.
[2] Y. Vodovotz,et al. Young and Aged Blunt Trauma Patients Display Major Differences in Circulating Inflammatory Mediator Profiles after Severe Injury. , 2019, Journal of the American College of Surgeons.
[3] T. Billiar,et al. Interleukin‐33 contributes to ILC2 activation and early inflammation‐associated lung injury during abdominal sepsis , 2018, Immunology and cell biology.
[4] J. Lord,et al. Prehospital immune responses and development of multiple organ dysfunction syndrome following traumatic injury: A prospective cohort study , 2017, PLoS medicine.
[5] D. Pennington,et al. Signatures of inflammation and impending multiple organ dysfunction in the hyperacute phase of trauma: A prospective cohort study , 2017, PLoS medicine.
[6] Y. Vodovotz,et al. IL33-mediated ILC2 activation and neutrophil IL5 production in the lung response after severe trauma: A reverse translation study from a human cohort to a mouse trauma model , 2017, PLoS medicine.
[7] J. Routy,et al. Plasma Level of Soluble ST2 in Chronically Infected HIV-1 Patients with Suppressed Viremia , 2017, The open AIDS journal.
[8] T. Billiar,et al. Role of the IL-33-ST2 axis in sepsis , 2017, Military Medical Research.
[9] Mohsen Naghavi,et al. US County-Level Trends in Mortality Rates for Major Causes of Death, 1980-2014. , 2016, JAMA.
[10] Qi Mi,et al. Individual-specific principal component analysis of circulating inflammatory mediators predicts early organ dysfunction in trauma patients. , 2016, Journal of critical care.
[11] A. Peitzman,et al. Elevated Admission Base Deficit Is Associated with a Complex Dynamic Network of Systemic Inflammation Which Drives Clinical Trajectories in Blunt Trauma Patients , 2016, Mediators of inflammation.
[12] A. Peitzman,et al. Computational Analysis Supports an Early, Type 17 Cell-Associated Divergence of Blunt Trauma Survival and Mortality* , 2016, Critical care medicine.
[13] F. Liew,et al. Interleukin-33 in health and disease , 2016, Nature Reviews Immunology.
[14] P. Krijnen,et al. Predictive value of cytokines for developing complications after polytrauma. , 2016, World journal of critical care medicine.
[15] H. Turnquist,et al. Peri-alloHCT IL-33 administration expands recipient T-regulatory cells that protect mice against acute GVHD. , 2016, Blood.
[16] Qi Mi,et al. Insights into the Role of Chemokines, Damage-Associated Molecular Patterns, and Lymphocyte-Derived Mediators from Computational Models of Trauma-Induced Inflammation. , 2015, Antioxidants & redox signaling.
[17] T. Macdonald,et al. The IL-33/ST2 axis: Role in health and disease. , 2015, Cytokine & growth factor reviews.
[18] H. Hanenberg,et al. ST2 blockade reduces sST2-producing T cells while maintaining protective mST2-expressing T cells during graft-versus-host disease , 2015, Science Translational Medicine.
[19] G. Bandara,et al. Activated mast cells synthesize and release soluble ST2‐a decoy receptor for IL‐33 , 2015, European journal of immunology.
[20] M. Hur,et al. Soluble ST2 Has a Prognostic Role in Patients With Suspected Sepsis , 2015, Annals of laboratory medicine.
[21] D. Artis,et al. IL-33 promotes an innate immune pathway of intestinal tissue protection dependent on amphiregulin–EGFR interactions , 2015, Proceedings of the National Academy of Sciences.
[22] A. Peitzman,et al. Impact of Injury Severity on Dynamic Inflammation Networks Following Blunt Trauma , 2015, Shock.
[23] F. Moore,et al. Persistent inflammatory, immunosuppressed, catabolic syndrome (PICS): A new phenotype of multiple organ failure. , 2015, Journal of advanced nutritional and human metabolism.
[24] T. Mueller,et al. Soluble ST2 in heart failure. , 2015, Clinica chimica acta; international journal of clinical chemistry.
[25] Zhiliang Gao,et al. Soluble ST2 Plasma Concentrations Predict Mortality in HBV-Related Acute-on-Chronic Liver Failure , 2015, Mediators of inflammation.
[26] Li Zhang,et al. IL-33 promotes ST2-dependent lung fibrosis by the induction of alternatively activated macrophages and innate lymphoid cells in mice , 2014, The Journal of allergy and clinical immunology.
[27] H. Turnquist,et al. IL-33 Is an Unconventional Alarmin That Stimulates IL-2 Secretion by Dendritic Cells To Selectively Expand IL-33R/ST2+ Regulatory T Cells , 2014, The Journal of Immunology.
[28] P. Chu,et al. Soluble ST2: A Novel Prognostic Biomarker of Heart Failure. , 2014, Acta Cardiologica Sinica.
[29] M. Cohen. Acute traumatic coagulopathy: clinical characterization and mechanistic investigation. , 2014, Thrombosis research.
[30] N. Arsenijević,et al. Interleukin‐33/ST2 axis promotes breast cancer growth and metastases by facilitating intratumoral accumulation of immunosuppressive and innate lymphoid cells , 2014, International journal of cancer.
[31] M. Drazner,et al. 2013 ACCF/AHA guideline for the management of heart failure: a report of the American College of Cardiology Foundation/American Heart Association Task Force on Practice Guidelines. , 2013, Journal of the American College of Cardiology.
[32] T. Braun,et al. ST2 as a marker for risk of therapy-resistant graft-versus-host disease and death. , 2013, The New England journal of medicine.
[33] V. Bühren,et al. Outcome after severe multiple trauma: a retrospective analysis , 2013, Journal of trauma management & outcomes.
[34] B. Kronenberger,et al. High Serum Levels of the Interleukin-33 Receptor Soluble ST2 as a Negative Prognostic Factor in Hepatocellular Carcinoma. , 2013, Translational oncology.
[35] T. Osler,et al. Outcomes of adult trauma patients admitted to trauma centers in Pennsylvania, 2000-2009. , 2012, Archives of surgery.
[36] F. Liew,et al. Interleukin-33 and the function of innate lymphoid cells. , 2012, Trends in immunology.
[37] A. Peitzman,et al. Racial disparities and sex-based outcomes differences after severe injury. , 2012, Journal of the American College of Surgeons.
[38] K. Brohi,et al. Acute traumatic coagulopathy , 2012, Current opinion in anaesthesiology.
[39] John D. Storey,et al. A genomic storm in critically injured humans , 2011, The Journal of experimental medicine.
[40] Ashley M. Miller,et al. IL‐33 induces skin inflammation with mast cell and neutrophil activation , 2011, European journal of immunology.
[41] R. Tompkins,et al. EARLY ELEVATION IN RANDOM PLASMA IL-6 AFTER SEVERE INJURY IS ASSOCIATED WITH DEVELOPMENT OF ORGAN FAILURE , 2010, Shock.
[42] M. Mildner,et al. Primary sources and immunological prerequisites for sST2 secretion in humans. , 2010, Cardiovascular research.
[43] A. Nathens,et al. The Attributable Mortality and Length of Stay of Trauma-Related Complications: A Matched Cohort Study , 2010, Annals of surgery.
[44] David Díaz-Jiménez,et al. Characterization of the novel ST2/IL‐33 system in patients with inflammatory bowel disease , 2010, Inflammatory bowel diseases.
[45] F. Liew,et al. Interleukin-33 attenuates sepsis by enhancing neutrophil influx to the site of infection , 2010, Nature Medicine.
[46] J. Januzzi,et al. ST2: A Novel Remodeling Biomarker in Acute and Chronic Heart Failure , 2010, Current heart failure reports.
[47] T. van der Poll,et al. Soluble ST2 plasma concentrations predict mortality in severe sepsis , 2010, Intensive Care Medicine.
[48] I. McInnes,et al. Disease-associated functions of IL-33: the new kid in the IL-1 family , 2010, Nature Reviews Immunology.
[49] T. van der Poll,et al. Soluble ST2 Levels Are Associated with Bleeding in Patients with Severe Leptospirosis , 2009, PLoS neglected tropical diseases.
[50] Dirk E. Smith,et al. Inhibition of interleukin-33 signaling attenuates the severity of experimental arthritis. , 2009, Arthritis and rheumatism.
[51] Richard T. Lee,et al. The IL-33/ST2 pathway: therapeutic target and novel biomarker , 2008, Nature Reviews Drug Discovery.
[52] A. Rothman,et al. Elevated levels of soluble ST2 protein in dengue virus infected patients. , 2008, Cytokine.
[53] G. Franklin,et al. Systemic inflammation after trauma. , 2007, Injury.
[54] S. Tschoeke,et al. Immunoparalysis after multiple trauma. , 2007, Injury.
[55] M. Hayakawa,et al. Soluble ST2 Blocks Interleukin-33 Signaling in Allergic Airway Inflammation* , 2007, Journal of Biological Chemistry.
[56] B. Maier,et al. EARLY VERSUS LATE ONSET OF MULTIPLE ORGAN FAILURE IS ASSOCIATED WITH DIFFERING PATTERNS OF PLASMA CYTOKINE BIOMARKER EXPRESSION AND OUTCOME AFTER SEVERE TRAUMA , 2007, Shock.
[57] Javed Siddiqui,et al. Circulating Cytokine/Inhibitor Profiles Reshape the Understanding of the SIRS/CARS Continuum in Sepsis and Predict Mortality1 , 2006, The Journal of Immunology.
[58] J Fernando Bazan,et al. IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines. , 2005, Immunity.
[59] S. Tominaga,et al. Functional SNPs in the distal promoter of the ST2 gene are associated with atopic dermatitis. , 2005, Human molecular genetics.
[60] Richard T. Lee,et al. Serum Levels of the Interleukin-1 Receptor Family Member ST2 Predict Mortality and Clinical Outcome in Acute Myocardial Infarction , 2004, Circulation.
[61] A. Spittler,et al. Increased levels of soluble ST2 protein and IgG1 production in patients with sepsis and trauma , 2004, Intensive Care Medicine.
[62] Richard T. Lee,et al. Identification of Serum Soluble ST2 Receptor as a Novel Heart Failure Biomarker , 2003, Circulation.
[63] S. Tominaga,et al. ST2 protein induced by inflammatory stimuli can modulate acute lung inflammation. , 2002, Biochemical and biophysical research communications.
[64] S. Tominaga,et al. Elevated soluble ST2 protein levels in sera of patients with asthma with an acute exacerbation. , 2001, American journal of respiratory and critical care medicine.
[65] C. Sprung,et al. Multiple organ dysfunction score: a reliable descriptor of a complex clinical outcome. , 1995, Critical care medicine.
[66] R. Sauerwein,et al. Cytokine Patterns in Patients After Major Vascular Surgery, Hemorrhagic Shock, and Severe Blunt Trauma Relation with Subsequent Adult Respiratory Distress Syndrome and Multiple Organ Failure , 1993, Annals of surgery.
[67] L. Carey,et al. Correlation of metabolic acidosis with outcome following injury and its value as a scoring tool , 1993, World Journal of Surgery.
[68] J. Siegel,et al. Oxygen debt and metabolic acidemia as quantitative predictors of mortality and the severity of the ischemic insult in hemorrhagic shock , 1991, Critical care medicine.
[69] W. Haddon,et al. The injury severity score: a method for describing patients with multiple injuries and evaluating emergency care. , 1974, The Journal of trauma.
[70] A. Peitzman,et al. Temporal Patterns of Circulating Inflammation Biomarker Networks Differentiate Susceptibility to Nosocomial Infection Following Blunt Trauma in Humans. , 2016, Annals of surgery.
[71] M. Maegele. Acute traumatic coagulopathy: Incidence, risk stratification and therapeutic options. , 2010, World journal of emergency medicine.
[72] G. Lang,et al. Increased soluble serum markers caspase‐cleaved cytokeratin‐18, histones, and ST2 indicate apoptotic turnover and chronic immune response in COPD , 2009, Journal of clinical laboratory analysis.
[73] I. Pallister. Current concepts of the inflammatory response after major trauma: an update. , 2005, Injury.